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Items: 1 to 20 of 124

1.

The DNA replication factor RFC1 is required for interference-sensitive meiotic crossovers in Arabidopsis thaliana.

Wang Y, Cheng Z, Huang J, Shi Q, Hong Y, Copenhaver GP, Gong Z, Ma H.

PLoS Genet. 2012;8(11):e1003039. doi: 10.1371/journal.pgen.1003039. Epub 2012 Nov 8.

2.

Formation of interference-sensitive meiotic cross-overs requires sufficient DNA leading-strand elongation.

Huang J, Cheng Z, Wang C, Hong Y, Su H, Wang J, Copenhaver GP, Ma H, Wang Y.

Proc Natl Acad Sci U S A. 2015 Oct 6;112(40):12534-9. doi: 10.1073/pnas.1507165112. Epub 2015 Sep 21.

3.

Replication factor C1 (RFC1) is required for double-strand break repair during meiotic homologous recombination in Arabidopsis.

Liu Y, Deng Y, Li G, Zhao J.

Plant J. 2013 Jan;73(1):154-65. doi: 10.1111/tpj.12024. Epub 2012 Nov 8.

4.

The Fanconi anemia ortholog FANCM ensures ordered homologous recombination in both somatic and meiotic cells in Arabidopsis.

Knoll A, Higgins JD, Seeliger K, Reha SJ, Dangel NJ, Bauknecht M, Schröpfer S, Franklin FC, Puchta H.

Plant Cell. 2012 Apr;24(4):1448-64. doi: 10.1105/tpc.112.096644. Epub 2012 Apr 30.

5.

Multiple mechanisms limit meiotic crossovers: TOP3α and two BLM homologs antagonize crossovers in parallel to FANCM.

Séguéla-Arnaud M, Crismani W, Larchevêque C, Mazel J, Froger N, Choinard S, Lemhemdi A, Macaisne N, Van Leene J, Gevaert K, De Jaeger G, Chelysheva L, Mercier R.

Proc Natl Acad Sci U S A. 2015 Apr 14;112(15):4713-8. doi: 10.1073/pnas.1423107112. Epub 2015 Mar 30.

6.

MCM8 is required for a pathway of meiotic double-strand break repair independent of DMC1 in Arabidopsis thaliana.

Crismani W, Portemer V, Froger N, Chelysheva L, Horlow C, Vrielynck N, Mercier R.

PLoS Genet. 2013;9(1):e1003165. doi: 10.1371/journal.pgen.1003165. Epub 2013 Jan 3.

7.

Arabidopsis PTD is required for type I crossover formation and affects recombination frequency in two different chromosomal regions.

Lu P, Wijeratne AJ, Wang Z, Copenhaver GP, Ma H.

J Genet Genomics. 2014 Mar 20;41(3):165-75. doi: 10.1016/j.jgg.2014.02.001. Epub 2014 Feb 22.

PMID:
24656236
8.

Alignment of Homologous Chromosomes and Effective Repair of Programmed DNA Double-Strand Breaks during Mouse Meiosis Require the Minichromosome Maintenance Domain Containing 2 (MCMDC2) Protein.

Finsterbusch F, Ravindranathan R, Dereli I, Stanzione M, Tränkner D, Tóth A.

PLoS Genet. 2016 Oct 19;12(10):e1006393. doi: 10.1371/journal.pgen.1006393. eCollection 2016 Oct.

9.

FANCM limits meiotic crossovers.

Crismani W, Girard C, Froger N, Pradillo M, Santos JL, Chelysheva L, Copenhaver GP, Horlow C, Mercier R.

Science. 2012 Jun 22;336(6088):1588-90. doi: 10.1126/science.1220381.

10.

Two meiotic crossover classes cohabit in Arabidopsis: one is dependent on MER3,whereas the other one is not.

Mercier R, Jolivet S, Vezon D, Huppe E, Chelysheva L, Giovanni M, Nogué F, Doutriaux MP, Horlow C, Grelon M, Mézard C.

Curr Biol. 2005 Apr 26;15(8):692-701.

11.

DNA methylation epigenetically silences crossover hot spots and controls chromosomal domains of meiotic recombination in Arabidopsis.

Yelina NE, Lambing C, Hardcastle TJ, Zhao X, Santos B, Henderson IR.

Genes Dev. 2015 Oct 15;29(20):2183-202. doi: 10.1101/gad.270876.115.

12.

Meiotic crossover control by concerted action of Rad51-Dmc1 in homolog template bias and robust homeostatic regulation.

Lao JP, Cloud V, Huang CC, Grubb J, Thacker D, Lee CY, Dresser ME, Hunter N, Bishop DK.

PLoS Genet. 2013;9(12):e1003978. doi: 10.1371/journal.pgen.1003978. Epub 2013 Dec 19.

14.

The fission yeast FANCM ortholog directs non-crossover recombination during meiosis.

Lorenz A, Osman F, Sun W, Nandi S, Steinacher R, Whitby MC.

Science. 2012 Jun 22;336(6088):1585-8. doi: 10.1126/science.1220111.

15.

Analysis of the Relationships between DNA Double-Strand Breaks, Synaptonemal Complex and Crossovers Using the Atfas1-4 Mutant.

Varas J, Sánchez-Morán E, Copenhaver GP, Santos JL, Pradillo M.

PLoS Genet. 2015 Jul 6;11(7):e1005301. doi: 10.1371/journal.pgen.1005301. eCollection 2015 Jul.

16.

COM-1 promotes homologous recombination during Caenorhabditis elegans meiosis by antagonizing Ku-mediated non-homologous end joining.

Lemmens BB, Johnson NM, Tijsterman M.

PLoS Genet. 2013;9(2):e1003276. doi: 10.1371/journal.pgen.1003276. Epub 2013 Feb 7.

17.

Joint molecule resolution requires the redundant activities of MUS-81 and XPF-1 during Caenorhabditis elegans meiosis.

O'Neil NJ, Martin JS, Youds JL, Ward JD, Petalcorin MI, Rose AM, Boulton SJ.

PLoS Genet. 2013;9(7):e1003582. doi: 10.1371/journal.pgen.1003582. Epub 2013 Jul 18.

18.

The Arabidopsis BLAP75/Rmi1 homologue plays crucial roles in meiotic double-strand break repair.

Chelysheva L, Vezon D, Belcram K, Gendrot G, Grelon M.

PLoS Genet. 2008 Dec;4(12):e1000309. doi: 10.1371/journal.pgen.1000309. Epub 2008 Dec 19.

19.

Inter-homolog crossing-over and synapsis in Arabidopsis meiosis are dependent on the chromosome axis protein AtASY3.

Ferdous M, Higgins JD, Osman K, Lambing C, Roitinger E, Mechtler K, Armstrong SJ, Perry R, Pradillo M, Cuñado N, Franklin FC.

PLoS Genet. 2012 Feb;8(2):e1002507. doi: 10.1371/journal.pgen.1002507. Epub 2012 Feb 2.

20.

RAD5A, RECQ4A, and MUS81 have specific functions in homologous recombination and define different pathways of DNA repair in Arabidopsis thaliana.

Mannuss A, Dukowic-Schulze S, Suer S, Hartung F, Pacher M, Puchta H.

Plant Cell. 2010 Oct;22(10):3318-30. doi: 10.1105/tpc.110.078568. Epub 2010 Oct 22.

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