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Cardiomyocyte dysfunction during the chronic phase of Chagas disease.

Roman-Campos D, Sales-Júnior P, Duarte HL, Gomes ER, Guatimosim S, Ropert C, Gazzinelli RT, Cruz JS.

Mem Inst Oswaldo Cruz. 2013 Apr;108(2):243-5.


Calcium entry via Na/Ca exchange during the action potential directly contributes to contraction of failing human ventricular myocytes.

Weisser-Thomas J, Piacentino V 3rd, Gaughan JP, Margulies K, Houser SR.

Cardiovasc Res. 2003 Mar 15;57(4):974-85.


Induced overexpression of Na+/Ca2+ exchanger transgene: altered myocyte contractility, [Ca2+]i transients, SR Ca2+ contents, and action potential duration.

Wang J, Chan TO, Zhang XQ, Gao E, Song J, Koch WJ, Feldman AM, Cheung JY.

Am J Physiol Heart Circ Physiol. 2009 Aug;297(2):H590-601. doi: 10.1152/ajpheart.00190.2009.


Simulation study of cellular electric properties in heart failure.

Priebe L, Beuckelmann DJ.

Circ Res. 1998 Jun 15;82(11):1206-23.


Increased cardiomyocyte function and Ca2+ transients in mice during early congestive heart failure.

Mørk HK, Sjaastad I, Sande JB, Periasamy M, Sejersted OM, Louch WE.

J Mol Cell Cardiol. 2007 Aug;43(2):177-86.


Intracellular Ca2+ release underlies the development of phase 2 in mouse ventricular action potentials.

Ferreiro M, Petrosky AD, Escobar AL.

Am J Physiol Heart Circ Physiol. 2012 Mar 1;302(5):H1160-72. doi: 10.1152/ajpheart.00524.2011.


Cellular basis of abnormal calcium transients of failing human ventricular myocytes.

Piacentino V 3rd, Weber CR, Chen X, Weisser-Thomas J, Margulies KB, Bers DM, Houser SR.

Circ Res. 2003 Apr 4;92(6):651-8.


Comparison of contraction and calcium handling between right and left ventricular myocytes from adult mouse heart: a role for repolarization waveform.

Kondo RP, Dederko DA, Teutsch C, Chrast J, Catalucci D, Chien KR, Giles WR.

J Physiol. 2006 Feb 15;571(Pt 1):131-46. Erratum in: J Physiol. 2006 Mar 1;571(Pt 2):499.


Novel insights into the development of chagasic cardiomyopathy: role of PI3Kinase/NO axis.

Roman-Campos D, Sales-Junior P, Duarte HL, Gomes ER, Lara A, Campos P, Rocha NN, Resende RR, Ferreira A, Guatimosim S, Gazzinelli RT, Ropert C, Cruz JS.

Int J Cardiol. 2013 Sep 10;167(6):3011-20. doi: 10.1016/j.ijcard.2012.09.020.


Sevoflurane protects ventricular myocytes against oxidative stress-induced cellular Ca2+ overload and hypercontracture.

Kojima A, Kitagawa H, Omatsu-Kanbe M, Matsuura H, Nosaka S.

Anesthesiology. 2013 Sep;119(3):606-20. doi: 10.1097/ALN.0b013e318292ee52.


Influence of age and run training on cardiac Na+/Ca2+ exchange.

Mace LC, Palmer BM, Brown DA, Jew KN, Lynch JM, Glunt JM, Parsons TA, Cheung JY, Moore RL.

J Appl Physiol (1985). 2003 Nov;95(5):1994-2003.


Insights into cardioprotection obtained from study of cellular Ca2+ handling in myocardium of true hibernating mammals.

Yatani A, Kim SJ, Kudej RK, Wang Q, Depre C, Irie K, Kranias EG, Vatner SF, Vatner DE.

Am J Physiol Heart Circ Physiol. 2004 Jun;286(6):H2219-28.


Properties and ionic mechanisms of action potential adaptation, restitution, and accommodation in canine epicardium.

Decker KF, Heijman J, Silva JR, Hund TJ, Rudy Y.

Am J Physiol Heart Circ Physiol. 2009 Apr;296(4):H1017-26. doi: 10.1152/ajpheart.01216.2008.


Alterations in early action potential repolarization causes localized failure of sarcoplasmic reticulum Ca2+ release.

Harris DM, Mills GD, Chen X, Kubo H, Berretta RM, Votaw VS, Santana LF, Houser SR.

Circ Res. 2005 Mar 18;96(5):543-50.

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