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Items: 1 to 20 of 113

1.

Non-steroidal anti-inflammatory drugs decrease E2F1 expression and inhibit cell growth in ovarian cancer cells.

Valle BL, D'Souza T, Becker KG, Wood WH 3rd, Zhang Y, Wersto RP, Morin PJ.

PLoS One. 2013 Apr 24;8(4):e61836. doi: 10.1371/journal.pone.0061836. Print 2013.

2.

Combinatorial effect of non-steroidal anti-inflammatory drugs and NF-κB inhibitors in ovarian cancer therapy.

Zerbini LF, Tamura RE, Correa RG, Czibere A, Cordeiro J, Bhasin M, Simabuco FM, Wang Y, Gu X, Li L, Sarkar D, Zhou JR, Fisher PB, Libermann TA.

PLoS One. 2011;6(9):e24285. doi: 10.1371/journal.pone.0024285. Epub 2011 Sep 12.

3.

Cyclooxygenase inhibitors induce apoptosis in sinonasal cancer cells by increased expression of nonsteroidal anti-inflammatory drug-activated gene.

Kim JH, Chang JH, Rhee KH, Yoon JH, Kwon SH, Song K, Lee KW, Cho CI, Jeon JH, Kim KS.

Int J Cancer. 2008 Apr 15;122(8):1765-73.

4.

Small molecule tolfenamic acid inhibits PC-3 cell proliferation and invasion in vitro, and tumor growth in orthotopic mouse model for prostate cancer.

Sankpal UT, Abdelrahim M, Connelly SF, Lee CM, Madero-Visbal R, Colon J, Smith J, Safe S, Maliakal P, Basha R.

Prostate. 2012 Nov;72(15):1648-58. doi: 10.1002/pros.22518. Epub 2012 Apr 2.

PMID:
22473873
5.

Comparative effects of indomethacin on cell proliferation and cell cycle progression in tumor cells grown in vitro and in vivo.

Eli Y, Przedecki F, Levin G, Kariv N, Raz A.

Biochem Pharmacol. 2001 Mar 1;61(5):565-71.

PMID:
11239499
6.

Diclofenac, a non-steroidal anti-inflammatory drug, suppresses apoptosis induced by endoplasmic reticulum stresses by inhibiting caspase signaling.

Yamazaki T, Muramoto M, Oe T, Morikawa N, Okitsu O, Nagashima T, Nishimura S, Katayama Y, Kita Y.

Neuropharmacology. 2006 Apr;50(5):558-67. Epub 2006 Jan 4.

PMID:
16388830
7.

Non-steroidal anti-inflammatory drugs (NSAIDs) inhibit vascular smooth muscle cell proliferation via differential effects on the cell cycle.

Brooks G, Yu XM, Wang Y, Crabbe MJ, Shattock MJ, Harper JV.

J Pharm Pharmacol. 2003 Apr;55(4):519-26.

PMID:
12803774
8.

Eugenol triggers apoptosis in breast cancer cells through E2F1/survivin down-regulation.

Al-Sharif I, Remmal A, Aboussekhra A.

BMC Cancer. 2013 Dec 13;13:600. doi: 10.1186/1471-2407-13-600.

9.

Antiproliferative effects of artemisinin on human breast cancer cells requires the downregulated expression of the E2F1 transcription factor and loss of E2F1-target cell cycle genes.

Tin AS, Sundar SN, Tran KQ, Park AH, Poindexter KM, Firestone GL.

Anticancer Drugs. 2012 Apr;23(4):370-9. doi: 10.1097/CAD.0b013e32834f6ea8.

PMID:
22185819
10.

Non-steroidal anti-inflammatory drug-induced apoptosis in gastric cancer cells is blocked by protein kinase C activation through inhibition of c-myc.

Zhu GH, Wong BC, Eggo MC, Ching CK, Yuen ST, Chan EY, Lai KC, Lam SK.

Br J Cancer. 1999 Feb;79(3-4):393-400.

11.

Nitroxide derivatives of non-steroidal anti-inflammatory drugs exert anti-inflammatory and superoxide dismutase scavenging properties in A459 cells.

Flores-Santana W, Moody T, Chen W, Gorczynski MJ, Shoman ME, Velázquez C, Thetford A, Mitchell JB, Cherukuri MK, King SB, Wink DA.

Br J Pharmacol. 2012 Feb;165(4b):1058-67. doi: 10.1111/j.1476-5381.2011.01527.x.

12.

Effects of indomethacin, nimesulide, and diclofenac on human MG-63 osteosarcoma cell line.

Díaz-Rodríguez L, García-Martínez O, Morales MA, Rodríguez-Pérez L, Rubio-Ruiz B, Ruiz C.

Biol Res Nurs. 2012 Jan;14(1):98-107. doi: 10.1177/1099800411398933. Epub 2011 Mar 8.

PMID:
21385796
13.

Downregulation of thymidylate synthase and E2F1 by arsenic trioxide in mesothelioma.

Lam SK, Li YY, Zheng CY, Ho JC.

Int J Oncol. 2015 Jan;46(1):113-22. doi: 10.3892/ijo.2014.2716. Epub 2014 Oct 21.

PMID:
25335113
14.

Ibuprofen and Diclofenac Restrict Migration and Proliferation of Human Glioma Cells by Distinct Molecular Mechanisms.

Leidgens V, Seliger C, Jachnik B, Welz T, Leukel P, Vollmann-Zwerenz A, Bogdahn U, Kreutz M, Grauer OM, Hau P.

PLoS One. 2015 Oct 20;10(10):e0140613. doi: 10.1371/journal.pone.0140613. eCollection 2015.

15.

Anti-inflammatory drugs suppress proliferation and induce apoptosis through altering expressions of cell cycle regulators and pro-apoptotic factors in cultured human osteoblasts.

Chang JK, Li CJ, Liao HJ, Wang CK, Wang GJ, Ho ML.

Toxicology. 2009 Apr 28;258(2-3):148-56. doi: 10.1016/j.tox.2009.01.016. Epub 2009 Jan 22.

PMID:
19428934
16.

Endoplasmic reticulum stress response is involved in nonsteroidal anti-inflammatory drug-induced apoptosis.

Tsutsumi S, Gotoh T, Tomisato W, Mima S, Hoshino T, Hwang HJ, Takenaka H, Tsuchiya T, Mori M, Mizushima T.

Cell Death Differ. 2004 Sep;11(9):1009-16.

17.

Carboxylesterases 1 and 2 hydrolyze phospho-nonsteroidal anti-inflammatory drugs: relevance to their pharmacological activity.

Wong CC, Cheng KW, Xie G, Zhou D, Zhu CH, Constantinides PP, Rigas B.

J Pharmacol Exp Ther. 2012 Feb;340(2):422-32. doi: 10.1124/jpet.111.188508. Epub 2011 Nov 15.

18.

Nonsteroidal anti-inflammatory drugs suppress cancer stem cells via inhibiting PTGS2 (cyclooxygenase 2) and NOTCH/HES1 and activating PPARG in colorectal cancer.

Moon CM, Kwon JH, Kim JS, Oh SH, Jin Lee K, Park JJ, Pil Hong S, Cheon JH, Kim TI, Kim WH.

Int J Cancer. 2014 Feb 1;134(3):519-29. doi: 10.1002/ijc.28381. Epub 2013 Aug 7.

19.

Nonsteroidal anti-inflammatory drugs inhibit the growth of C6 and U138-MG glioma cell lines.

Bernardi A, Jacques-Silva MC, Delgado-Cañedo A, Lenz G, Battastini AM.

Eur J Pharmacol. 2006 Feb 27;532(3):214-22. Epub 2006 Feb 17.

PMID:
16487511
20.

Repercussions of NSAIDS drugs on bone tissue: the osteoblast.

García-Martínez O, De Luna-Bertos E, Ramos-Torrecillas J, Manzano-Moreno FJ, Ruiz C.

Life Sci. 2015 Feb 15;123:72-7. doi: 10.1016/j.lfs.2015.01.009. Epub 2015 Jan 24. Review.

PMID:
25625244

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