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Important role of PLC-γ1 in hypoxic increase in intracellular calcium in pulmonary arterial smooth muscle cells.

Yadav VR, Song T, Joseph L, Mei L, Zheng YM, Wang YX.

Am J Physiol Lung Cell Mol Physiol. 2013 Feb 1;304(3):L143-51. doi: 10.1152/ajplung.00310.2012. Epub 2012 Nov 30.


Hypoxia-induced mitogenic factor/FIZZ1 induces intracellular calcium release through the PLC-IP(3) pathway.

Fan C, Su Q, Li Y, Liang L, Angelini DJ, Guggino WB, Johns RA.

Am J Physiol Lung Cell Mol Physiol. 2009 Aug;297(2):L263-70. doi: 10.1152/ajplung.90416.2008. Epub 2009 May 8.


PLCγ1-PKCε-IP3R1 signaling plays an important role in hypoxia-induced calcium response in pulmonary artery smooth muscle cells.

Yadav VR, Song T, Mei L, Joseph L, Zheng YM, Wang YX.

Am J Physiol Lung Cell Mol Physiol. 2018 May 1;314(5):L724-L735. doi: 10.1152/ajplung.00243.2017. Epub 2018 Feb 1.


Primary role of mitochondrial Rieske iron-sulfur protein in hypoxic ROS production in pulmonary artery myocytes.

Korde AS, Yadav VR, Zheng YM, Wang YX.

Free Radic Biol Med. 2011 Apr 15;50(8):945-52. doi: 10.1016/j.freeradbiomed.2011.01.010. Epub 2011 Jan 14.


Cross Talk Between Mitochondrial Reactive Oxygen Species and Sarcoplasmic Reticulum Calcium in Pulmonary Arterial Smooth Muscle Cells.

Song T, Zheng YM, Wang YX.

Adv Exp Med Biol. 2017;967:289-298. doi: 10.1007/978-3-319-63245-2_17. Review.


Type 1 inositol 1,4,5-trisphosphate receptors mediate UTP-induced cation currents, Ca2+ signals, and vasoconstriction in cerebral arteries.

Zhao G, Adebiyi A, Blaskova E, Xi Q, Jaggar JH.

Am J Physiol Cell Physiol. 2008 Nov;295(5):C1376-84. doi: 10.1152/ajpcell.00362.2008. Epub 2008 Sep 17. Erratum in: Am J Physiol Cell Physiol. 2009 Jul;297(1):C226.


Roles of different mitochondrial electron transport chain complexes in hypoxia-induced pulmonary vasoconstriction.

Yang Z, Zhuan B, Yan Y, Jiang S, Wang T.

Cell Biol Int. 2016 Feb;40(2):188-95. doi: 10.1002/cbin.10550. Epub 2015 Dec 3.


Superoxide generated at mitochondrial complex III triggers acute responses to hypoxia in the pulmonary circulation.

Waypa GB, Marks JD, Guzy RD, Mungai PT, Schriewer JM, Dokic D, Ball MK, Schumacker PT.

Am J Respir Crit Care Med. 2013 Feb 15;187(4):424-32. doi: 10.1164/rccm.201207-1294OC. Epub 2013 Jan 17.


Ca2+ responses of pulmonary arterial myocytes to acute hypoxia require release from ryanodine and inositol trisphosphate receptors in sarcoplasmic reticulum.

Wang J, Shimoda LA, Sylvester JT.

Am J Physiol Lung Cell Mol Physiol. 2012 Jul;303(2):L161-8. doi: 10.1152/ajplung.00348.2011. Epub 2012 May 11.


Hypoxia activates NADPH oxidase to increase [ROS]i and [Ca2+]i through the mitochondrial ROS-PKCepsilon signaling axis in pulmonary artery smooth muscle cells.

Rathore R, Zheng YM, Niu CF, Liu QH, Korde A, Ho YS, Wang YX.

Free Radic Biol Med. 2008 Nov 1;45(9):1223-31. doi: 10.1016/j.freeradbiomed.2008.06.012. Epub 2008 Jun 21.


Role of mitochondrial reactive oxygen species in hypoxia-dependent increase in intracellular calcium in pulmonary artery myocytes.

Wang QS, Zheng YM, Dong L, Ho YS, Guo Z, Wang YX.

Free Radic Biol Med. 2007 Mar 1;42(5):642-53. Epub 2006 Dec 14.


Function and expression of ryanodine receptors and inositol 1,4,5-trisphosphate receptors in smooth muscle cells of murine feed arteries and arterioles.

Westcott EB, Goodwin EL, Segal SS, Jackson WF.

J Physiol. 2012 Apr 15;590(8):1849-69. doi: 10.1113/jphysiol.2011.222083. Epub 2012 Feb 13.


Increases in mitochondrial reactive oxygen species trigger hypoxia-induced calcium responses in pulmonary artery smooth muscle cells.

Waypa GB, Guzy R, Mungai PT, Mack MM, Marks JD, Roe MW, Schumacker PT.

Circ Res. 2006 Oct 27;99(9):970-8. Epub 2006 Sep 28.


Role of FKBP12.6 in hypoxia- and norepinephrine-induced Ca2+ release and contraction in pulmonary artery myocytes.

Zheng YM, Mei QB, Wang QS, Abdullaev I, Lai FA, Xin HB, Kotlikoff MI, Wang YX.

Cell Calcium. 2004 Apr;35(4):345-55.


IP3 constricts cerebral arteries via IP3 receptor-mediated TRPC3 channel activation and independently of sarcoplasmic reticulum Ca2+ release.

Xi Q, Adebiyi A, Zhao G, Chapman KE, Waters CM, Hassid A, Jaggar JH.

Circ Res. 2008 May 9;102(9):1118-26. doi: 10.1161/CIRCRESAHA.108.173948. Epub 2008 Apr 3. Erratum in: Circ Res. 2009 Jul 2;105(1):e1.


Mitochondrial hyperpolarization in pulmonary vascular remodeling. Mitochondrial uncoupling protein deficiency as disease model.

Pak O, Sommer N, Hoeres T, Bakr A, Waisbrod S, Sydykov A, Haag D, Esfandiary A, Kojonazarov B, Veit F, Fuchs B, Weisel FC, Hecker M, Schermuly RT, Grimminger F, Ghofrani HA, Seeger W, Weissmann N.

Am J Respir Cell Mol Biol. 2013 Sep;49(3):358-67. doi: 10.1165/rcmb.2012-0361OC.


Sildenafil inhibits hypoxia-induced transient receptor potential canonical protein expression in pulmonary arterial smooth muscle via cGMP-PKG-PPARγ axis.

Wang J, Yang K, Xu L, Zhang Y, Lai N, Jiang H, Zhang Y, Zhong N, Ran P, Lu W.

Am J Respir Cell Mol Biol. 2013 Aug;49(2):231-40. doi: 10.1165/rcmb.2012-0185OC.


Cell culture alters Ca2+ entry pathways activated by store-depletion or hypoxia in canine pulmonary arterial smooth muscle cells.

Ng LC, Kyle BD, Lennox AR, Shen XM, Hatton WJ, Hume JR.

Am J Physiol Cell Physiol. 2008 Jan;294(1):C313-23. Epub 2007 Oct 31.

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