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Items: 1 to 20 of 259

1.

Drosophila Trap1 protects against mitochondrial dysfunction in a PINK1/parkin model of Parkinson's disease.

Costa AC, Loh SH, Martins LM.

Cell Death Dis. 2013 Jan 17;4:e467. doi: 10.1038/cddis.2012.205.

2.

TRAP1 rescues PINK1 loss-of-function phenotypes.

Zhang L, Karsten P, Hamm S, Pogson JH, Müller-Rischart AK, Exner N, Haass C, Whitworth AJ, Winklhofer KF, Schulz JB, Voigt A.

Hum Mol Genet. 2013 Jul 15;22(14):2829-41. doi: 10.1093/hmg/ddt132.

3.
4.

The complex I subunit NDUFA10 selectively rescues Drosophila pink1 mutants through a mechanism independent of mitophagy.

Pogson JH, Ivatt RM, Sanchez-Martinez A, Tufi R, Wilson E, Mortiboys H, Whitworth AJ.

PLoS Genet. 2014 Nov 20;10(11):e1004815. doi: 10.1371/journal.pgen.1004815.

5.

Drosophila pink1 is required for mitochondrial function and interacts genetically with parkin.

Clark IE, Dodson MW, Jiang C, Cao JH, Huh JR, Seol JH, Yoo SJ, Hay BA, Guo M.

Nature. 2006 Jun 29;441(7097):1162-6.

PMID:
16672981
6.

The yeast complex I equivalent NADH dehydrogenase rescues pink1 mutants.

Vilain S, Esposito G, Haddad D, Schaap O, Dobreva MP, Vos M, Van Meensel S, Morais VA, De Strooper B, Verstreken P.

PLoS Genet. 2012 Jan;8(1):e1002456. doi: 10.1371/journal.pgen.1002456.

7.

PINK1 as a molecular checkpoint in the maintenance of mitochondrial function and integrity.

Koh H, Chung J.

Mol Cells. 2012 Jul;34(1):7-13. doi: 10.1007/s10059-012-0100-8. Review.

8.

Parkinson's disease-associated kinase PINK1 regulates Miro protein level and axonal transport of mitochondria.

Liu S, Sawada T, Lee S, Yu W, Silverio G, Alapatt P, Millan I, Shen A, Saxton W, Kanao T, Takahashi R, Hattori N, Imai Y, Lu B.

PLoS Genet. 2012;8(3):e1002537. doi: 10.1371/journal.pgen.1002537.

9.

The mitochondrial chaperone protein TRAP1 mitigates α-Synuclein toxicity.

Butler EK, Voigt A, Lutz AK, Toegel JP, Gerhardt E, Karsten P, Falkenburger B, Reinartz A, Winklhofer KF, Schulz JB.

PLoS Genet. 2012 Feb;8(2):e1002488. doi: 10.1371/journal.pgen.1002488.

10.

The loss of PGAM5 suppresses the mitochondrial degeneration caused by inactivation of PINK1 in Drosophila.

Imai Y, Kanao T, Sawada T, Kobayashi Y, Moriwaki Y, Ishida Y, Takeda K, Ichijo H, Lu B, Takahashi R.

PLoS Genet. 2010 Dec 2;6(12):e1001229. doi: 10.1371/journal.pgen.1001229.

11.

PINK1-mediated phosphorylation of Parkin boosts Parkin activity in Drosophila.

Shiba-Fukushima K, Inoshita T, Hattori N, Imai Y.

PLoS Genet. 2014 Jun 5;10(6):e1004391. doi: 10.1371/journal.pgen.1004391.

12.

UCP4A protects against mitochondrial dysfunction and degeneration in pink1/parkin models of Parkinson's disease.

Wu K, Liu J, Zhuang N, Wang T.

FASEB J. 2014 Dec;28(12):5111-21. doi: 10.1096/fj.14-255802.

13.
14.

Mask loss-of-function rescues mitochondrial impairment and muscle degeneration of Drosophila pink1 and parkin mutants.

Zhu M, Li X, Tian X, Wu C.

Hum Mol Genet. 2015 Jun 1;24(11):3272-85. doi: 10.1093/hmg/ddv081.

15.
16.

Glutathione S-transferase omega suppresses the defective phenotypes caused by PINK1 loss-of-function in Drosophila.

Kim K, Yim J.

Biochem Biophys Res Commun. 2013 Aug 9;437(4):615-9. doi: 10.1016/j.bbrc.2013.07.011.

PMID:
23867819
17.

PINK1 protects against oxidative stress by phosphorylating mitochondrial chaperone TRAP1.

Pridgeon JW, Olzmann JA, Chin LS, Li L.

PLoS Biol. 2007 Jul;5(7):e172.

18.

PINK1 and Parkin complementarily protect dopaminergic neurons in vertebrates.

Matsui H, Gavinio R, Asano T, Uemura N, Ito H, Taniguchi Y, Kobayashi Y, Maki T, Shen J, Takeda S, Uemura K, Yamakado H, Takahashi R.

Hum Mol Genet. 2013 Jun 15;22(12):2423-34. doi: 10.1093/hmg/ddt095.

PMID:
23449626
19.

Clueless, a protein required for mitochondrial function, interacts with the PINK1-Parkin complex in Drosophila.

Sen A, Kalvakuri S, Bodmer R, Cox RT.

Dis Model Mech. 2015 Jun;8(6):577-89. doi: 10.1242/dmm.019208.

20.

MUL1 acts in parallel to the PINK1/parkin pathway in regulating mitofusin and compensates for loss of PINK1/parkin.

Yun J, Puri R, Yang H, Lizzio MA, Wu C, Sheng ZH, Guo M.

Elife. 2014 Jun 4;3:e01958. doi: 10.7554/eLife.01958.

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