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Items: 1 to 20 of 129

1.

Barrier abnormality due to ceramide deficiency leads to psoriasiform inflammation in a mouse model.

Nakajima K, Terao M, Takaishi M, Kataoka S, Goto-Inoue N, Setou M, Horie K, Sakamoto F, Ito M, Azukizawa H, Kitaba S, Murota H, Itami S, Katayama I, Takeda J, Sano S.

J Invest Dermatol. 2013 Nov;133(11):2555-2565. doi: 10.1038/jid.2013.199. Epub 2013 Apr 30.

2.

Epidermal CCR6+ γδ T cells are major producers of IL-22 and IL-17 in a murine model of psoriasiform dermatitis.

Mabuchi T, Takekoshi T, Hwang ST.

J Immunol. 2011 Nov 15;187(10):5026-31. doi: 10.4049/jimmunol.1101817. Epub 2011 Oct 7.

3.

IL-23 from Langerhans cells is required for the development of imiquimod-induced psoriasis-like dermatitis by induction of IL-17A-producing γδ T cells.

Yoshiki R, Kabashima K, Honda T, Nakamizo S, Sawada Y, Sugita K, Yoshioka H, Ohmori S, Malissen B, Tokura Y, Nakamura M.

J Invest Dermatol. 2014 Jul;134(7):1912-1921. doi: 10.1038/jid.2014.98. Epub 2014 Feb 25.

4.

Psoriasiform dermatitis is driven by IL-36-mediated DC-keratinocyte crosstalk.

Tortola L, Rosenwald E, Abel B, Blumberg H, Schäfer M, Coyle AJ, Renauld JC, Werner S, Kisielow J, Kopf M.

J Clin Invest. 2012 Nov;122(11):3965-76. doi: 10.1172/JCI63451. Epub 2012 Oct 15.

5.

IL-22 is required for imiquimod-induced psoriasiform skin inflammation in mice.

Van Belle AB, de Heusch M, Lemaire MM, Hendrickx E, Warnier G, Dunussi-Joannopoulos K, Fouser LA, Renauld JC, Dumoutier L.

J Immunol. 2012 Jan 1;188(1):462-9. doi: 10.4049/jimmunol.1102224. Epub 2011 Nov 30.

6.

Loss of epidermal Evi/Wls results in a phenotype resembling psoriasiform dermatitis.

Augustin I, Gross J, Baumann D, Korn C, Kerr G, Grigoryan T, Mauch C, Birchmeier W, Boutros M.

J Exp Med. 2013 Aug 26;210(9):1761-77. doi: 10.1084/jem.20121871. Epub 2013 Aug 5.

7.

Obesity exacerbates imiquimod-induced psoriasis-like epidermal hyperplasia and interleukin-17 and interleukin-22 production in mice.

Kanemaru K, Matsuyuki A, Nakamura Y, Fukami K.

Exp Dermatol. 2015 Jun;24(6):436-42. doi: 10.1111/exd.12691. Epub 2015 Apr 16.

PMID:
25777289
8.

CCR6 is required for epidermal trafficking of γδ-T cells in an IL-23-induced model of psoriasiform dermatitis.

Mabuchi T, Singh TP, Takekoshi T, Jia GF, Wu X, Kao MC, Weiss I, Farber JM, Hwang ST.

J Invest Dermatol. 2013 Jan;133(1):164-71. doi: 10.1038/jid.2012.260. Epub 2012 Aug 16.

9.

CD8(+) T cells mediate RAS-induced psoriasis-like skin inflammation through IFN-γ.

Gunderson AJ, Mohammed J, Horvath FJ, Podolsky MA, Anderson CR, Glick AB.

J Invest Dermatol. 2013 Apr;133(4):955-63. doi: 10.1038/jid.2012.390. Epub 2012 Nov 15.

10.

Counterregulation between thymic stromal lymphopoietin- and IL-23-driven immune axes shapes skin inflammation in mice with epidermal barrier defects.

Li J, Leyva-Castillo JM, Hener P, Eisenmann A, Zaafouri S, Jonca N, Serre G, Birling MC, Li M.

J Allergy Clin Immunol. 2016 Jul;138(1):150-161.e13. doi: 10.1016/j.jaci.2016.01.013. Epub 2016 Mar 15.

PMID:
26993035
11.

Rorγt+ innate lymphocytes and γδ T cells initiate psoriasiform plaque formation in mice.

Pantelyushin S, Haak S, Ingold B, Kulig P, Heppner FL, Navarini AA, Becher B.

J Clin Invest. 2012 Jun;122(6):2252-6. doi: 10.1172/JCI61862. Epub 2012 May 1.

12.

Imiquimod-induced psoriasis-like skin inflammation in mice is mediated via the IL-23/IL-17 axis.

van der Fits L, Mourits S, Voerman JS, Kant M, Boon L, Laman JD, Cornelissen F, Mus AM, Florencia E, Prens EP, Lubberts E.

J Immunol. 2009 May 1;182(9):5836-45. doi: 10.4049/jimmunol.0802999.

13.

Nociceptive sensory neurons drive interleukin-23-mediated psoriasiform skin inflammation.

Riol-Blanco L, Ordovas-Montanes J, Perro M, Naval E, Thiriot A, Alvarez D, Paust S, Wood JN, von Andrian UH.

Nature. 2014 Jun 5;510(7503):157-61. doi: 10.1038/nature13199. Epub 2014 Apr 23.

14.

Thymic stromal lymphopoietin-induced interleukin-17A is involved in the development of IgE-mediated atopic dermatitis-like skin lesions in mice.

Mizutani N, Sae-Wong C, Kangsanant S, Nabe T, Yoshino S.

Immunology. 2015 Dec;146(4):568-81. doi: 10.1111/imm.12528. Epub 2015 Sep 24.

15.

Th1 cytokines accentuate but Th2 cytokines attenuate ceramide production in the stratum corneum of human epidermal equivalents: an implication for the disrupted barrier mechanism in atopic dermatitis.

Sawada E, Yoshida N, Sugiura A, Imokawa G.

J Dermatol Sci. 2012 Oct;68(1):25-35. doi: 10.1016/j.jdermsci.2012.07.004. Epub 2012 Jul 20.

PMID:
22884781
16.
17.

Distinct roles of IL-23 and IL-17 in the development of psoriasis-like lesions in a mouse model.

Nakajima K, Kanda T, Takaishi M, Shiga T, Miyoshi K, Nakajima H, Kamijima R, Tarutani M, Benson JM, Elloso MM, Gutshall LL, Naso MF, Iwakura Y, DiGiovanni J, Sano S.

J Immunol. 2011 Apr 1;186(7):4481-9. doi: 10.4049/jimmunol.1000148. Epub 2011 Feb 23. Erratum in: J Immunol. 2011 Dec 1;187(11):6157-8.

18.

Epidermal IL-15Rα acts as an endogenous antagonist of psoriasiform inflammation in mouse and man.

Bouchaud G, Gehrke S, Krieg C, Kolios A, Hafner J, Navarini AA, French LE, Boyman O.

J Exp Med. 2013 Sep 23;210(10):2105-17. doi: 10.1084/jem.20130291. Epub 2013 Sep 9.

19.

Pivotal role of dermal IL-17-producing γδ T cells in skin inflammation.

Cai Y, Shen X, Ding C, Qi C, Li K, Li X, Jala VR, Zhang HG, Wang T, Zheng J, Yan J.

Immunity. 2011 Oct 28;35(4):596-610. doi: 10.1016/j.immuni.2011.08.001. Epub 2011 Oct 6. Erratum in: Immunity. 2011 Oct 28;35(4):649.

20.

IL-6 regulates neutrophil microabscess formation in IL-17A-driven psoriasiform lesions.

Croxford AL, Karbach S, Kurschus FC, Wörtge S, Nikolaev A, Yogev N, Klebow S, Schüler R, Reissig S, Piotrowski C, Brylla E, Bechmann I, Scheller J, Rose-John S, Thomas Wunderlich F, Münzel T, von Stebut E, Waisman A.

J Invest Dermatol. 2014 Mar;134(3):728-735. doi: 10.1038/jid.2013.404. Epub 2013 Sep 25.

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