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Items: 1 to 20 of 107

1.

Social isolation exacerbates schizophrenia-like phenotypes via oxidative stress in cortical interneurons.

Jiang Z, Rompala GR, Zhang S, Cowell RM, Nakazawa K.

Biol Psychiatry. 2013 May 15;73(10):1024-34. doi: 10.1016/j.biopsych.2012.12.004. Epub 2013 Jan 21.

2.

Postnatal NMDA receptor ablation in corticolimbic interneurons confers schizophrenia-like phenotypes.

Belforte JE, Zsiros V, Sklar ER, Jiang Z, Yu G, Li Y, Quinlan EM, Nakazawa K.

Nat Neurosci. 2010 Jan;13(1):76-83. doi: 10.1038/nn.2447. Epub 2009 Nov 15.

3.

Sepsis-induced selective parvalbumin interneuron phenotype loss and cognitive impairments may be mediated by NADPH oxidase 2 activation in mice.

Ji MH, Qiu LL, Tang H, Ju LS, Sun XR, Zhang H, Jia M, Zuo ZY, Shen JC, Yang JJ.

J Neuroinflammation. 2015 Sep 29;12:182. doi: 10.1186/s12974-015-0401-x.

4.

Behavioral and neurochemical consequences of cortical oxidative stress on parvalbumin-interneuron maturation in rodent models of schizophrenia.

Powell SB, Sejnowski TJ, Behrens MM.

Neuropharmacology. 2012 Mar;62(3):1322-31. doi: 10.1016/j.neuropharm.2011.01.049. Epub 2011 Feb 17. Review.

5.

GABAergic interneuron origin of schizophrenia pathophysiology.

Nakazawa K, Zsiros V, Jiang Z, Nakao K, Kolata S, Zhang S, Belforte JE.

Neuropharmacology. 2012 Mar;62(3):1574-83. doi: 10.1016/j.neuropharm.2011.01.022. Epub 2011 Jan 26. Review.

6.

Involvement of NOX2 in the development of behavioral and pathologic alterations in isolated rats.

Schiavone S, Sorce S, Dubois-Dauphin M, Jaquet V, Colaianna M, Zotti M, Cuomo V, Trabace L, Krause KH.

Biol Psychiatry. 2009 Aug 15;66(4):384-92. doi: 10.1016/j.biopsych.2009.04.033. Epub 2009 Jun 26.

PMID:
19559404
7.

Early-life insults impair parvalbumin interneurons via oxidative stress: reversal by N-acetylcysteine.

Cabungcal JH, Steullet P, Kraftsik R, Cuenod M, Do KQ.

Biol Psychiatry. 2013 Mar 15;73(6):574-82. doi: 10.1016/j.biopsych.2012.09.020. Epub 2012 Nov 7.

PMID:
23140664
8.

Mice lacking the transcriptional coactivator PGC-1α exhibit alterations in inhibitory synaptic transmission in the motor cortex.

Dougherty SE, Bartley AF, Lucas EK, Hablitz JJ, Dobrunz LE, Cowell RM.

Neuroscience. 2014 Jun 20;271:137-48. doi: 10.1016/j.neuroscience.2014.04.023. Epub 2014 Apr 24.

9.

PGC-1α provides a transcriptional framework for synchronous neurotransmitter release from parvalbumin-positive interneurons.

Lucas EK, Dougherty SE, McMeekin LJ, Reid CS, Dobrunz LE, West AB, Hablitz JJ, Cowell RM.

J Neurosci. 2014 Oct 22;34(43):14375-87. doi: 10.1523/JNEUROSCI.1222-14.2014.

10.

Assessment of resveratrol, apocynin and taurine on mechanical-metabolic uncoupling and oxidative stress in a mouse model of duchenne muscular dystrophy: A comparison with the gold standard, α-methyl prednisolone.

Capogrosso RF, Cozzoli A, Mantuano P, Camerino GM, Massari AM, Sblendorio VT, De Bellis M, Tamma R, Giustino A, Nico B, Montagnani M, De Luca A.

Pharmacol Res. 2016 Apr;106:101-113. doi: 10.1016/j.phrs.2016.02.016. Epub 2016 Feb 27.

PMID:
26930420
11.

Cortical parvalbumin GABAergic deficits with α7 nicotinic acetylcholine receptor deletion: implications for schizophrenia.

Lin H, Hsu FC, Baumann BH, Coulter DA, Anderson SA, Lynch DR.

Mol Cell Neurosci. 2014 Jul;61:163-75. doi: 10.1016/j.mcn.2014.06.007. Epub 2014 Jun 28.

12.

Convergence of genetic and environmental factors on parvalbumin-positive interneurons in schizophrenia.

Jiang Z, Cowell RM, Nakazawa K.

Front Behav Neurosci. 2013 Sep 3;7:116. doi: 10.3389/fnbeh.2013.00116. Review.

13.

Contribution of NMDA receptor hypofunction in prefrontal and cortical excitatory neurons to schizophrenia-like phenotypes.

Rompala GR, Zsiros V, Zhang S, Kolata SM, Nakazawa K.

PLoS One. 2013 Apr 16;8(4):e61278. doi: 10.1371/journal.pone.0061278. Print 2013.

14.

Region-specific impairments in parvalbumin interneurons in social isolation-reared mice.

Ueno H, Suemitsu S, Murakami S, Kitamura N, Wani K, Okamoto M, Matsumoto Y, Ishihara T.

Neuroscience. 2017 Sep 17;359:196-208. doi: 10.1016/j.neuroscience.2017.07.016. Epub 2017 Jul 16.

PMID:
28723388
15.

Neuroligins Are Selectively Essential for NMDAR Signaling in Cerebellar Stellate Interneurons.

Zhang B, Südhof TC.

J Neurosci. 2016 Aug 31;36(35):9070-83. doi: 10.1523/JNEUROSCI.1356-16.2016.

16.

Intermittent Hypoxia-Induced Parvalbumin-Immunoreactive Interneurons Loss and Neurobehavioral Impairment is Mediated by NADPH-Oxidase-2.

Yuan L, Wu J, Liu J, Li G, Liang D.

Neurochem Res. 2015 Jun;40(6):1232-42. doi: 10.1007/s11064-015-1586-1. Epub 2015 Apr 25.

PMID:
25911467
17.

Amelioration of oxidative stress-induced phenotype loss of parvalbumin interneurons might contribute to the beneficial effects of environmental enrichment in a rat model of post-traumatic stress disorder.

Sun XR, Zhang H, Zhao HT, Ji MH, Li HH, Wu J, Li KY, Yang JJ.

Behav Brain Res. 2016 Oct 1;312:84-92. doi: 10.1016/j.bbr.2016.06.016. Epub 2016 Jun 11.

PMID:
27297027
18.

Ketamine Administration During the Second Postnatal Week Alters Synaptic Properties of Fast-Spiking Interneurons in the Medial Prefrontal Cortex of Adult Mice.

Jeevakumar V, Kroener S.

Cereb Cortex. 2016 Mar;26(3):1117-29. doi: 10.1093/cercor/bhu293. Epub 2014 Dec 4.

PMID:
25477370
19.

Schizophrenia-Like Dopamine Release Abnormalities in a Mouse Model of NMDA Receptor Hypofunction.

Nakao K, Jeevakumar V, Jiang SZ, Fujita Y, Diaz NB, Pretell Annan CA, Eskow Jaunarajs KL, Hashimoto K, Belforte JE, Nakazawa K.

Schizophr Bull. 2018 Jan 31. doi: 10.1093/schbul/sby003. [Epub ahead of print]

PMID:
29394409

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