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Items: 1 to 20 of 106

1.

The synergy in cytokine production through MyD88-TRIF pathways is co-ordinated with ERK phosphorylation in macrophages.

Suet Ting Tan R, Lin B, Liu Q, Tucker-Kellogg L, Ho B, Leung BP, Ling Ding J.

Immunol Cell Biol. 2013 May;91(5):377-87. doi: 10.1038/icb.2013.13.

PMID:
23567895
2.

Phosphatase PTP1B negatively regulates MyD88- and TRIF-dependent proinflammatory cytokine and type I interferon production in TLR-triggered macrophages.

Xu H, An H, Hou J, Han C, Wang P, Yu Y, Cao X.

Mol Immunol. 2008 Aug;45(13):3545-52. doi: 10.1016/j.molimm.2008.05.006.

PMID:
18571728
3.

Role of adaptor TRIF in the MyD88-independent toll-like receptor signaling pathway.

Yamamoto M, Sato S, Hemmi H, Hoshino K, Kaisho T, Sanjo H, Takeuchi O, Sugiyama M, Okabe M, Takeda K, Akira S.

Science. 2003 Aug 1;301(5633):640-3.

4.

Signaling of apoptosis through TLRs critically involves toll/IL-1 receptor domain-containing adapter inducing IFN-beta, but not MyD88, in bacteria-infected murine macrophages.

Ruckdeschel K, Pfaffinger G, Haase R, Sing A, Weighardt H, Häcker G, Holzmann B, Heesemann J.

J Immunol. 2004 Sep 1;173(5):3320-8.

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Differential induction of MyD88- and TRIF-dependent pathways in equine monocytes by Toll-like receptor agonists.

Figueiredo MD, Vandenplas ML, Hurley DJ, Moore JN.

Vet Immunol Immunopathol. 2009 Jan 15;127(1-2):125-34. doi: 10.1016/j.vetimm.2008.09.028.

PMID:
19019456
9.

Dynamic regulation of pro- and anti-inflammatory cytokines by MAPK phosphatase 1 (MKP-1) in innate immune responses.

Chi H, Barry SP, Roth RJ, Wu JJ, Jones EA, Bennett AM, Flavell RA.

Proc Natl Acad Sci U S A. 2006 Feb 14;103(7):2274-9.

10.

TLR3-mediated IFN-β gene induction is negatively regulated by the TLR adaptor MyD88 adaptor-like.

Siednienko J, Halle A, Nagpal K, Golenbock DT, Miggin SM.

Eur J Immunol. 2010 Nov;40(11):3150-60. doi: 10.1002/eji.201040547.

11.

MyD88 signaling is indispensable for primary influenza A virus infection but dispensable for secondary infection.

Seo SU, Kwon HJ, Song JH, Byun YH, Seong BL, Kawai T, Akira S, Kweon MN.

J Virol. 2010 Dec;84(24):12713-22. doi: 10.1128/JVI.01675-10.

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Melatonin modulates TLR4-mediated inflammatory genes through MyD88- and TRIF-dependent signaling pathways in lipopolysaccharide-stimulated RAW264.7 cells.

Xia MZ, Liang YL, Wang H, Chen X, Huang YY, Zhang ZH, Chen YH, Zhang C, Zhao M, Xu DX, Song LH.

J Pineal Res. 2012 Nov;53(4):325-34. doi: 10.1111/j.1600-079X.2012.01002.x.

PMID:
22537289
15.

Release of IL-12 by dendritic cells activated by TLR ligation is dependent on MyD88 signaling, whereas TRIF signaling is indispensable for TLR synergy.

Krummen M, Balkow S, Shen L, Heinz S, Loquai C, Probst HC, Grabbe S.

J Leukoc Biol. 2010 Jul;88(1):189-99. doi: 10.1189/jlb.0408228.

PMID:
20360404
16.

Type 1 cytokine/chemokine production by mouse NK cells following activation of their TLR/MyD88-mediated pathways.

Sawaki J, Tsutsui H, Hayashi N, Yasuda K, Akira S, Tanizawa T, Nakanishi K.

Int Immunol. 2007 Mar;19(3):311-20.

17.

PKC-alpha controls MYD88-dependent TLR/IL-1R signaling and cytokine production in mouse and human dendritic cells.

Langlet C, Springael C, Johnson J, Thomas S, Flamand V, Leitges M, Goldman M, Aksoy E, Willems F.

Eur J Immunol. 2010 Feb;40(2):505-15. doi: 10.1002/eji.200939391.

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Innate immune responses to endosymbiotic Wolbachia bacteria in Brugia malayi and Onchocerca volvulus are dependent on TLR2, TLR6, MyD88, and Mal, but not TLR4, TRIF, or TRAM.

Hise AG, Daehnel K, Gillette-Ferguson I, Cho E, McGarry HF, Taylor MJ, Golenbock DT, Fitzgerald KA, Kazura JW, Pearlman E.

J Immunol. 2007 Jan 15;178(2):1068-76.

20.

Effect of TLR3 and TLR7 activation in uterine NK cells from non-obese diabetic (NOD) mice.

Lin Y, Ren L, Wang W, Di J, Zeng S, Saito S.

J Reprod Immunol. 2009 Oct;82(1):12-23. doi: 10.1016/j.jri.2009.03.004.

PMID:
19560213
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