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Items: 1 to 20 of 115

1.

Angiotensin type 1a receptors in the subfornical organ are required for deoxycorticosterone acetate-salt hypertension.

Hilzendeger AM, Cassell MD, Davis DR, Stauss HM, Mark AL, Grobe JL, Sigmund CD.

Hypertension. 2013 Mar;61(3):716-22. doi: 10.1161/HYPERTENSIONAHA.111.00356. Epub 2012 Dec 24.

2.

Brain ACE2 overexpression reduces DOCA-salt hypertension independently of endoplasmic reticulum stress.

Xia H, de Queiroz TM, Sriramula S, Feng Y, Johnson T, Mungrue IN, Lazartigues E.

Am J Physiol Regul Integr Comp Physiol. 2015 Mar 1;308(5):R370-8. doi: 10.1152/ajpregu.00366.2014. Epub 2014 Dec 17.

3.

Angiotensinergic signaling in the brain mediates metabolic effects of deoxycorticosterone (DOCA)-salt in C57 mice.

Grobe JL, Buehrer BA, Hilzendeger AM, Liu X, Davis DR, Xu D, Sigmund CD.

Hypertension. 2011 Mar;57(3):600-7. doi: 10.1161/HYPERTENSIONAHA.110.165829. Epub 2011 Jan 24.

4.

Brain endoplasmic reticulum stress mechanistically distinguishes the saline-intake and hypertensive response to deoxycorticosterone acetate-salt.

Jo F, Jo H, Hilzendeger AM, Thompson AP, Cassell MD, Rutkowski DT, Davisson RL, Grobe JL, Sigmund CD.

Hypertension. 2015 Jun;65(6):1341-8. doi: 10.1161/HYPERTENSIONAHA.115.05377. Epub 2015 Apr 20.

5.

Role of the NADPH oxidases in the subfornical organ in angiotensin II-induced hypertension.

Lob HE, Schultz D, Marvar PJ, Davisson RL, Harrison DG.

Hypertension. 2013 Feb;61(2):382-7. doi: 10.1161/HYPERTENSIONAHA.111.00546. Epub 2012 Dec 17.

6.

Genetic knockdown of estrogen receptor-alpha in the subfornical organ augments ANG II-induced hypertension in female mice.

Xue B, Zhang Z, Beltz TG, Guo F, Hay M, Johnson AK.

Am J Physiol Regul Integr Comp Physiol. 2015 Mar 15;308(6):R507-16. doi: 10.1152/ajpregu.00406.2014. Epub 2014 Dec 31.

7.

Role of angiotensin II type 1a receptor in renal injury induced by deoxycorticosterone acetate-salt hypertension.

Hisamichi M, Kamijo-Ikemori A, Sugaya T, Ichikawa D, Natsuki T, Hoshino S, Kimura K, Shibagaki Y.

FASEB J. 2017 Jan;31(1):72-84. doi: 10.1096/fj.201600684RR. Epub 2016 Sep 23.

8.

Brain endogenous angiotensin II receptor type 2 (AT2-R) protects against DOCA/salt-induced hypertension in female rats.

Dai SY, Peng W, Zhang YP, Li JD, Shen Y, Sun XF.

J Neuroinflammation. 2015 Mar 8;12:47. doi: 10.1186/s12974-015-0261-4.

9.

Activity of protein kinase C-α within the subfornical organ is necessary for fluid intake in response to brain angiotensin.

Coble JP, Johnson RF, Cassell MD, Johnson AK, Grobe JL, Sigmund CD.

Hypertension. 2014 Jul;64(1):141-8. doi: 10.1161/HYPERTENSIONAHA.114.03461. Epub 2014 Apr 28.

10.

The role of the subfornical organ in angiotensin II-salt hypertension in the rat.

Osborn JW, Hendel MD, Collister JP, Ariza-Guzman PA, Fink GD.

Exp Physiol. 2012 Jan;97(1):80-8. doi: 10.1113/expphysiol.2011.060491. Epub 2011 Oct 3.

11.

Effect of subfornical organ lesion on the development of mineralocorticoid-salt hypertension.

Osborn JW, Jacob F, Hendel M, Collister JP, Clark L, Guzman PA.

Brain Res. 2006 Sep 13;1109(1):74-82. Epub 2006 Jul 21.

PMID:
16859651
12.

Central cardiovascular circuits contribute to the neurovascular dysfunction in angiotensin II hypertension.

Capone C, Faraco G, Peterson JR, Coleman C, Anrather J, Milner TA, Pickel VM, Davisson RL, Iadecola C.

J Neurosci. 2012 Apr 4;32(14):4878-86. doi: 10.1523/JNEUROSCI.6262-11.2012.

13.

Stimulation of angiotensin type 1A receptors on catecholaminergic cells contributes to angiotensin-dependent hypertension.

Jancovski N, Bassi JK, Carter DA, Choong YT, Connelly A, Nguyen TP, Chen D, Lukoshkova EV, Menuet C, Head GA, Allen AM.

Hypertension. 2013 Nov;62(5):866-71. doi: 10.1161/HYPERTENSIONAHA.113.01474. Epub 2013 Sep 3.

14.

Local production of angiotensin II in the subfornical organ causes elevated drinking.

Sakai K, Agassandian K, Morimoto S, Sinnayah P, Cassell MD, Davisson RL, Sigmund CD.

J Clin Invest. 2007 Apr;117(4):1088-95.

15.

The brain subfornical organ mediates leptin-induced increases in renal sympathetic activity but not its metabolic effects.

Young CN, Morgan DA, Butler SD, Mark AL, Davisson RL.

Hypertension. 2013 Mar;61(3):737-44. doi: 10.1161/HYPERTENSIONAHA.111.00405. Epub 2013 Jan 28.

16.

Effects of exercise training on SFO-mediated sympathoexcitation during chronic heart failure.

Llewellyn TL, Sharma NM, Zheng H, Patel KP.

Am J Physiol Heart Circ Physiol. 2014 Jan 1;306(1):H121-31. doi: 10.1152/ajpheart.00534.2013. Epub 2013 Oct 25.

17.

Subfornical organ mediates sympathetic and hemodynamic responses to blood-borne proinflammatory cytokines.

Wei SG, Zhang ZH, Beltz TG, Yu Y, Johnson AK, Felder RB.

Hypertension. 2013 Jul;62(1):118-25. doi: 10.1161/HYPERTENSIONAHA.113.01404. Epub 2013 May 13.

18.

Genetic ablation of angiotensinogen in the subfornical organ of the brain prevents the central angiotensinergic pressor response.

Sinnayah P, Lazartigues E, Sakai K, Sharma RV, Sigmund CD, Davisson RL.

Circ Res. 2006 Nov 10;99(10):1125-31. Epub 2006 Oct 19.

19.

Cerebral microvascular inflammation in DOCA salt-induced hypertension: role of angiotensin II and mitochondrial superoxide.

Rodrigues SF, Granger DN.

J Cereb Blood Flow Metab. 2012 Feb;32(2):368-75. doi: 10.1038/jcbfm.2011.139. Epub 2011 Oct 5.

20.

Angiotensin II type 1a receptors in subfornical organ contribute towards chronic intermittent hypoxia-associated sustained increase in mean arterial pressure.

Saxena A, Little JT, Nedungadi TP, Cunningham JT.

Am J Physiol Heart Circ Physiol. 2015 Mar 1;308(5):H435-46. doi: 10.1152/ajpheart.00747.2014. Epub 2014 Dec 24.

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