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Items: 1 to 20 of 61

1.

Tissue inhibitor of metalloproteinase 1 is preferentially expressed in Th1 and Th17 T-helper cell subsets and is a direct STAT target gene.

Adamson A, Ghoreschi K, Rittler M, Chen Q, Sun HW, Vahedi G, Kanno Y, Stetler-Stevenson WG, O'Shea JJ, Laurence A.

PLoS One. 2013;8(3):e59367. doi: 10.1371/journal.pone.0059367. Epub 2013 Mar 26.

2.

STAT4 controls GM-CSF production by both Th1 and Th17 cells during EAE.

McWilliams IL, Rajbhandari R, Nozell S, Benveniste E, Harrington LE.

J Neuroinflammation. 2015 Jun 30;12:128. doi: 10.1186/s12974-015-0351-3.

3.

T cell-specific BLIMP-1 deficiency exacerbates experimental autoimmune encephalomyelitis in nonobese diabetic mice by increasing Th1 and Th17 cells.

Lin MH, Yeh LT, Chen SJ, Chiou HY, Chu CC, Yen LB, Lin KI, Chang DM, Sytwu HK.

Clin Immunol. 2014 Apr;151(2):101-13. doi: 10.1016/j.clim.2014.02.006. Epub 2014 Feb 21.

PMID:
24568746
4.

Role of Th17 cells in the pathogenesis of CNS inflammatory demyelination.

Rostami A, Ciric B.

J Neurol Sci. 2013 Oct 15;333(1-2):76-87. doi: 10.1016/j.jns.2013.03.002. Epub 2013 Apr 8. Review.

5.

Loss of STAT3 in CD4+ T cells prevents development of experimental autoimmune diseases.

Liu X, Lee YS, Yu CR, Egwuagu CE.

J Immunol. 2008 May 1;180(9):6070-6.

6.

Th1 versus Th17: are T cell cytokines relevant in multiple sclerosis?

Lovett-Racke AE, Yang Y, Racke MK.

Biochim Biophys Acta. 2011 Feb;1812(2):246-51. doi: 10.1016/j.bbadis.2010.05.012. Epub 2010 Jun 18. Review.

7.

A novel human truncated IL12rβ1-Fc fusion protein ameliorates experimental autoimmune encephalomyelitis via specific binding of p40 to inhibit Th1 and Th17 cell differentiation.

Guo W, Wang C, Wang X, Luo C, Yu D, Wang Y, Chen Y, Lei W, Gao X, Yao W.

Oncotarget. 2015 Oct 6;6(30):28539-55. doi: 10.18632/oncotarget.5164.

8.

Functional and pathogenic differences of Th1 and Th17 cells in experimental autoimmune encephalomyelitis.

Domingues HS, Mues M, Lassmann H, Wekerle H, Krishnamoorthy G.

PLoS One. 2010 Nov 29;5(11):e15531. doi: 10.1371/journal.pone.0015531.

9.

miR-20b suppresses Th17 differentiation and the pathogenesis of experimental autoimmune encephalomyelitis by targeting RORγt and STAT3.

Zhu E, Wang X, Zheng B, Wang Q, Hao J, Chen S, Zhao Q, Zhao L, Wu Z, Yin Z.

J Immunol. 2014 Jun 15;192(12):5599-609. doi: 10.4049/jimmunol.1303488. Epub 2014 May 19.

10.

Diverting T helper cell trafficking through increased plasticity attenuates autoimmune encephalomyelitis.

Califano D, Sweeney KJ, Le H, VanValkenburgh J, Yager E, O'Connor W Jr, Kennedy JS, Jones DM, Avram D.

J Clin Invest. 2014 Jan;124(1):174-87. doi: 10.1172/JCI70103. Epub 2013 Dec 9.

11.

Novel IL27p28/IL12p40 cytokine suppressed experimental autoimmune uveitis by inhibiting autoreactive Th1/Th17 cells and promoting expansion of regulatory T cells.

Wang RX, Yu CR, Mahdi RM, Egwuagu CE.

J Biol Chem. 2012 Oct 19;287(43):36012-21. doi: 10.1074/jbc.M112.390625. Epub 2012 Aug 30.

12.

Cutting edge: loss of α4 integrin expression differentially affects the homing of Th1 and Th17 cells.

Glatigny S, Duhen R, Oukka M, Bettelli E.

J Immunol. 2011 Dec 15;187(12):6176-9. doi: 10.4049/jimmunol.1102515. Epub 2011 Nov 14.

13.

The transcription factor STAT3 is required for T helper 2 cell development.

Stritesky GL, Muthukrishnan R, Sehra S, Goswami R, Pham D, Travers J, Nguyen ET, Levy DE, Kaplan MH.

Immunity. 2011 Jan 28;34(1):39-49. doi: 10.1016/j.immuni.2010.12.013. Epub 2011 Jan 6.

14.

Halofuginone-induced amino acid starvation regulates Stat3-dependent Th17 effector function and reduces established autoimmune inflammation.

Carlson TJ, Pellerin A, Djuretic IM, Trivigno C, Koralov SB, Rao A, Sundrud MS.

J Immunol. 2014 Mar 1;192(5):2167-76. doi: 10.4049/jimmunol.1302316. Epub 2014 Jan 31.

15.
16.

STAT3 activates miR-155 in Th17 cells and acts in concert to promote experimental autoimmune uveitis.

Escobar T, Yu CR, Muljo SA, Egwuagu CE.

Invest Ophthalmol Vis Sci. 2013 Jun 10;54(6):4017-25. doi: 10.1167/iovs.13-11937.

17.

RORγt-specific transcriptional interactomic inhibition suppresses autoimmunity associated with TH17 cells.

Park TY, Park SD, Cho JY, Moon JS, Kim NY, Park K, Seong RH, Lee SW, Morio T, Bothwell AL, Lee SK.

Proc Natl Acad Sci U S A. 2014 Dec 30;111(52):18673-8. doi: 10.1073/pnas.1413687112. Epub 2014 Dec 19. Erratum in: Proc Natl Acad Sci U S A. 2015 Apr 21;112(16):E2115.

18.

The transcription factor Twist1 limits T helper 17 and T follicular helper cell development by repressing the gene encoding the interleukin-6 receptor α chain.

Pham D, Walline CC, Hollister K, Dent AL, Blum JS, Firulli AB, Kaplan MH.

J Biol Chem. 2013 Sep 20;288(38):27423-33. doi: 10.1074/jbc.M113.497248. Epub 2013 Aug 9.

19.

Dehydrodiconiferyl alcohol (DHCA) modulates the differentiation of Th17 and Th1 cells and suppresses experimental autoimmune encephalomyelitis.

Lee J, Choi J, Lee W, Ko K, Kim S.

Mol Immunol. 2015 Dec;68(2 Pt B):434-44. doi: 10.1016/j.molimm.2015.09.028. Epub 2015 Oct 23.

PMID:
26477735
20.

B cells promote induction of experimental autoimmune encephalomyelitis by facilitating reactivation of T cells in the central nervous system.

Pierson ER, Stromnes IM, Goverman JM.

J Immunol. 2014 Feb 1;192(3):929-39. doi: 10.4049/jimmunol.1302171. Epub 2013 Dec 23.

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