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Items: 1 to 20 of 112

1.

Structural basis for activation of ZAP-70 by phosphorylation of the SH2-kinase linker.

Yan Q, Barros T, Visperas PR, Deindl S, Kadlecek TA, Weiss A, Kuriyan J.

Mol Cell Biol. 2013 Jun;33(11):2188-201. doi: 10.1128/MCB.01637-12. Epub 2013 Mar 25.

2.
3.

Activation of Zap-70 tyrosine kinase due to a structural rearrangement induced by tyrosine phosphorylation and/or ITAM binding.

Visco C, Magistrelli G, Bosotti R, Perego R, Rusconi L, Toma S, Zamai M, Acuto O, Isacchi A.

Biochemistry. 2000 Mar 14;39(10):2784-91.

PMID:
10704231
4.

Mutation of tyrosines 492/493 in the kinase domain of ZAP-70 affects multiple T-cell receptor signaling pathways.

Mège D, Di Bartolo V, Germain V, Tuosto L, Michel F, Acuto O.

J Biol Chem. 1996 Dec 20;271(51):32644-52.

6.

Modification by covalent reaction or oxidation of cysteine residues in the tandem-SH2 domains of ZAP-70 and Syk can block phosphopeptide binding.

Visperas PR, Winger JA, Horton TM, Shah NH, Aum DJ, Tao A, Barros T, Yan Q, Wilson CG, Arkin MR, Weiss A, Kuriyan J.

Biochem J. 2015 Jan 1;465(1):149-61. doi: 10.1042/BJ20140793.

7.

Antagonists of the Src homology 2 (SH2) domains of Grb2, Src, Lck and ZAP-70.

García-Echeverria C.

Curr Med Chem. 2001 Nov;8(13):1589-604. Review.

PMID:
11562287
8.

Tyrosine 319 in the interdomain B of ZAP-70 is a binding site for the Src homology 2 domain of Lck.

Pelosi M, Di Bartolo V, Mounier V, Mège D, Pascussi JM, Dufour E, Blondel A, Acuto O.

J Biol Chem. 1999 May 14;274(20):14229-37.

9.

A weak Lck tail bite is necessary for Lck function in T cell antigen receptor signaling.

Nika K, Tautz L, Arimura Y, Vang T, Williams S, Mustelin T.

J Biol Chem. 2007 Dec 7;282(49):36000-9. Epub 2007 Sep 26.

10.

Structural and biophysical characterization of the Syk activation switch.

Grädler U, Schwarz D, Dresing V, Musil D, Bomke J, Frech M, Greiner H, Jäkel S, Rysiok T, Müller-Pompalla D, Wegener A.

J Mol Biol. 2013 Jan 23;425(2):309-33. doi: 10.1016/j.jmb.2012.11.007. Epub 2012 Nov 12.

PMID:
23154170
11.

Signaling of the ITK (interleukin 2-inducible T cell kinase)-SYK (spleen tyrosine kinase) fusion kinase is dependent on adapter SLP-76 and on the adapter function of the kinases SYK and ZAP70.

Hussain A, Mohammad DK, Gustafsson MO, Uslu M, Hamasy A, Nore BF, Mohamed AJ, Smith CI.

J Biol Chem. 2013 Mar 8;288(10):7338-50. doi: 10.1074/jbc.M112.374967. Epub 2013 Jan 4.

12.

Structural basis for the inhibition of tyrosine kinase activity of ZAP-70.

Deindl S, Kadlecek TA, Brdicka T, Cao X, Weiss A, Kuriyan J.

Cell. 2007 May 18;129(4):735-46.

13.

Phosphorylation of Tyr319 in ZAP-70 is required for T-cell antigen receptor-dependent phospholipase C-gamma1 and Ras activation.

Williams BL, Irvin BJ, Sutor SL, Chini CC, Yacyshyn E, Bubeck Wardenburg J, Dalton M, Chan AC, Abraham RT.

EMBO J. 1999 Apr 1;18(7):1832-44.

17.

Reconstitution of T cell antigen receptor-induced Erk2 kinase activation in Lck-negative JCaM1 cells by Syk.

Williams S, Couture C, Gilman J, Jascur T, Deckert M, Altman A, Mustelin T.

Eur J Biochem. 1997 Apr 1;245(1):84-90.

18.

The Syk protein tyrosine kinase can function independently of CD45 or Lck in T cell antigen receptor signaling.

Chu DH, Spits H, Peyron JF, Rowley RB, Bolen JB, Weiss A.

EMBO J. 1996 Nov 15;15(22):6251-61.

19.
20.

The N-terminal SH2 domains of Syk and ZAP-70 mediate phosphotyrosine-independent binding to integrin beta cytoplasmic domains.

Woodside DG, Obergfell A, Talapatra A, Calderwood DA, Shattil SJ, Ginsberg MH.

J Biol Chem. 2002 Oct 18;277(42):39401-8. Epub 2002 Aug 8.

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