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Items: 1 to 20 of 153

1.

Reduction of cardiomyocyte S-nitrosylation by S-nitrosoglutathione reductase protects against sepsis-induced myocardial depression.

Sips PY, Irie T, Zou L, Shinozaki S, Sakai M, Shimizu N, Nguyen R, Stamler JS, Chao W, Kaneki M, Ichinose F.

Am J Physiol Heart Circ Physiol. 2013 Apr 15;304(8):H1134-46. doi: 10.1152/ajpheart.00887.2012. Epub 2013 Feb 15.

2.

Dynamic denitrosylation via S-nitrosoglutathione reductase regulates cardiovascular function.

Beigi F, Gonzalez DR, Minhas KM, Sun QA, Foster MW, Khan SA, Treuer AV, Dulce RA, Harrison RW, Saraiva RM, Premer C, Schulman IH, Stamler JS, Hare JM.

Proc Natl Acad Sci U S A. 2012 Mar 13;109(11):4314-9. doi: 10.1073/pnas.1113319109. Epub 2012 Feb 24.

3.

Cardiomyocyte-specific overexpression of nitric oxide synthase 3 prevents myocardial dysfunction in murine models of septic shock.

Ichinose F, Buys ES, Neilan TG, Furutani EM, Morgan JG, Jassal DS, Graveline AR, Searles RJ, Lim CC, Kaneki M, Picard MH, Scherrer-Crosbie M, Janssens S, Liao R, Bloch KD.

Circ Res. 2007 Jan 5;100(1):130-9. Epub 2006 Nov 30.

4.

S-Nitrosoglutathione Reductase Deficiency Enhances the Proliferative Expansion of Adult Heart Progenitors and Myocytes Post Myocardial Infarction.

Hatzistergos KE, Paulino EC, Dulce RA, Takeuchi LM, Bellio MA, Kulandavelu S, Cao Y, Balkan W, Kanashiro-Takeuchi RM, Hare JM.

J Am Heart Assoc. 2015 Jul 15;4(7). pii: e001974. doi: 10.1161/JAHA.115.001974.

5.

Proteomic analysis of the role of S-nitrosoglutathione reductase in lipopolysaccharide-challenged mice.

Ozawa K, Tsumoto H, Wei W, Tang CH, Komatsubara AT, Kawafune H, Shimizu K, Liu L, Tsujimoto G.

Proteomics. 2012 Jun;12(12):2024-35. doi: 10.1002/pmic.201100666.

6.

Hypoxia-induced changes in protein s-nitrosylation in female mouse brainstem.

Palmer LA, Kimberly deRonde, Brown-Steinke K, Gunter S, Jyothikumar V, Forbes MS, Lewis SJ.

Am J Respir Cell Mol Biol. 2015 Jan;52(1):37-45. doi: 10.1165/rcmb.2013-0359OC.

7.

[Impact of nitric oxide synthase 3 on myocardial dysfunction in sepsis].

Ichinose F.

Masui. 2008 Mar;57(3):294-301. Review. Japanese.

PMID:
18340997
8.

Toll-like receptor 4, nitric oxide, and myocardial depression in endotoxemia.

Baumgarten G, Knuefermann P, Schuhmacher G, Vervölgyi V, von Rappard J, Dreiner U, Fink K, Djoufack C, Hoeft A, Grohé C, Knowlton AA, Meyer R.

Shock. 2006 Jan;25(1):43-9.

PMID:
16369185
9.

Cardiomyocyte Toll-like receptor 4 is involved in heart dysfunction following septic shock or myocardial ischemia.

Fallach R, Shainberg A, Avlas O, Fainblut M, Chepurko Y, Porat E, Hochhauser E.

J Mol Cell Cardiol. 2010 Jun;48(6):1236-44. doi: 10.1016/j.yjmcc.2010.02.020. Epub 2010 Mar 4.

PMID:
20211628
10.

[Cardiac metallothionein overexpression improves cardiac contractile function and attenuates oxidative stress in lipopolysaccharide-treated mice].

Zhao P, Zhang J, Chu X, Yuan HT, Cui LQ, Zhu XL.

Zhonghua Xin Xue Guan Bing Za Zhi. 2011 Aug;39(8):711-6. Chinese.

PMID:
22169416
11.

Myocardial contractile function and heart rate in mice with myocyte-specific overexpression of endothelial nitric oxide synthase.

Brunner F, Andrew P, Wölkart G, Zechner R, Mayer B.

Circulation. 2001 Dec 18;104(25):3097-102.

12.

Increased susceptibility to Klebsiella pneumonia and mortality in GSNOR-deficient mice.

Tang CH, Seeley EJ, Huang X, Wolters PJ, Liu L.

Biochem Biophys Res Commun. 2013 Dec 6;442(1-2):122-6. doi: 10.1016/j.bbrc.2013.11.028. Epub 2013 Nov 15.

13.

Transnitrosylation: A Factor in Nitric Oxide-Mediated Penile Erection.

Musicki B, Lagoda G, Goetz T, La Favor JD, Burnett AL.

J Sex Med. 2016 May;13(5):808-14. doi: 10.1016/j.jsxm.2016.03.003.

14.

Essential roles of S-nitrosothiols in vascular homeostasis and endotoxic shock.

Liu L, Yan Y, Zeng M, Zhang J, Hanes MA, Ahearn G, McMahon TJ, Dickfeld T, Marshall HE, Que LG, Stamler JS.

Cell. 2004 Feb 20;116(4):617-28.

15.

Cardiac-specific overexpression of catalase attenuates lipopolysaccharide-induced myocardial contractile dysfunction: role of autophagy.

Turdi S, Han X, Huff AF, Roe ND, Hu N, Gao F, Ren J.

Free Radic Biol Med. 2012 Sep 15;53(6):1327-38. doi: 10.1016/j.freeradbiomed.2012.07.084. Epub 2012 Aug 5.

16.

Gene deletion of protein tyrosine phosphatase 1B protects against sepsis-induced cardiovascular dysfunction and mortality.

Coquerel D, Neviere R, Delile E, Mulder P, Marechal X, Montaigne D, Renet S, Remy-Jouet I, Gomez E, Henry JP, do Rego JC, Richard V, Tamion F.

Arterioscler Thromb Vasc Biol. 2014 May;34(5):1032-44. doi: 10.1161/ATVBAHA.114.303450. Epub 2014 Feb 27.

17.

S-nitrosoglutathione reductase deficiency increases mutagenesis from alkylation in mouse liver.

Leung J, Wei W, Liu L.

Carcinogenesis. 2013 May;34(5):984-9. doi: 10.1093/carcin/bgt031. Epub 2013 Jan 25.

18.

CD14-deficient mice are protected against lipopolysaccharide-induced cardiac inflammation and left ventricular dysfunction.

Knuefermann P, Nemoto S, Misra A, Nozaki N, Defreitas G, Goyert SM, Carabello BA, Mann DL, Vallejo JG.

Circulation. 2002 Nov 12;106(20):2608-15.

19.

Overexpression of cardiac I-kappaBalpha prevents endotoxin-induced myocardial dysfunction.

Haudek SB, Spencer E, Bryant DD, White DJ, Maass D, Horton JW, Chen ZJ, Giroir BP.

Am J Physiol Heart Circ Physiol. 2001 Mar;280(3):H962-8.

20.

The alarmin cytokine, high mobility group box 1, is produced by viable cardiomyocytes and mediates the lipopolysaccharide-induced myocardial dysfunction via a TLR4/phosphatidylinositol 3-kinase gamma pathway.

Xu H, Su Z, Wu J, Yang M, Penninger JM, Martin CM, Kvietys PR, Rui T.

J Immunol. 2010 Feb 1;184(3):1492-8. doi: 10.4049/jimmunol.0902660. Epub 2009 Dec 18.

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