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Items: 1 to 20 of 130

1.

Myofibrillar Ca(2+) sensitivity is uncoupled from troponin I phosphorylation in hypertrophic obstructive cardiomyopathy due to abnormal troponin T.

Bayliss CR, Jacques AM, Leung MC, Ward DG, Redwood CS, Gallon CE, Copeland O, McKenna WJ, Dos Remedios C, Marston SB, Messer AE.

Cardiovasc Res. 2013 Mar 1;97(3):500-8. doi: 10.1093/cvr/cvs322. Epub 2012 Oct 24.

2.

Familial dilated cardiomyopathy mutations uncouple troponin I phosphorylation from changes in myofibrillar Ca²⁺ sensitivity.

Memo M, Leung MC, Ward DG, dos Remedios C, Morimoto S, Zhang L, Ravenscroft G, McNamara E, Nowak KJ, Marston SB, Messer AE.

Cardiovasc Res. 2013 Jul 1;99(1):65-73. doi: 10.1093/cvr/cvt071. Epub 2013 Mar 27.

4.

Uncoupling of myofilament Ca2+ sensitivity from troponin I phosphorylation by mutations can be reversed by epigallocatechin-3-gallate.

Papadaki M, Vikhorev PG, Marston SB, Messer AE.

Cardiovasc Res. 2015 Oct 1;108(1):99-110. doi: 10.1093/cvr/cvv181. Epub 2015 Jun 24.

6.

Desensitization of myofilaments to Ca2+ as a therapeutic target for hypertrophic cardiomyopathy with mutations in thin filament proteins.

Alves ML, Dias FA, Gaffin RD, Simon JN, Montminy EM, Biesiadecki BJ, Hinken AC, Warren CM, Utter MS, Davis RT 3rd, Sadayappan S, Robbins J, Wieczorek DF, Solaro RJ, Wolska BM.

Circ Cardiovasc Genet. 2014 Apr;7(2):132-43. doi: 10.1161/CIRCGENETICS.113.000324. Epub 2014 Feb 28.

7.

Dephosphorylation specificities of protein phosphatase for cardiac troponin I, troponin T, and sites within troponin T.

Jideama NM, Crawford BH, Hussain AK, Raynor RL.

Int J Biol Sci. 2006;2(1):1-9. Epub 2006 Mar 1.

8.

Functional analysis of a unique troponin c mutation, GLY159ASP, that causes familial dilated cardiomyopathy, studied in explanted heart muscle.

Dyer EC, Jacques AM, Hoskins AC, Ward DG, Gallon CE, Messer AE, Kaski JP, Burch M, Kentish JC, Marston SB.

Circ Heart Fail. 2009 Sep;2(5):456-64. doi: 10.1161/CIRCHEARTFAILURE.108.818237. Epub 2009 Jul 6.

10.

Functional changes in troponin T by a splice donor site mutation that causes hypertrophic cardiomyopathy.

Nakaura H, Morimoto S, Yanaga F, Nakata M, Nishi H, Imaizumi T, Ohtsuki I.

Am J Physiol. 1999 Aug;277(2 Pt 1):C225-32.

11.

Perturbed length-dependent activation in human hypertrophic cardiomyopathy with missense sarcomeric gene mutations.

Sequeira V, Wijnker PJ, Nijenkamp LL, Kuster DW, Najafi A, Witjas-Paalberends ER, Regan JA, Boontje N, Ten Cate FJ, Germans T, Carrier L, Sadayappan S, van Slegtenhorst MA, Zaremba R, Foster DB, Murphy AM, Poggesi C, Dos Remedios C, Stienen GJ, Ho CY, Michels M, van der Velden J.

Circ Res. 2013 May 24;112(11):1491-505. doi: 10.1161/CIRCRESAHA.111.300436. Epub 2013 Mar 18. Erratum in: Circ Res. 2013 Sep 27;113(8):e87.

12.

Gene transfer of troponin I isoforms, mutants, and chimeras.

Westfall MV, Metzger JM.

Adv Exp Med Biol. 2003;538:169-74; discussion 174.

PMID:
15098664
13.

Increased Ca2+-sensitivity of the contractile apparatus in end-stage human heart failure results from altered phosphorylation of contractile proteins.

van der Velden J, Papp Z, Zaremba R, Boontje NM, de Jong JW, Owen VJ, Burton PB, Goldmann P, Jaquet K, Stienen GJ.

Cardiovasc Res. 2003 Jan;57(1):37-47.

14.

Sexually dimorphic myofilament function and cardiac troponin I phosphospecies distribution in hypertrophic cardiomyopathy mice.

McKee LA, Chen H, Regan JA, Behunin SM, Walker JW, Walker JS, Konhilas JP.

Arch Biochem Biophys. 2013 Jul 1;535(1):39-48. doi: 10.1016/j.abb.2012.12.023. Epub 2013 Jan 23.

15.

Altered phosphorylation and calcium sensitivity of cardiac myofibrillar proteins during sepsis.

Wu LL, Tang C, Liu MS.

Am J Physiol Regul Integr Comp Physiol. 2001 Aug;281(2):R408-16.

16.

Mutations in troponin T associated with Hypertrophic Cardiomyopathy increase Ca(2+)-sensitivity and suppress the modulation of Ca(2+)-sensitivity by troponin I phosphorylation.

Messer AE, Bayliss CR, El-Mezgueldi M, Redwood CS, Ward DG, Leung MC, Papadaki M, Dos Remedios C, Marston SB.

Arch Biochem Biophys. 2016 Jul 1;601:113-20. doi: 10.1016/j.abb.2016.03.027. Epub 2016 Mar 29.

17.

Cardiac troponin I tyrosine 26 phosphorylation decreases myofilament Ca2+ sensitivity and accelerates deactivation.

Salhi HE, Walton SD, Hassel NC, Brundage EA, de Tombe PP, Janssen PM, Davis JP, Biesiadecki BJ.

J Mol Cell Cardiol. 2014 Nov;76:257-64. doi: 10.1016/j.yjmcc.2014.09.013. Epub 2014 Sep 22.

18.

Length-dependent activation is modulated by cardiac troponin I bisphosphorylation at Ser23 and Ser24 but not by Thr143 phosphorylation.

Wijnker PJ, Sequeira V, Foster DB, Li Y, Dos Remedios CG, Murphy AM, Stienen GJ, van der Velden J.

Am J Physiol Heart Circ Physiol. 2014 Apr 15;306(8):H1171-81. doi: 10.1152/ajpheart.00580.2013. Epub 2014 Feb 28.

19.

The dilated cardiomyopathy-causing mutation ACTC E361G in cardiac muscle myofibrils specifically abolishes modulation of Ca(2+) regulation by phosphorylation of troponin I.

Vikhorev PG, Song W, Wilkinson R, Copeland O, Messer AE, Ferenczi MA, Marston SB.

Biophys J. 2014 Nov 18;107(10):2369-80. doi: 10.1016/j.bpj.2014.10.024.

20.
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