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Expression of DNA-PKcs and BRCA1 as prognostic indicators in nasopharyngeal carcinoma following intensity-modulated radiation therapy.

Yang J, Xu X, Hao Y, Chen J, Lu H, Qin J, Peng L, Chen B.

Oncol Lett. 2013 Apr;5(4):1199-1204. Epub 2013 Feb 18.


[Expression and clinical significance of DNA-PKcs in nasopharyngeal carcinoma].

Yan SS, Liu L, Liu ZG, Zeng MS, Song LB, Xia YF.

Ai Zheng. 2008 Sep;27(9):979-83. Chinese.


BRCA1 modulates the autophosphorylation status of DNA-PKcs in S phase of the cell cycle.

Davis AJ, Chi L, So S, Lee KJ, Mori E, Fattah K, Yang J, Chen DJ.

Nucleic Acids Res. 2014 Oct;42(18):11487-501. doi: 10.1093/nar/gku824. Epub 2014 Sep 15.


Adverse prognostic and predictive significance of low DNA-dependent protein kinase catalytic subunit (DNA-PKcs) expression in early-stage breast cancers.

Abdel-Fatah T, Arora A, Agarwal D, Moseley P, Perry C, Thompson N, Green AR, Rakha E, Chan S, Ball G, Ellis IO, Madhusudan S.

Breast Cancer Res Treat. 2014 Jul;146(2):309-20. doi: 10.1007/s10549-014-3035-2. Epub 2014 Jun 28.


Expressions of Ku70 and DNA-PKcs as prognostic indicators of local control in nasopharyngeal carcinoma.

Lee SW, Cho KJ, Park JH, Kim SY, Nam SY, Lee BJ, Kim SB, Choi SH, Kim JH, Ahn SD, Shin SS, Choi EK, Yu E.

Int J Radiat Oncol Biol Phys. 2005 Aug 1;62(5):1451-7.


PI3K-Akt signaling regulates basal, but MAP-kinase signaling regulates radiation-induced XRCC1 expression in human tumor cells in vitro.

Toulany M, Dittmann K, Fehrenbacher B, Schaller M, Baumann M, Rodemann HP.

DNA Repair (Amst). 2008 Oct 1;7(10):1746-56. doi: 10.1016/j.dnarep.2008.06.017. Epub 2008 Aug 22.


DNA-PKcs expression predicts response to radiotherapy in prostate cancer.

Bouchaert P, Guerif S, Debiais C, Irani J, Fromont G.

Int J Radiat Oncol Biol Phys. 2012 Dec 1;84(5):1179-85. doi: 10.1016/j.ijrobp.2012.02.014. Epub 2012 Apr 9.


Regulation of DNA double-strand break repair pathway choice.

Shrivastav M, De Haro LP, Nickoloff JA.

Cell Res. 2008 Jan;18(1):134-47. Review.


Nuclear survivin and its relationship to DNA damage repair genes in non-small cell lung cancer investigated using tissue array.

Hu S, Qu Y, Xu X, Xu Q, Geng J, Xu J.

PLoS One. 2013 Sep 16;8(9):e74161. doi: 10.1371/journal.pone.0074161. eCollection 2013. Erratum in: PLoS One. 2013;8(12). doi:10.1371/annotation/a87d46d5-04ee-4c57-af2b-7b9af5166cd8.


DNA-dependent protein kinase catalytic subunit functions in metastasis and influences survival in advanced-stage laryngeal squamous cell carcinoma.

He SS, Chen Y, Shen XM, Wang HZ, Sun P, Dong J, Guo GF, Chen JG, Xia LP, Hu PL, Qiu HJ, Liu SS, Zhou YX, Wang W, Hu WH, Cai XY.

J Cancer. 2017 Jul 22;8(12):2410-2416. doi: 10.7150/jca.20069. eCollection 2017.


Interactive competition between homologous recombination and non-homologous end joining.

Allen C, Halbrook J, Nickoloff JA.

Mol Cancer Res. 2003 Oct;1(12):913-20.


Akt promotes post-irradiation survival of human tumor cells through initiation, progression, and termination of DNA-PKcs-dependent DNA double-strand break repair.

Toulany M, Lee KJ, Fattah KR, Lin YF, Fehrenbacher B, Schaller M, Chen BP, Chen DJ, Rodemann HP.

Mol Cancer Res. 2012 Jul;10(7):945-57. doi: 10.1158/1541-7786.MCR-11-0592. Epub 2012 May 17.


Suppression of DNA-dependent protein kinase sensitize cells to radiation without affecting DSB repair.

Gustafsson AS, Abramenkovs A, Stenerlöw B.

Mutat Res. 2014 Nov;769:1-10. doi: 10.1016/j.mrfmmm.2014.06.004. Epub 2014 Jun 22.


Genetic evidence for the involvement of DNA ligase IV in the DNA-PK-dependent pathway of non-homologous end joining in mammalian cells.

Wang H, Zeng ZC, Perrault AR, Cheng X, Qin W, Iliakis G.

Nucleic Acids Res. 2001 Apr 15;29(8):1653-60.


DNA-dependent protein kinase suppresses double-strand break-induced and spontaneous homologous recombination.

Allen C, Kurimasa A, Brenneman MA, Chen DJ, Nickoloff JA.

Proc Natl Acad Sci U S A. 2002 Mar 19;99(6):3758-63.


Phosphatidylinositol 3-kinase/Akt signaling as a key mediator of tumor cell responsiveness to radiation.

Toulany M, Rodemann HP.

Semin Cancer Biol. 2015 Dec;35:180-90. doi: 10.1016/j.semcancer.2015.07.003. Epub 2015 Jul 17. Review.


Biochemical evidence for Ku-independent backup pathways of NHEJ.

Wang H, Perrault AR, Takeda Y, Qin W, Wang H, Iliakis G.

Nucleic Acids Res. 2003 Sep 15;31(18):5377-88.


Reduced DNA double strand breaks in chlorambucil resistant cells are related to high DNA-PKcs activity and low oxidative stress.

Boldogh I, Roy G, Lee MS, Bacsi A, Hazra TK, Bhakat KK, Das GC, Mitra S.

Toxicology. 2003 Nov 15;193(1-2):137-52.


Mechanisms of DNA double strand break repair and chromosome aberration formation.

Iliakis G, Wang H, Perrault AR, Boecker W, Rosidi B, Windhofer F, Wu W, Guan J, Terzoudi G, Pantelias G.

Cytogenet Genome Res. 2004;104(1-4):14-20. Review.


DNA-PKcs and ATM co-regulate DNA double-strand break repair.

Shrivastav M, Miller CA, De Haro LP, Durant ST, Chen BP, Chen DJ, Nickoloff JA.

DNA Repair (Amst). 2009 Aug 6;8(8):920-9. doi: 10.1016/j.dnarep.2009.05.006. Epub 2009 Jun 16.

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