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Items: 1 to 20 of 102


Disruption of the transcription factor RBP-J results in osteopenia attributable to attenuated osteoclast differentiation.

Ma J, Liu YL, Hu YY, Wei YN, Zhao XC, Dong GY, Qin HY, Ding Y, Han H.

Mol Biol Rep. 2013 Mar;40(3):2097-105. doi: 10.1007/s11033-012-2268-6. Epub 2012 Dec 7.


TNF-induced osteoclastogenesis and inflammatory bone resorption are inhibited by transcription factor RBP-J.

Zhao B, Grimes SN, Li S, Hu X, Ivashkiv LB.

J Exp Med. 2012 Feb 13;209(2):319-34. doi: 10.1084/jem.20111566. Epub 2012 Jan 16.


The Blimp1-Bcl6 axis is critical to regulate osteoclast differentiation and bone homeostasis.

Miyauchi Y, Ninomiya K, Miyamoto H, Sakamoto A, Iwasaki R, Hoshi H, Miyamoto K, Hao W, Yoshida S, Morioka H, Chiba K, Kato S, Tokuhisa T, Saitou M, Toyama Y, Suda T, Miyamoto T.

J Exp Med. 2010 Apr 12;207(4):751-62. doi: 10.1084/jem.20091957. Epub 2010 Apr 5.


Myeloid-specific disruption of recombination signal binding protein Jκ ameliorates hepatic fibrosis by attenuating inflammation through cylindromatosis in mice.

He F, Guo FC, Li Z, Yu HC, Ma PF, Zhao JL, Feng L, Li WN, Liu XW, Qin HY, Dou KF, Han H.

Hepatology. 2015 Jan;61(1):303-14. doi: 10.1002/hep.27394. Epub 2014 Nov 24.


Notch-RBP-J signaling regulates the mobilization and function of endothelial progenitor cells by dynamic modulation of CXCR4 expression in mice.

Wang L, Wang YC, Hu XB, Zhang BF, Dou GR, He F, Gao F, Feng F, Liang YM, Dou KF, Han H.

PLoS One. 2009 Oct 27;4(10):e7572. doi: 10.1371/journal.pone.0007572.


Knockout of TRPV6 causes osteopenia in mice by increasing osteoclastic differentiation and activity.

Chen F, Ni B, Yang YO, Ye T, Chen A.

Cell Physiol Biochem. 2014;33(3):796-809. doi: 10.1159/000358653. Epub 2014 Mar 21.


RBP-J imposes a requirement for ITAM-mediated costimulation of osteoclastogenesis.

Li S, Miller CH, Giannopoulou E, Hu X, Ivashkiv LB, Zhao B.

J Clin Invest. 2014 Nov;124(11):5057-73. doi: 10.1172/JCI71882. Epub 2014 Oct 20.


Identifying the relative contributions of Rac1 and Rac2 to osteoclastogenesis.

Wang Y, Lebowitz D, Sun C, Thang H, Grynpas MD, Glogauer M.

J Bone Miner Res. 2008 Feb;23(2):260-70.


Saurolactam inhibits osteoclast differentiation and stimulates apoptosis of mature osteoclasts.

Kim MH, Ryu SY, Choi JS, Min YK, Kim SH.

J Cell Physiol. 2009 Dec;221(3):618-28. doi: 10.1002/jcp.21892.


Deletion of CD74, a putative MIF receptor, in mice enhances osteoclastogenesis and decreases bone mass.

Mun SH, Won HY, Hernandez P, Aguila HL, Lee SK.

J Bone Miner Res. 2013 Apr;28(4):948-59. doi: 10.1002/jbmr.1787.


Myeloid-Specific Blockade of Notch Signaling by RBP-J Knockout Attenuates Spinal Cord Injury Accompanied by Compromised Inflammation Response in Mice.

Chen BY, Zheng MH, Chen Y, Du YL, Sun XL, Zhang X, Duan L, Gao F, Liang L, Qin HY, Luo ZJ, Han H.

Mol Neurobiol. 2015 Dec;52(3):1378-90. doi: 10.1007/s12035-014-8934-z. Epub 2014 Oct 26.


Coenzyme q10 regulates osteoclast and osteoblast differentiation.

Moon HJ, Ko WK, Jung MS, Kim JH, Lee WJ, Park KS, Heo JK, Bang JB, Kwon IK.

J Food Sci. 2013 May;78(5):H785-891. doi: 10.1111/1750-3841.12116. Epub 2013 Apr 12.


Notch target gene deregulation and maintenance of the leukemogenic phenotype do not require RBP-J kappa in Ikaros null mice.

Chari S, Umetsu SE, Winandy S.

J Immunol. 2010 Jul 1;185(1):410-7. doi: 10.4049/jimmunol.0903688. Epub 2010 May 28.


The transcription factor RBP-J is essential for retinal cell differentiation and lamination.

Zheng MH, Shi M, Pei Z, Gao F, Han H, Ding YQ.

Mol Brain. 2009 Dec 18;2:38. doi: 10.1186/1756-6606-2-38.


Inducible gene knockout of transcription factor recombination signal binding protein-J reveals its essential role in T versus B lineage decision.

Han H, Tanigaki K, Yamamoto N, Kuroda K, Yoshimoto M, Nakahata T, Ikuta K, Honjo T.

Int Immunol. 2002 Jun;14(6):637-45.


Deletion of RBP-J in dendritic cells compromises TLR-mediated DC activation accompanied by abnormal cytoskeleton reorganization.

Chen YR, Feng F, Wang L, Qu SY, Zhang ZQ, Liu L, Qin HY, Liang YM, Han H.

Mol Biol Rep. 2013 Feb;40(2):1531-9. doi: 10.1007/s11033-012-2198-3. Epub 2012 Nov 9.


Genetic evidence that intestinal Notch functions vary regionally and operate through a common mechanism of Math1 repression.

Kim TH, Shivdasani RA.

J Biol Chem. 2011 Apr 1;286(13):11427-33. doi: 10.1074/jbc.M110.188797. Epub 2011 Jan 31.


Inhibition of Notch/RBP-J signaling induces hair cell formation in neonate mouse cochleas.

Yamamoto N, Tanigaki K, Tsuji M, Yabe D, Ito J, Honjo T.

J Mol Med (Berl). 2006 Jan;84(1):37-45. Epub 2005 Nov 8.


Notch-RBP-J signaling is required by bone marrow stromal cells for the treatment of acute graft versus host disease.

Wang YC, Wang SH, Wei YN, Du DW, Xu H, Gao CC, Zheng MH, Xie J, Li JC, Dong GY, Li L, Xiao Y, Han H.

Stem Cell Res. 2013 Sep;11(2):721-35. doi: 10.1016/j.scr.2013.04.009. Epub 2013 Apr 30.

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