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Items: 1 to 20 of 119

1.

Dynamin-2 mediates heart failure by modulating Ca2+ -dependent cardiomyocyte apoptosis.

Li J, Zhang DS, Ye JC, Li CM, Qi M, Liang DD, Xu XR, Xu L, Liu Y, Zhang H, Zhang YY, Deng FF, Feng J, Shi D, Chen JJ, Li L, Chen G, Sun YF, Peng LY, Chen YH.

Int J Cardiol. 2013 Oct 3;168(3):2109-19. doi: 10.1016/j.ijcard.2013.01.006. Epub 2013 Feb 11.

PMID:
23410488
2.

Aberrant dynamin 2-dependent Na(+) /H(+) exchanger-1 trafficking contributes to cardiomyocyte apoptosis.

Li J, Xu L, Ye J, Li X, Zhang D, Liang D, Xu X, Qi M, Li C, Zhang H, Wang J, Liu Y, Zhang Y, Zhou Z, Liang X, Li J, Peng L, Zhu W, Chen YH.

J Cell Mol Med. 2013 Sep;17(9):1119-27. doi: 10.1111/jcmm.12086. Epub 2013 Jul 9.

3.

Role of the calcium-sensing receptor in cardiomyocyte apoptosis via the sarcoplasmic reticulum and mitochondrial death pathway in cardiac hypertrophy and heart failure.

Lu FH, Fu SB, Leng X, Zhang X, Dong S, Zhao YJ, Ren H, Li H, Zhong X, Xu CQ, Zhang WH.

Cell Physiol Biochem. 2013;31(4-5):728-43. doi: 10.1159/000350091. Epub 2013 May 23.

4.

FOXO3a regulates BNIP3 and modulates mitochondrial calcium, dynamics, and function in cardiac stress.

Chaanine AH, Kohlbrenner E, Gamb SI, Guenzel AJ, Klaus K, Fayyaz AU, Nair KS, Hajjar RJ, Redfield MM.

Am J Physiol Heart Circ Physiol. 2016 Dec 1;311(6):H1540-H1559. doi: 10.1152/ajpheart.00549.2016. Epub 2016 Sep 30.

5.

Ultrastructural remodelling of Ca(2+) signalling apparatus in failing heart cells.

Wu HD, Xu M, Li RC, Guo L, Lai YS, Xu SM, Li SF, Lü QL, Li LL, Zhang HB, Zhang YY, Zhang CM, Wang SQ.

Cardiovasc Res. 2012 Sep 1;95(4):430-8. doi: 10.1093/cvr/cvs195. Epub 2012 Jun 15.

6.

Reduction in dynamin-2 is implicated in ischaemic cardiac arrhythmias.

Shi D, Xie D, Zhang H, Zhao H, Huang J, Li C, Liu Y, Lv F, The E, Liu Y, Yuan T, Wang S, Chen J, Pan L, Yu Z, Liang D, Zhu W, Zhang Y, Li L, Peng L, Li J, Chen YH.

J Cell Mol Med. 2014 Oct;18(10):1992-9. doi: 10.1111/jcmm.12335. Epub 2014 Aug 5.

7.

Potential role of BNIP3 in cardiac remodeling, myocardial stiffness, and endoplasmic reticulum: mitochondrial calcium homeostasis in diastolic and systolic heart failure.

Chaanine AH, Gordon RE, Kohlbrenner E, Benard L, Jeong D, Hajjar RJ.

Circ Heart Fail. 2013 May;6(3):572-83. doi: 10.1161/CIRCHEARTFAILURE.112.000200. Epub 2013 Mar 18.

8.

Stage-dependent benefits and risks of pimobendan in mice with genetic dilated cardiomyopathy and progressive heart failure.

Nonaka M, Morimoto S, Murayama T, Kurebayashi N, Li L, Wang YY, Arioka M, Yoshihara T, Takahashi-Yanaga F, Sasaguri T.

Br J Pharmacol. 2015 May;172(9):2369-82. doi: 10.1111/bph.13062. Epub 2015 Mar 17.

9.

Mitochondrial calcium overload is a key determinant in heart failure.

Santulli G, Xie W, Reiken SR, Marks AR.

Proc Natl Acad Sci U S A. 2015 Sep 8;112(36):11389-94. doi: 10.1073/pnas.1513047112. Epub 2015 Jul 27.

10.

Ca2+- and mitochondrial-dependent cardiomyocyte necrosis as a primary mediator of heart failure.

Nakayama H, Chen X, Baines CP, Klevitsky R, Zhang X, Zhang H, Jaleel N, Chua BH, Hewett TE, Robbins J, Houser SR, Molkentin JD.

J Clin Invest. 2007 Sep;117(9):2431-44.

11.

Ca2+/calmodulin-dependent protein kinase modulates cardiac ryanodine receptor phosphorylation and sarcoplasmic reticulum Ca2+ leak in heart failure.

Ai X, Curran JW, Shannon TR, Bers DM, Pogwizd SM.

Circ Res. 2005 Dec 9;97(12):1314-22. Epub 2005 Nov 3.

12.

Ca2+ influx-induced sarcoplasmic reticulum Ca2+ overload causes mitochondrial-dependent apoptosis in ventricular myocytes.

Chen X, Zhang X, Kubo H, Harris DM, Mills GD, Moyer J, Berretta R, Potts ST, Marsh JD, Houser SR.

Circ Res. 2005 Nov 11;97(10):1009-17. Epub 2005 Oct 6.

13.

Ventricular assist device therapy normalizes inducible nitric oxide synthase expression and reduces cardiomyocyte apoptosis in the failing human heart.

Patten RD, Denofrio D, El-Zaru M, Kakkar R, Saunders J, Celestin F, Warner K, Rastegar H, Khabbaz KR, Udelson JE, Konstam MA, Karas RH.

J Am Coll Cardiol. 2005 May 3;45(9):1419-24.

14.

Effects of left ventricular volume overload on mitochondrial and death-receptor-mediated apoptotic pathways in the transition to heart failure.

Moorjani N, Westaby S, Narula J, Catarino PA, Brittin R, Kemp TJ, Narula N, Sugden PH.

Am J Cardiol. 2009 May 1;103(9):1261-8. doi: 10.1016/j.amjcard.2009.01.013.

PMID:
19406269
15.

Altered myocardial Ca2+ cycling after left ventricular assist device support in the failing human heart.

Chaudhary KW, Rossman EI, Piacentino V 3rd, Kenessey A, Weber C, Gaughan JP, Ojamaa K, Klein I, Bers DM, Houser SR, Margulies KB.

J Am Coll Cardiol. 2004 Aug 18;44(4):837-45.

16.

Aconitine-induced Ca2+ overload causes arrhythmia and triggers apoptosis through p38 MAPK signaling pathway in rats.

Sun GB, Sun H, Meng XB, Hu J, Zhang Q, Liu B, Wang M, Xu HB, Sun XB.

Toxicol Appl Pharmacol. 2014 Aug 15;279(1):8-22. doi: 10.1016/j.taap.2014.05.005. Epub 2014 May 17.

PMID:
24840785
17.

Ca2+/calmodulin-dependent protein kinase II and protein kinase A differentially regulate sarcoplasmic reticulum Ca2+ leak in human cardiac pathology.

Fischer TH, Herting J, Tirilomis T, Renner A, Neef S, Toischer K, Ellenberger D, Förster A, Schmitto JD, Gummert J, Schöndube FA, Hasenfuss G, Maier LS, Sossalla S.

Circulation. 2013 Aug 27;128(9):970-81. doi: 10.1161/CIRCULATIONAHA.113.001746. Epub 2013 Jul 19.

18.

SR calcium handling and calcium after-transients in a rabbit model of heart failure.

Baartscheer A, Schumacher CA, Belterman CN, Coronel R, Fiolet JW.

Cardiovasc Res. 2003 Apr 1;58(1):99-108.

PMID:
12667950
19.

Cardiomyocyte Ca2+ handling and structure is regulated by degree and duration of mechanical load variation.

Ibrahim M, Kukadia P, Siedlecka U, Cartledge JE, Navaratnarajah M, Tokar S, Van Doorn C, Tsang VT, Gorelik J, Yacoub MH, Terracciano CM.

J Cell Mol Med. 2012 Dec;16(12):2910-8. doi: 10.1111/j.1582-4934.2012.01611.x.

20.

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