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MyD88 signaling in CD4 T cells promotes IFN-γ production and hematopoietic progenitor cell expansion in response to intracellular bacterial infection.

Zhang Y, Jones M, McCabe A, Winslow GM, Avram D, MacNamara KC.

J Immunol. 2013 May 1;190(9):4725-35. doi: 10.4049/jimmunol.1203024.


Infection-induced myelopoiesis during intracellular bacterial infection is critically dependent upon IFN-γ signaling.

MacNamara KC, Oduro K, Martin O, Jones DD, McLaughlin M, Choi K, Borjesson DL, Winslow GM.

J Immunol. 2011 Jan 15;186(2):1032-43. doi: 10.4049/jimmunol.1001893.


Cutting edge: bacterial infection induces hematopoietic stem and progenitor cell expansion in the absence of TLR signaling.

Scumpia PO, Kelly-Scumpia KM, Delano MJ, Weinstein JS, Cuenca AG, Al-Quran S, Bovio I, Akira S, Kumagai Y, Moldawer LL.

J Immunol. 2010 Mar 1;184(5):2247-51. doi: 10.4049/jimmunol.0903652.


Staphylococcus aureus recognition by hematopoietic stem and progenitor cells via TLR2/MyD88/PGE2 stimulates granulopoiesis in wounds.

Granick JL, Falahee PC, Dahmubed D, Borjesson DL, Miller LS, Simon SI.

Blood. 2013 Sep 5;122(10):1770-8. doi: 10.1182/blood-2012-11-466268.


Myd88 Initiates Early Innate Immune Responses and Promotes CD4 T Cells during Coronavirus Encephalomyelitis.

Butchi N, Kapil P, Puntambekar S, Stohlman SA, Hinton DR, Bergmann CC.

J Virol. 2015 Sep;89(18):9299-312. doi: 10.1128/JVI.01199-15.


Production of IFN-gamma by CD4 T cells is essential for resolving ehrlichia infection.

Bitsaktsis C, Huntington J, Winslow G.

J Immunol. 2004 Jun 1;172(11):6894-901.


Type I interferons promote severe disease in a mouse model of lethal ehrlichiosis.

Zhang Y, Thai V, McCabe A, Jones M, MacNamara KC.

Infect Immun. 2014 Apr;82(4):1698-709. doi: 10.1128/IAI.01564-13.


MyD88-dependent signaling contributes to host defense against ehrlichial infection.

Koh YS, Koo JE, Biswas A, Kobayashi KS.

PLoS One. 2010 Jul 23;5(7):e11758. doi: 10.1371/journal.pone.0011758.


TLR4 signaling via MyD88 and TRIF differentially shape the CD4+ T cell response to Porphyromonas gingivalis hemagglutinin B.

Gaddis DE, Michalek SM, Katz J.

J Immunol. 2011 May 15;186(10):5772-83. doi: 10.4049/jimmunol.1003192.


T and B cells are not required for clearing Staphylococcus aureus in systemic infection despite a strong TLR2-MyD88-dependent T cell activation.

Schmaler M, Jann NJ, Ferracin F, Landmann R.

J Immunol. 2011 Jan 1;186(1):443-52. doi: 10.4049/jimmunol.1001407.


IFN-γ-induced priming maintains long-term strain-transcending immunity against blood-stage Plasmodium chabaudi malaria.

da Silva HB, de Salles EM, Panatieri RH, Boscardin SB, Rodríguez-Málaga SM, Alvarez JM, D'Império Lima MR.

J Immunol. 2013 Nov 15;191(10):5160-9. doi: 10.4049/jimmunol.1300462.


CD4+ T cell expression of MyD88 is essential for normal resolution of Chlamydia muridarum genital tract infection.

Frazer LC, Sullivan JE, Zurenski MA, Mintus M, Tomasak TE, Prantner D, Nagarajan UM, Darville T.

J Immunol. 2013 Oct 15;191(8):4269-79. doi: 10.4049/jimmunol.1301547.


Transient activation of hematopoietic stem and progenitor cells by IFNγ during acute bacterial infection.

MacNamara KC, Jones M, Martin O, Winslow GM.

PLoS One. 2011;6(12):e28669. doi: 10.1371/journal.pone.0028669.


MyD88-dependent pathway in T cells directly modulates the expansion of colitogenic CD4+ T cells in chronic colitis.

Tomita T, Kanai T, Fujii T, Nemoto Y, Okamoto R, Tsuchiya K, Totsuka T, Sakamoto N, Akira S, Watanabe M.

J Immunol. 2008 Apr 15;180(8):5291-9.


MyD88-mediated instructive signals in dendritic cells regulate pulmonary immune responses during respiratory virus infection.

Rudd BD, Schaller MA, Smit JJ, Kunkel SL, Neupane R, Kelley L, Berlin AA, Lukacs NW.

J Immunol. 2007 May 1;178(9):5820-7.

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