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Items: 1 to 20 of 83

1.

[Remodeling of the cardiovascular system and development of chronic kidney disease in patients with metabolic syndrome and obesity: role of eNOS, subunit p22-phox of NADPH-oxidase and MTHFR genes].

Saginova EA, Galliamov MG, Balatskiĭ AV, Kolotvin AV, Severova MV, Samokhodskaia LM, Fomin VV, Krasnova TN, Mukhin NA.

Ter Arkh. 2012;84(6):26-31. Russian.

PMID:
22997914
2.

[Prognostic value of allelic variants affecting the hemostatic system in the development of antiphospholipid syndrome and kidney lesion in patients with systemic lupus erythematosus].

Borisov EN, Krasnova TN, Samohodskaia LM, Ivanitskiĭ LV, Nikiforova NV, Mukhin NA.

Ter Arkh. 2014;86(6):57-62. Russian.

PMID:
25095657
3.

Lack of association of eNOS (G894T) and p22phox NADPH oxidase subunit (C242T) polymorphisms with systemic sclerosis in a cohort of French Caucasian patients.

Allanore Y, Borderie D, Lemaréchal H, Ekindjian OG, Kahan A.

Clin Chim Acta. 2004 Dec;350(1-2):51-5.

PMID:
15530459
4.

Endothelial nitric oxide synthetase, methylenetetrahydrofolate reductase polymorphisms, and cardiovascular complications in Tunisian patients with nondiabetic renal disease.

Kerkeni M, Letaief A, Achour A, Miled A, Trivin F, Maaroufi K.

Clin Biochem. 2009 Jul;42(10-11):958-64. doi: 10.1016/j.clinbiochem.2009.04.005.

PMID:
19376104
5.

Interaction of eNOS polymorphism with MTHFR variants increase the risk of diabetic nephropathy and its progression in type 2 diabetes mellitus patients.

Jafari Y, Rahimi Z, Vaisi-Raygani A, Rezaei M.

Mol Cell Biochem. 2011 Jul;353(1-2):23-34. doi: 10.1007/s11010-011-0770-0.

PMID:
21380725
6.

Endothelial nitric oxide synthase and methylenetetrahydrofolate reductase gene polymorphisms are associated with endothelial dysfunction in young, healthy men.

Imamura A, Okumura K, Matsui H, Mizuno T, Ogawa Y, Imai H, Numaguchi Y, Sakai K, Murohara T.

Can J Cardiol. 2004 Oct;20(12):1229-34.

PMID:
15494775
7.

The C242T polymorphism of the p22-phox gene (CYBA) is associated with higher left ventricular mass in Brazilian hypertensive patients.

Schreiber R, Ferreira-Sae MC, Ronchi JA, Pio-Magalhães JA, Cipolli JA, Matos-Souza JR, Mill JG, Vercesi AE, Krieger JE, Franchini KG, Pereira AC, Nadruz Junior W.

BMC Med Genet. 2011 Aug 31;12:114. doi: 10.1186/1471-2350-12-114.

9.

[Significance of the factors of hypoxia and endothelial dysfunction in kidney injury in the presence of obesity].

Galiamov MG, Saginova EA, Severova MM, Samokhodskaia LM, Krasnova TN, Sholomova VI, Sorokin IuD, Mukhin NA.

Ter Arkh. 2013;85(6):31-7. Russian.

PMID:
23866596
10.

[Endothelial dysfunction gene polymorphisms and the rate of liver fibrosis in chronic hepatitis C].

Taratina OV, Krasnova TN, Samokhodskaia LM, Lopatkina TN, Tkachuk VA, Mukhina NA.

Ter Arkh. 2014;86(4):45-51. Russian.

PMID:
24864467
11.

Overweight and obese humans demonstrate increased vascular endothelial NAD(P)H oxidase-p47(phox) expression and evidence of endothelial oxidative stress.

Silver AE, Beske SD, Christou DD, Donato AJ, Moreau KL, Eskurza I, Gates PE, Seals DR.

Circulation. 2007 Feb 6;115(5):627-37.

12.

The 894G > T (Glu298Asp) variant in the endothelial NOS gene and MTHFR polymorphisms influence homocysteine levels in patients with cognitive decline.

Ferlazzo N, Gorgone G, Caccamo D, Currò M, Condello S, Pisani F, Vernieri F, Rossini PM, Ientile R.

Neuromolecular Med. 2011 Sep;13(3):167-74. doi: 10.1007/s12017-011-8148-8.

PMID:
21607713
13.
14.

MTHFR C677T, FII G20210A, FV Leiden G1691A, NOS3 intron 4 VNTR, and APOE epsilon4 gene polymorphisms are not associated with spontaneous cervical artery dissection.

Jara-Prado A, Alonso ME, Martínez Ruano L, Guerrero Camacho J, Leyva A, López M, Gutierrez-Castrellon P, Arauz A.

Int J Stroke. 2010 Apr;5(2):80-5. doi: 10.1111/j.1747-4949.2010.00412.x.

PMID:
20446941
15.

The impact of eNOS, MTR and MTHFR polymorphisms on renal graft survival in children and young adults.

Artifoni L, Benetti E, Centi S, Negrisolo S, Ghiggeri GM, Ginevri F, Ghio L, Edefonti A, Brambilla C, Cagni N, Murer L.

Nephrol Dial Transplant. 2009 Sep;24(9):2931-7. doi: 10.1093/ndt/gfp161.

PMID:
19349296
16.

Impact of NAD(P)H oxidase p22 phox gene polymorphism on vascular aging in Korean centenarian and nonagenarian.

Kim KI, Na JE, Kang SY, Cho YS, Choi DJ, Kim CH, Kim HS, Oh BH, Choi YH, Kwon IS, Park SC.

Int J Cardiol. 2007 Dec 15;123(1):18-22.

PMID:
17307262
17.

Non-effect of p22-phox -930A/G polymorphism on end-organ damage in Brazilian hypertensive patients.

Sales ML, Ferreira MC, Leme CA Jr, Velloso LA, Gallani MC, Colombo RC, Franchini KG, Nadruz W Jr.

J Hum Hypertens. 2007 Jun;21(6):504-6.

PMID:
17314996
18.

Investigation of association between donors' and recipients' NADPH oxidase p22(phox) C242T polymorphism and acute rejection, delayed graft function and blood pressure in renal allograft recipients.

Mandegary A, Rahmanian-Koshkaki S, Mohammadifar MA, Pourgholi L, Mehdipour M, Etminan A, Ebadzadeh MR, Fazeli F, Azmandian J.

Transpl Immunol. 2015 Jan;32(1):46-50. doi: 10.1016/j.trim.2014.08.004.

PMID:
25173715
19.

Renal insufficiency in non-diabetic subjects: relationship of MTHFR C677t gene polymorphism and left ventricular hypertrophy.

Trovato GM, Catalano D, Ragusa A, Martines GF, Tonzuso A, Pirri C, Buccheri MA, Di Nora C, Trovato FM.

Ren Fail. 2013;35(5):615-23. doi: 10.3109/0886022X.2013.779895.

PMID:
23534584
20.

NADH/NADPH oxidase p22 phox gene polymorphism is associated with improved coronary endothelial vasodilator function.

Schächinger V, Britten MB, Dimmeler S, Zeiher AM.

Eur Heart J. 2001 Jan;22(1):96-101.

PMID:
11133215

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