Format
Sort by
Items per page

Send to

Choose Destination

Links from PubMed

Items: 1 to 20 of 62

1.

Pillars article: MyD88: an adapter that recruits IRAK to the IL-1 receptor complex. Immunity. 1997. 7: 837-847.

Wesche H, Henzel WJ, Shillinglaw W, Li S, Cao Z.

J Immunol. 2013 Jan 1;190(1):5-15. No abstract available.

2.

MyD88: an adapter that recruits IRAK to the IL-1 receptor complex.

Wesche H, Henzel WJ, Shillinglaw W, Li S, Cao Z.

Immunity. 1997 Dec;7(6):837-47.

3.

Sequential autophosphorylation steps in the interleukin-1 receptor-associated kinase-1 regulate its availability as an adapter in interleukin-1 signaling.

Kollewe C, Mackensen AC, Neumann D, Knop J, Cao P, Li S, Wesche H, Martin MU.

J Biol Chem. 2004 Feb 13;279(7):5227-36. Epub 2003 Nov 18.

4.

MyD88S, a splice variant of MyD88, differentially modulates NF-kappaB- and AP-1-dependent gene expression.

Janssens S, Burns K, Vercammen E, Tschopp J, Beyaert R.

FEBS Lett. 2003 Jul 31;548(1-3):103-7.

5.
6.

Identification of TIFA as an adapter protein that links tumor necrosis factor receptor-associated factor 6 (TRAF6) to interleukin-1 (IL-1) receptor-associated kinase-1 (IRAK-1) in IL-1 receptor signaling.

Takatsuna H, Kato H, Gohda J, Akiyama T, Moriya A, Okamoto Y, Yamagata Y, Otsuka M, Umezawa K, Semba K, Inoue J.

J Biol Chem. 2003 Apr 4;278(14):12144-50. Epub 2003 Feb 3.

7.

Mal and MyD88: adapter proteins involved in signal transduction by Toll-like receptors.

O'Neill LA, Dunne A, Edjeback M, Gray P, Jefferies C, Wietek C.

J Endotoxin Res. 2003;9(1):55-9. Review.

PMID:
12691620
8.

IRAK (Pelle) family member IRAK-2 and MyD88 as proximal mediators of IL-1 signaling.

Muzio M, Ni J, Feng P, Dixit VM.

Science. 1997 Nov 28;278(5343):1612-5.

9.

Mal (MyD88-adapter-like) is required for Toll-like receptor-4 signal transduction.

Fitzgerald KA, Palsson-McDermott EM, Bowie AG, Jefferies CA, Mansell AS, Brady G, Brint E, Dunne A, Gray P, Harte MT, McMurray D, Smith DE, Sims JE, Bird TA, O'Neill LA.

Nature. 2001 Sep 6;413(6851):78-83.

PMID:
11544529
11.

Contribution of globular death domains and unstructured linkers to MyD88.IRAK-4 heterodimer formation: an explanation for the antagonistic activity of MyD88s.

Mendoza-Barberá E, Corral-Rodríguez MA, Soares-Schanoski A, Velarde M, Macieira S, Messerschmidt A, López-Collazo E, Fuentes-Prior P.

Biochem Biophys Res Commun. 2009 Feb 27;380(1):183-7. doi: 10.1016/j.bbrc.2009.01.069. Epub 2009 Jan 22.

PMID:
19167362
12.
13.

MyD88, an adapter protein involved in interleukin-1 signaling.

Burns K, Martinon F, Esslinger C, Pahl H, Schneider P, Bodmer JL, Di Marco F, French L, Tschopp J.

J Biol Chem. 1998 May 15;273(20):12203-9.

14.

MyD88-induced downregulation of IRAK-4 and its structural requirements.

Hatao F, Yamamoto M, Muroi M, Kaminishi M, Tanamoto K.

FEMS Immunol Med Microbiol. 2008 Jul;53(2):260-4. doi: 10.1111/j.1574-695X.2008.00425.x. Epub 2008 May 21.

15.

Phosphoinositide 3 kinase mediates Toll-like receptor 4-induced activation of NF-kappa B in endothelial cells.

Li X, Tupper JC, Bannerman DD, Winn RK, Rhodes CJ, Harlan JM.

Infect Immun. 2003 Aug;71(8):4414-20.

16.

IRAK-4 as the central TIR signaling mediator in innate immunity.

Suzuki N, Suzuki S, Yeh WC.

Trends Immunol. 2002 Oct;23(10):503-6. Review.

PMID:
12297423
17.
18.

Severe impairment of interleukin-1 and Toll-like receptor signalling in mice lacking IRAK-4.

Suzuki N, Suzuki S, Duncan GS, Millar DG, Wada T, Mirtsos C, Takada H, Wakeham A, Itie A, Li S, Penninger JM, Wesche H, Ohashi PS, Mak TW, Yeh WC.

Nature. 2002 Apr 18;416(6882):750-6. Epub 2002 Mar 31.

PMID:
11923871
19.

TRAF6 distinctively mediates MyD88- and IRAK-1-induced activation of NF-kappaB.

Muroi M, Tanamoto K.

J Leukoc Biol. 2008 Mar;83(3):702-7. Epub 2007 Dec 10.

PMID:
18070982
20.

Tollip, a new component of the IL-1RI pathway, links IRAK to the IL-1 receptor.

Burns K, Clatworthy J, Martin L, Martinon F, Plumpton C, Maschera B, Lewis A, Ray K, Tschopp J, Volpe F.

Nat Cell Biol. 2000 Jun;2(6):346-51.

PMID:
10854325

Supplemental Content

Support Center