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Items: 1 to 20 of 98

1.

Regulation of IL-17 family members by adrenal hormones during experimental sepsis in mice.

Bosmann M, Meta F, Ruemmler R, Haggadone MD, Sarma JV, Zetoune FS, Ward PA.

Am J Pathol. 2013 Apr;182(4):1124-30. doi: 10.1016/j.ajpath.2013.01.005.

2.

MyD88-dependent production of IL-17F is modulated by the anaphylatoxin C5a via the Akt signaling pathway.

Bosmann M, Patel VR, Russkamp NF, Pache F, Zetoune FS, Sarma JV, Ward PA.

FASEB J. 2011 Dec;25(12):4222-32. doi: 10.1096/fj.11-191205.

3.

Elevated expression of IL-23/IL-17 pathway-related mediators correlates with exacerbation of pulmonary inflammation during polymicrobial sepsis.

Cauvi DM, Williams MR, Bermudez JA, Armijo G, De Maio A.

Shock. 2014 Sep;42(3):246-55. doi: 10.1097/SHK.0000000000000207.

4.

Effects of aging on mortality, hypothermia, and cytokine induction in mice with endotoxemia or sepsis.

Saito H, Sherwood ER, Varma TK, Evers BM.

Mech Ageing Dev. 2003 Dec;124(10-12):1047-58.

PMID:
14659593
5.
6.

Exogenous interleukin-10 fails to decrease the mortality or morbidity of sepsis.

Remick DG, Garg SJ, Newcomb DE, Wollenberg G, Huie TK, Bolgos GL.

Crit Care Med. 1998 May;26(5):895-904.

PMID:
9590320
7.

IL-17A and IL-17F stimulate chemokines via MAPK pathways (ERK1/2 and p38 but not JNK) in mouse cultured mesangial cells: synergy with TNF-alpha and IL-1beta.

Iyoda M, Shibata T, Kawaguchi M, Hizawa N, Yamaoka T, Kokubu F, Akizawa T.

Am J Physiol Renal Physiol. 2010 Mar;298(3):F779-87. doi: 10.1152/ajprenal.00198.2009.

8.

Bone marrow and nonbone marrow Toll like receptor 4 regulate acute hepatic injury induced by endotoxemia.

Hochhauser E, Avlas O, Fallach R, Bachmetov L, Zemel R, Pappo O, Shainberg A, Ben Ari Z.

PLoS One. 2013 Aug 15;8(8):e73041. doi: 10.1371/journal.pone.0073041.

9.
10.

Exogenous glucose administration impairs glucose tolerance and pancreatic insulin secretion during acute sepsis in non-diabetic mice.

Watanabe Y, Singamsetty S, Zou B, Guo L, Stefanovski D, Alonso LC, Garcia-Ocana A, O'Donnell CP, McVerry BJ.

PLoS One. 2013 Jun 24;8(6):e67716. doi: 10.1371/journal.pone.0067716.

11.

STAT3-mediated IL-17 production by postseptic T cells exacerbates viral immunopathology of the lung.

Mukherjee S, Allen RM, Lukacs NW, Kunkel SL, Carson WF 4th.

Shock. 2012 Nov;38(5):515-23. doi: 10.1097/SHK.0b013e31826f862c.

12.

Anti-inflammatory effects of benzenediamine derivate FC-98 on sepsis injury in mice via suppression of JNK, NF-κB and IRF3 signaling pathways.

Song Y, Liu X, Yue H, Ji J, Dou H, Hou Y.

Mol Immunol. 2015 Oct;67(2 Pt B):183-92. doi: 10.1016/j.molimm.2015.05.005.

PMID:
26032013
13.

Modulatory effects of glucocorticoids and catecholamines on human interleukin-12 and interleukin-10 production: clinical implications.

Elenkov IJ, Papanicolaou DA, Wilder RL, Chrousos GP.

Proc Assoc Am Physicians. 1996 Sep;108(5):374-81.

PMID:
8902882
14.

Regulatory role of C5a in LPS-induced IL-6 production by neutrophils during sepsis.

Riedemann NC, Guo RF, Hollmann TJ, Gao H, Neff TA, Reuben JS, Speyer CL, Sarma JV, Wetsel RA, Zetoune FS, Ward PA.

FASEB J. 2004 Feb;18(2):370-2.

15.

Adverse functions of IL-17A in experimental sepsis.

Flierl MA, Rittirsch D, Gao H, Hoesel LM, Nadeau BA, Day DE, Zetoune FS, Sarma JV, Huber-Lang MS, Ferrara JL, Ward PA.

FASEB J. 2008 Jul;22(7):2198-205. doi: 10.1096/fj.07-105221.

16.

ATP-gated P2X1 ion channels protect against endotoxemia by dampening neutrophil activation.

Lecut C, Faccinetto C, Delierneux C, van Oerle R, Spronk HM, Evans RJ, El Benna J, Bours V, Oury C.

J Thromb Haemost. 2012 Mar;10(3):453-65. doi: 10.1111/j.1538-7836.2011.04606.x.

17.

Endotoxemia and sepsis mortality reduction by non-anticoagulant activated protein C.

Kerschen EJ, Fernandez JA, Cooley BC, Yang XV, Sood R, Mosnier LO, Castellino FJ, Mackman N, Griffin JH, Weiler H.

J Exp Med. 2007 Oct 1;204(10):2439-48.

19.

Deficiency of alpha-calcitonin gene-related peptide induces inflammatory responses and lethality in sepsis.

Lee JK, Jung JS, Park SH, Sim YB, Suh HW.

Cytokine. 2013 Nov;64(2):548-54. doi: 10.1016/j.cyto.2013.07.030.

PMID:
24021706
20.

Interaction between the innate and adaptive immune systems is required to survive sepsis and control inflammation after injury.

Shelley O, Murphy T, Paterson H, Mannick JA, Lederer JA.

Shock. 2003 Aug;20(2):123-9.

PMID:
12865655
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