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The role of innate immune signals in immunity to Brucella abortus.

Gomes MT, Campos PC, de Almeida LA, Oliveira FS, Costa MM, Marim FM, Pereira GS, Oliveira SC.

Front Cell Infect Microbiol. 2012 Oct 25;2:130. doi: 10.3389/fcimb.2012.00130. eCollection 2012. Review.


The role of innate immune receptors in the control of Brucella abortus infection: toll-like receptors and beyond.

Oliveira SC, de Oliveira FS, Macedo GC, de Almeida LA, Carvalho NB.

Microbes Infect. 2008 Jul;10(9):1005-9. doi: 10.1016/j.micinf.2008.07.005. Epub 2008 Jul 10. Review.


Interleukin-1 receptor-associated kinase 4 is essential for initial host control of Brucella abortus infection.

Oliveira FS, Carvalho NB, Brandão AP, Gomes MT, de Almeida LA, Oliveira SC.

Infect Immun. 2011 Nov;79(11):4688-95. doi: 10.1128/IAI.05289-11. Epub 2011 Aug 15.


Brucella abortus DNA is a major bacterial agonist to activate the host innate immune system.

Campos PC, Gomes MT, Guimarães G, Costa Franco MM, Marim FM, Oliveira SC.

Microbes Infect. 2014 Dec;16(12):979-84. doi: 10.1016/j.micinf.2014.08.010. Epub 2014 Aug 27. Review.


Update on the role of innate immune receptors during Brucella abortus infection.

Oliveira SC, de Almeida LA, Carvalho NB, Oliveira FS, Lacerda TL.

Vet Immunol Immunopathol. 2012 Jul 15;148(1-2):129-35. doi: 10.1016/j.vetimm.2011.05.036. Epub 2011 Jun 6. Review.


Toll-like receptor 6 plays an important role in host innate resistance to Brucella abortus infection in mice.

de Almeida LA, Macedo GC, Marinho FA, Gomes MT, Corsetti PP, Silva AM, Cassataro J, Giambartolomei GH, Oliveira SC.

Infect Immun. 2013 May;81(5):1654-62. doi: 10.1128/IAI.01356-12. Epub 2013 Mar 4.


Critical role of ASC inflammasomes and bacterial type IV secretion system in caspase-1 activation and host innate resistance to Brucella abortus infection.

Gomes MT, Campos PC, Oliveira FS, Corsetti PP, Bortoluci KR, Cunha LD, Zamboni DS, Oliveira SC.

J Immunol. 2013 Apr 1;190(7):3629-38. doi: 10.4049/jimmunol.1202817. Epub 2013 Mar 4.


Brucella abortus induces Irgm3 and Irga6 expression via type-I IFN by a MyD88-dependent pathway, without the requirement of TLR2, TLR4, TLR5 and TLR9.

Lapaque N, Muller A, Alexopoulou L, Howard JC, Gorvel JP.

Microb Pathog. 2009 Dec;47(6):299-304. doi: 10.1016/j.micpath.2009.09.005. Epub 2009 Sep 9.


NLRP12 negatively regulates proinflammatory cytokine production and host defense against Brucella abortus.

Silveira TN, Gomes MT, Oliveira LS, Campos PC, Machado GG, Oliveira SC.

Eur J Immunol. 2017 Jan;47(1):51-59. doi: 10.1002/eji.201646502. Epub 2016 Dec 5.


Central role of MyD88-dependent dendritic cell maturation and proinflammatory cytokine production to control Brucella abortus infection.

Macedo GC, Magnani DM, Carvalho NB, Bruna-Romero O, Gazzinelli RT, Oliveira SC.

J Immunol. 2008 Jan 15;180(2):1080-7.


Murine and bovine γδ T cells enhance innate immunity against Brucella abortus infections.

Skyberg JA, Thornburg T, Rollins M, Huarte E, Jutila MA, Pascual DW.

PLoS One. 2011;6(7):e21978. doi: 10.1371/journal.pone.0021978. Epub 2011 Jul 12.


Role of Toll-like receptor 4 in induction of cell-mediated immunity and resistance to Brucella abortus infection in mice.

Campos MA, Rosinha GM, Almeida IC, Salgueiro XS, Jarvis BW, Splitter GA, Qureshi N, Bruna-Romero O, Gazzinelli RT, Oliveira SC.

Infect Immun. 2004 Jan;72(1):176-86.


5-Lipoxygenase negatively regulates Th1 response during Brucella abortus infection in mice.

Fahel JS, de Souza MB, Gomes MT, Corsetti PP, Carvalho NB, Marinho FA, de Almeida LA, Caliari MV, Machado FS, Oliveira SC.

Infect Immun. 2015 Mar;83(3):1210-6. doi: 10.1128/IAI.02592-14. Epub 2015 Jan 12.


TLR2 signaling subpathways regulate TLR9 signaling for the effective induction of IL-12 upon stimulation by heat-killed Brucella abortus.

Zhang CY, Bai N, Zhang ZH, Liang N, Dong L, Xiang R, Liu CH.

Cell Mol Immunol. 2012 Jul;9(4):324-33. doi: 10.1038/cmi.2012.11. Epub 2012 May 28.


MyD88 and STING signaling pathways are required for IRF3-mediated IFN-β induction in response to Brucella abortus infection.

de Almeida LA, Carvalho NB, Oliveira FS, Lacerda TL, Vasconcelos AC, Nogueira L, Bafica A, Silva AM, Oliveira SC.

PLoS One. 2011;6(8):e23135. doi: 10.1371/journal.pone.0023135. Epub 2011 Aug 2.


TLR9 is required for MAPK/NF-κB activation but does not cooperate with TLR2 or TLR6 to induce host resistance to Brucella abortus.

Gomes MT, Campos PC, Pereira Gde S, Bartholomeu DC, Splitter G, Oliveira SC.

J Leukoc Biol. 2016 May;99(5):771-80. doi: 10.1189/jlb.4A0815-346R. Epub 2015 Nov 17.


Heat-killed Brucella abortus induces TNF and IL-12p40 by distinct MyD88-dependent pathways: TNF, unlike IL-12p40 secretion, is Toll-like receptor 2 dependent.

Huang LY, Aliberti J, Leifer CA, Segal DM, Sher A, Golenbock DT, Golding B.

J Immunol. 2003 Aug 1;171(3):1441-6.


MyD88, but not toll-like receptors 4 and 2, is required for efficient clearance of Brucella abortus.

Weiss DS, Takeda K, Akira S, Zychlinsky A, Moreno E.

Infect Immun. 2005 Aug;73(8):5137-43.


Characterization of recombinant B. abortus strain RB51SOD toward understanding the uncorrelated innate and adaptive immune responses induced by RB51SOD compared to its parent vaccine strain RB51.

Zhu J, Larson CB, Ramaker MA, Quandt K, Wendte JM, Ku KP, Chen F, Jourdian GW, Vemulapalli R, Schurig GG, He Y.

Front Cell Infect Microbiol. 2011 Nov 25;1:10. doi: 10.3389/fcimb.2011.00010. eCollection 2011.

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