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After GWAS: mice to the rescue?

Ermann J, Glimcher LH.

Curr Opin Immunol. 2012 Oct;24(5):564-70. doi: 10.1016/j.coi.2012.09.005. Epub 2012 Sep 29. Review.


What have we learned from six years of GWAS in autoimmune diseases, and what is next?

Hu X, Daly M.

Curr Opin Immunol. 2012 Oct;24(5):571-5. doi: 10.1016/j.coi.2012.09.001. Epub 2012 Sep 24. Review.


Congenic mice: cutting tools for complex immune disorders.

Rogner UC, Avner P.

Nat Rev Immunol. 2003 Mar;3(3):243-52. Review.


Autoimmunity: insights from human genomics.

Raychaudhuri S, Rich SS.

Curr Opin Immunol. 2012 Oct;24(5):513-5. doi: 10.1016/j.coi.2012.09.006. Epub 2012 Oct 1. No abstract available.


Influence of genetic background on genetically engineered mouse phenotypes.

Doetschman T.

Methods Mol Biol. 2009;530:423-33. doi: 10.1007/978-1-59745-471-1_23. Review.


The genetics of autoimmune diseases: a networked perspective.

Baranzini SE.

Curr Opin Immunol. 2009 Dec;21(6):596-605. doi: 10.1016/j.coi.2009.09.014. Epub 2009 Nov 5. Review.


Coordinating GWAS results with gene expression in a systems immunologic paradigm in autoimmunity.

Stranger BE, De Jager PL.

Curr Opin Immunol. 2012 Oct;24(5):544-51. doi: 10.1016/j.coi.2012.09.002. Epub 2012 Oct 3. Review.


Mouse BMD quantitative trait loci show improved concordance with human genome-wide association loci when recalculated on a new, common mouse genetic map.

Ackert-Bicknell CL, Karasik D, Li Q, Smith RV, Hsu YH, Churchill GA, Paigen BJ, Tsaih SW.

J Bone Miner Res. 2010 Aug;25(8):1808-20. doi: 10.1002/jbmr.72.


The genetics of human autoimmune disease: A perspective on progress in the field and future directions.

Seldin MF.

J Autoimmun. 2015 Nov;64:1-12. doi: 10.1016/j.jaut.2015.08.015. Epub 2015 Sep 4. Review.


Alcohol Dependence Genetics: Lessons Learned From Genome-Wide Association Studies (GWAS) and Post-GWAS Analyses.

Hart AB, Kranzler HR.

Alcohol Clin Exp Res. 2015 Aug;39(8):1312-27. doi: 10.1111/acer.12792. Epub 2015 Jun 25. Review.


Pharmacogenomics discovery and implementation in genome-wide association studies era.

Ni X, Zhang W, Huang RS.

Wiley Interdiscip Rev Syst Biol Med. 2013 Jan-Feb;5(1):1-9. doi: 10.1002/wsbm.1199. Epub 2012 Nov 27. Review.


Genetic determinants of autoimmune disease and coronary vasculitis in the MRL-lpr/lpr mouse model of systemic lupus erythematosus.

Gu L, Weinreb A, Wang XP, Zack DJ, Qiao JH, Weisbart R, Lusis AJ.

J Immunol. 1998 Dec 15;161(12):6999-7006.


Using epigenetic mechanisms to understand the impact of common disease causing alleles.

Leung A, Schones DE, Natarajan R.

Curr Opin Immunol. 2012 Oct;24(5):558-63. doi: 10.1016/j.coi.2012.07.004. Epub 2012 Aug 2. Review.


After the GWAS rush: nuggets of insight into the pathogenesis of autoimmune disease.

Danska JS, Poussier P.

Semin Immunol. 2009 Dec;21(6):313-7. doi: 10.1016/j.smim.2009.10.007. Epub 2009 Nov 18. No abstract available.


From genome-wide association studies to disease mechanisms: celiac disease as a model for autoimmune diseases.

Kumar V, Wijmenga C, Withoff S.

Semin Immunopathol. 2012 Jul;34(4):567-80. doi: 10.1007/s00281-012-0312-1. Epub 2012 May 14. Review.


Aod1 controlling day 3 thymectomy-induced autoimmune ovarian dysgenesis in mice encompasses two linked quantitative trait loci with opposing allelic effects on disease susceptibility.

Roper RJ, McAllister RD, Biggins JE, Michael SD, Min SH, Tung KS, Call SB, Gao J, Teuscher C.

J Immunol. 2003 Jun 15;170(12):5886-91.


Extreme-phenotype genome-wide association study (XP-GWAS): a method for identifying trait-associated variants by sequencing pools of individuals selected from a diversity panel.

Yang J, Jiang H, Yeh CT, Yu J, Jeddeloh JA, Nettleton D, Schnable PS.

Plant J. 2015 Nov;84(3):587-96. doi: 10.1111/tpj.13029.


Approaches to Investigating Complex Genetic Traits in a Large-Scale Inbred Mouse Aging Study.

Sundberg JP, Berndt A, Sundberg BA, Silva KA, Kennedy V, Smith RS, Cooper TK, Schofield PN.

Vet Pathol. 2016 Mar;53(2):456-67. doi: 10.1177/0300985815612556.


Identifying multiple causative genes at a single GWAS locus.

Flister MJ, Tsaih SW, O'Meara CC, Endres B, Hoffman MJ, Geurts AM, Dwinell MR, Lazar J, Jacob HJ, Moreno C.

Genome Res. 2013 Dec;23(12):1996-2002. doi: 10.1101/gr.160283.113. Epub 2013 Sep 4.


Using gene expression to investigate the genetic basis of complex disorders.

Nica AC, Dermitzakis ET.

Hum Mol Genet. 2008 Oct 15;17(R2):R129-34. doi: 10.1093/hmg/ddn285. Review.

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