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Items: 1 to 20 of 103

1.

Dual role of G-runs and hnRNP F in the regulation of a mutation-activated pseudoexon in the fibrinogen gamma-chain transcript.

Rimoldi V, Soldà G, Asselta R, Spena S, Stuani C, Buratti E, Duga S.

PLoS One. 2013;8(3):e59333. doi: 10.1371/journal.pone.0059333.

2.

hnRNP A1 and the SR proteins ASF/SF2 and SC35 have antagonistic functions in splicing of beta-tropomyosin exon 6B.

Expert-Bezançon A, Sureau A, Durosay P, Salesse R, Groeneveld H, Lecaer JP, Marie J.

J Biol Chem. 2004 Sep 10;279(37):38249-59.

PMID:
15208309
3.
4.

Roles for SR proteins and hnRNP A1 in the regulation of c-src exon N1.

Rooke N, Markovtsov V, Cagavi E, Black DL.

Mol Cell Biol. 2003 Mar;23(6):1874-84.

5.
7.

hnRNP F directs formation of an exon 4 minus variant of tumor-associated NADH oxidase (ENOX2).

Tang X, Kane VD, Morré DM, Morré DJ.

Mol Cell Biochem. 2011 Nov;357(1-2):55-63. doi: 10.1007/s11010-011-0875-5.

PMID:
21625959
8.

hnRNP H and hnRNP F complex with Fox2 to silence fibroblast growth factor receptor 2 exon IIIc.

Mauger DM, Lin C, Garcia-Blanco MA.

Mol Cell Biol. 2008 Sep;28(17):5403-19. doi: 10.1128/MCB.00739-08.

9.
10.

Heterogeneous nuclear ribonucleoprotein F/H proteins modulate the alternative splicing of the apoptotic mediator Bcl-x.

Garneau D, Revil T, Fisette JF, Chabot B.

J Biol Chem. 2005 Jun 17;280(24):22641-50.

PMID:
15837790
12.

Multiple splicing defects in an intronic false exon.

Sun H, Chasin LA.

Mol Cell Biol. 2000 Sep;20(17):6414-25.

13.

The prevalent deep intronic c. 639+919 G>A GLA mutation causes pseudoexon activation and Fabry disease by abolishing the binding of hnRNPA1 and hnRNP A2/B1 to a splicing silencer.

Palhais B, Dembic M, Sabaratnam R, Nielsen KS, Doktor TK, Bruun GH, Andresen BS.

Mol Genet Metab. 2016 Nov;119(3):258-269. doi: 10.1016/j.ymgme.2016.08.007.

PMID:
27595546
14.

hnRNP H enhances skipping of a nonfunctional exon P3A in CHRNA1 and a mutation disrupting its binding causes congenital myasthenic syndrome.

Masuda A, Shen XM, Ito M, Matsuura T, Engel AG, Ohno K.

Hum Mol Genet. 2008 Dec 15;17(24):4022-35. doi: 10.1093/hmg/ddn305.

15.

Silencer elements as possible inhibitors of pseudoexon splicing.

Sironi M, Menozzi G, Riva L, Cagliani R, Comi GP, Bresolin N, Giorda R, Pozzoli U.

Nucleic Acids Res. 2004 Mar 19;32(5):1783-91.

16.

A second exon splicing silencer within human immunodeficiency virus type 1 tat exon 2 represses splicing of Tat mRNA and binds protein hnRNP H.

Jacquenet S, Méreau A, Bilodeau PS, Damier L, Stoltzfus CM, Branlant C.

J Biol Chem. 2001 Nov 2;276(44):40464-75.

PMID:
11526107
17.

hnRNP H1 and intronic G runs in the splicing control of the human rpL3 gene.

Russo A, Siciliano G, Catillo M, Giangrande C, Amoresano A, Pucci P, Pietropaolo C, Russo G.

Biochim Biophys Acta. 2010 May-Jun;1799(5-6):419-28. doi: 10.1016/j.bbagrm.2010.01.008.

PMID:
20100605
18.

hnRNP A/B proteins are required for inhibition of HIV-1 pre-mRNA splicing.

Caputi M, Mayeda A, Krainer AR, Zahler AM.

EMBO J. 1999 Jul 15;18(14):4060-7.

20.
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