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Items: 1 to 20 of 122

1.

Helicobacter pylori disrupts host cell membranes, initiating a repair response and cell proliferation.

Lin LL, Huang HC, Ogihara S, Wang JT, Wu MC, McNeil PL, Chen CN, Juan HF.

Int J Mol Sci. 2012;13(8):10176-92. doi: 10.3390/ijms130810176. Epub 2012 Aug 15.

2.

The Helicobacter pylori vacA s1, m1 genotype and cagA is associated with gastric carcinoma in Germany.

Miehlke S, Kirsch C, Agha-Amiri K, Günther T, Lehn N, Malfertheiner P, Stolte M, Ehninger G, Bayerdörffer E.

Int J Cancer. 2000 Aug 1;87(3):322-7.

4.

Clinical relevance of Helicobacter pylori vacA and cagA genotypes in gastric carcinoma.

Ferreira RM, Machado JC, Figueiredo C.

Best Pract Res Clin Gastroenterol. 2014 Dec;28(6):1003-15. doi: 10.1016/j.bpg.2014.09.004. Epub 2014 Oct 2. Review.

PMID:
25439067
5.

Helicobacter pylori infection induces genetic instability of nuclear and mitochondrial DNA in gastric cells.

Machado AM, Figueiredo C, Touati E, Máximo V, Sousa S, Michel V, Carneiro F, Nielsen FC, Seruca R, Rasmussen LJ.

Clin Cancer Res. 2009 May 1;15(9):2995-3002. doi: 10.1158/1078-0432.CCR-08-2686. Epub 2009 Apr 21.

6.

Helicobacter pylori promotes the expression of Krüppel-like factor 5, a mediator of carcinogenesis, in vitro and in vivo.

Noto JM, Khizanishvili T, Chaturvedi R, Piazuelo MB, Romero-Gallo J, Delgado AG, Khurana SS, Sierra JC, Krishna US, Suarez G, Powell AE, Goldenring JR, Coffey RJ, Yang VW, Correa P, Mills JC, Wilson KT, Peek RM Jr.

PLoS One. 2013;8(1):e54344. doi: 10.1371/journal.pone.0054344. Epub 2013 Jan 23.

7.

β-Catenin and p120 mediate PPARδ-dependent proliferation induced by Helicobacter pylori in human and rodent epithelia.

Nagy TA, Wroblewski LE, Wang D, Piazuelo MB, Delgado A, Romero-Gallo J, Noto J, Israel DA, Ogden SR, Correa P, Cover TL, Peek RM Jr.

Gastroenterology. 2011 Aug;141(2):553-64. doi: 10.1053/j.gastro.2011.05.004. Epub 2011 May 17.

8.

Helicobacter pylori counteracts the apoptotic action of its VacA toxin by injecting the CagA protein into gastric epithelial cells.

Oldani A, Cormont M, Hofman V, Chiozzi V, Oregioni O, Canonici A, Sciullo A, Sommi P, Fabbri A, Ricci V, Boquet P.

PLoS Pathog. 2009 Oct;5(10):e1000603. doi: 10.1371/journal.ppat.1000603. Epub 2009 Oct 2.

9.

The number of Helicobacter pylori CagA EPIYA C tyrosine phosphorylation motifs influences the pattern of gastritis and the development of gastric carcinoma.

Ferreira RM, Machado JC, Leite M, Carneiro F, Figueiredo C.

Histopathology. 2012 May;60(6):992-8. doi: 10.1111/j.1365-2559.2012.04190.x. Epub 2012 Feb 20. Erratum in: Histopathology. 2016 Mar;68(4):620.

PMID:
22348604
10.

Regulation of gastric carcinogenesis by Helicobacter pylori virulence factors.

Franco AT, Johnston E, Krishna U, Yamaoka Y, Israel DA, Nagy TA, Wroblewski LE, Piazuelo MB, Correa P, Peek RM Jr.

Cancer Res. 2008 Jan 15;68(2):379-87. doi: 10.1158/0008-5472.CAN-07-0824.

11.

Augmented gp130-mediated cytokine signalling accompanies human gastric cancer progression.

Jackson CB, Judd LM, Menheniott TR, Kronborg I, Dow C, Yeomans ND, Boussioutas A, Robb L, Giraud AS.

J Pathol. 2007 Oct;213(2):140-51.

PMID:
17724739
12.

Higher frequency of cagA EPIYA-C phosphorylation sites in H. pylori strains from first-degree relatives of gastric cancer patients.

Queiroz DM, Silva CI, Goncalves MH, Braga-Neto MB, Fialho AB, Fialho AM, Rocha GA, Rocha AM, Batista SA, Guerrant RL, Lima AA, Braga LL.

BMC Gastroenterol. 2012 Aug 14;12:107. doi: 10.1186/1471-230X-12-107.

13.

Divergence of virulence factors of Helicobacter pylori among clinical isolates does not correlate with disease specificity.

Kodama K, Ito A, Nishizono A, Fujioka T, Nasu M, Yahiro K, Hirayama T, Uemura N.

J Gastroenterol. 1999;34 Suppl 11:6-9.

PMID:
10616758
14.

H. pylori-encoded CagA disrupts tight junctions and induces invasiveness of AGS gastric carcinoma cells via Cdx2-dependent targeting of Claudin-2.

Song X, Chen HX, Wang XY, Deng XY, Xi YX, He Q, Peng TL, Chen J, Chen W, Wong BC, Chen MH.

Cell Immunol. 2013 Nov-Dec;286(1-2):22-30. doi: 10.1016/j.cellimm.2013.10.008. Epub 2013 Nov 9.

PMID:
24287273
15.

The Helicobacter pylori cytotoxin CagA is essential for suppressing host heat shock protein expression.

J Lang B, J Gorrell R, Tafreshi M, Hatakeyama M, Kwok T, T Price J.

Cell Stress Chaperones. 2016 May;21(3):523-33. doi: 10.1007/s12192-016-0680-x. Epub 2016 Mar 1.

16.

Helicobacter pylori infection of gastric cancer cells elevates the level of expression and activation of protein kinase D2.

Shabelnik MY, Kostyuk OV, Merentsev SV, Tarasova TO, Sidorenko SP.

Exp Oncol. 2009 Sep;31(3):134-9.

PMID:
19783969
17.

Helicobacter pylori cagA+ strains and dissociation of gastric epithelial cell proliferation from apoptosis.

Peek RM Jr, Moss SF, Tham KT, Pérez-Pérez GI, Wang S, Miller GG, Atherton JC, Holt PR, Blaser MJ.

J Natl Cancer Inst. 1997 Jun 18;89(12):863-8.

PMID:
9196252
18.

Virulence factors of Helicobacter pylori responsible for gastric diseases in Mongolian gerbil.

Ogura K, Maeda S, Nakao M, Watanabe T, Tada M, Kyutoku T, Yoshida H, Shiratori Y, Omata M.

J Exp Med. 2000 Dec 4;192(11):1601-10.

19.

Relationship between Helicobacter pylori virulence genes and clinical outcomes in Saudi patients.

Marie MA.

J Korean Med Sci. 2012 Feb;27(2):190-3. doi: 10.3346/jkms.2012.27.2.190. Epub 2012 Jan 27.

20.

Clinical and histological associations of cagA and vacA genotypes in Helicobacter pylori gastritis.

Warburton VJ, Everett S, Mapstone NP, Axon AT, Hawkey P, Dixon MF.

J Clin Pathol. 1998 Jan;51(1):55-61.

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