Format
Sort by
Items per page

Send to

Choose Destination

Links from PubMed

Items: 1 to 20 of 101

1.

Model composition through model reduction: a combined model of CD95 and NF-κB signaling pathways.

Kutumova E, Zinovyev A, Sharipov R, Kolpakov F.

BMC Syst Biol. 2013 Feb 15;7:13. doi: 10.1186/1752-0509-7-13.

2.

Dynamics within the CD95 death-inducing signaling complex decide life and death of cells.

Neumann L, Pforr C, Beaudouin J, Pappa A, Fricker N, Krammer PH, Lavrik IN, Eils R.

Mol Syst Biol. 2010;6:352. doi: 10.1038/msb.2010.6. Epub 2010 Mar 9.

3.

Quantification of CD95-induced apoptosis and NF-κB activation at the single cell level.

Schmidt JH, Pietkiewicz S, Naumann M, Lavrik IN.

J Immunol Methods. 2015 Aug;423:12-7. doi: 10.1016/j.jim.2015.04.026. Epub 2015 May 9.

PMID:
25967949
4.

The relevance of NF-kappaB for CD95 signaling in tumor cells.

Legembre P, Barnhart BC, Peter ME.

Cell Cycle. 2004 Oct;3(10):1235-9. Epub 2004 Oct 26.

PMID:
15467462
5.

Mathematical modeling reveals threshold mechanism in CD95-induced apoptosis.

Bentele M, Lavrik I, Ulrich M, Stösser S, Heermann DW, Kalthoff H, Krammer PH, Eils R.

J Cell Biol. 2004 Sep 13;166(6):839-51.

6.
7.

Robust simplifications of multiscale biochemical networks.

Radulescu O, Gorban AN, Zinovyev A, Lilienbaum A.

BMC Syst Biol. 2008 Oct 14;2:86. doi: 10.1186/1752-0509-2-86.

8.

Practical limits for reverse engineering of dynamical systems: a statistical analysis of sensitivity and parameter inferability in systems biology models.

Erguler K, Stumpf MP.

Mol Biosyst. 2011 May;7(5):1593-602. doi: 10.1039/c0mb00107d. Epub 2011 Mar 4.

PMID:
21380410
9.

Parameter identification using stochastic simulations reveals a robustness in CD95 apoptotic response.

Zimmer C, Schleich K, Lavrik I.

Mol Biosyst. 2016 Apr 26;12(5):1486-95. doi: 10.1039/c6mb00028b.

PMID:
27004466
10.

Model reduction of the intracellular-signaling subsystem of apoptosis.

Bykov V, Gol'dshtein V.

Math Biosci. 2016 May;275:39-50. doi: 10.1016/j.mbs.2016.01.007. Epub 2016 Feb 12.

PMID:
26880618
11.

The CD95 (APO-1/Fas) receptor activates NF-kappaB independently of its cytotoxic function.

Ponton A, Clément MV, Stamenkovic I.

J Biol Chem. 1996 Apr 12;271(15):8991-5.

12.

EBV latency III immortalization program sensitizes B cells to induction of CD95-mediated apoptosis via LMP1: role of NF-kappaB, STAT1, and p53.

Le Clorennec C, Youlyouz-Marfak I, Adriaenssens E, Coll J, Bornkamm GW, Feuillard J.

Blood. 2006 Mar 1;107(5):2070-8. Epub 2005 Nov 29.

13.

Induction of apoptosis and activation of NF-kappaB by CD95 require different signalling thresholds.

Legembre P, Barnhart BC, Zheng L, Vijayan S, Straus SE, Puck J, Dale JK, Lenardo M, Peter ME.

EMBO Rep. 2004 Nov;5(11):1084-9.

14.

A simple work flow for biologically inspired model reduction--application to early JAK-STAT signaling.

Quaiser T, Dittrich A, Schaper F, Mönnigmann M.

BMC Syst Biol. 2011 Feb 21;5:30. doi: 10.1186/1752-0509-5-30.

15.

Communicating oscillatory networks: frequency domain analysis.

Ihekwaba AE, Sedwards S.

BMC Syst Biol. 2011 Dec 22;5:203. doi: 10.1186/1752-0509-5-203.

17.

Regulation of Fas (CD95)-induced apoptosis by nuclear factor-kappaB and tumor necrosis factor-alpha in macrophages.

Lu B, Wang L, Medan D, Toledo D, Huang C, Chen F, Shi X, Rojanasakul Y.

Am J Physiol Cell Physiol. 2002 Sep;283(3):C831-8.

18.

Clustering reveals limits of parameter identifiability in multi-parameter models of biochemical dynamics.

Nienałtowski K, Włodarczyk M, Lipniacki T, Komorowski M.

BMC Syst Biol. 2015 Sep 29;9:65. doi: 10.1186/s12918-015-0205-8.

19.

Integrated mechanistic and data-driven modelling for multivariate analysis of signalling pathways.

Hua F, Hautaniemi S, Yokoo R, Lauffenburger DA.

J R Soc Interface. 2006 Aug 22;3(9):515-26.

20.

A novel signaling mechanism for soluble CD95 ligand. Synergy with anti-CD95 monoclonal antibodies for apoptosis and NF-kappaB nuclear translocation.

Xiao S, Jodo S, Sung SS, Marshak-Rothstein A, Ju ST.

J Biol Chem. 2002 Dec 27;277(52):50907-13. Epub 2002 Oct 21.

Supplemental Content

Support Center