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Items: 1 to 20 of 83


Malaria infection alters the expression of B-cell activating factor resulting in diminished memory antibody responses and survival.

Liu XQ, Stacey KJ, Horne-Debets JM, Cridland JA, Fischer K, Narum D, Mackay F, Pierce SK, Wykes MN.

Eur J Immunol. 2012 Dec;42(12):3291-301. doi: 10.1002/eji.201242689. Epub 2012 Oct 16.


Functional memory B cells and long-lived plasma cells are generated after a single Plasmodium chabaudi infection in mice.

Ndungu FM, Cadman ET, Coulcher J, Nduati E, Couper E, Macdonald DW, Ng D, Langhorne J.

PLoS Pathog. 2009 Dec;5(12):e1000690. doi: 10.1371/journal.ppat.1000690. Epub 2009 Dec 11.


Systemic tumor necrosis factor generated during lethal Plasmodium infections impairs dendritic cell function.

Wykes MN, Liu XQ, Jiang S, Hirunpetcharat C, Good MF.

J Immunol. 2007 Sep 15;179(6):3982-7.


The enigma of memory B cells in malaria.

Njau MN, Jacob J.

Eur J Immunol. 2012 Dec;42(12):3146-9. doi: 10.1002/eji.201243056. Review.


Differential effector pathways regulate memory CD8 T cell immunity against Plasmodium berghei versus P. yoelii sporozoites.

Butler NS, Schmidt NW, Harty JT.

J Immunol. 2010 Mar 1;184(5):2528-38. doi: 10.4049/jimmunol.0903529. Epub 2010 Jan 22.


Dual effect of Plasmodium-infected erythrocytes on dendritic cell maturation.

Bettiol E, Carapau D, Galan-Rodriguez C, Ocaña-Morgner C, Rodriguez A.

Malar J. 2010 Mar 1;9:64. doi: 10.1186/1475-2875-9-64.


Plasmodium yoelii blood-stage antigens newly identified by immunoaffinity using purified IgG antibodies from malaria-resistant mice.

Kamali AN, Marín-García P, Azcárate IG, Diez A, Puyet A, Bautista JM.

Immunobiology. 2012 Aug;217(8):823-30. doi: 10.1016/j.imbio.2012.05.002. Epub 2012 May 11.


Dendritic cells from malaria-infected mice are fully functional APC.

Perry JA, Rush A, Wilson RJ, Olver CS, Avery AC.

J Immunol. 2004 Jan 1;172(1):475-82.


How malaria modulates memory: activation and dysregulation of B cells in Plasmodium infection.

Scholzen A, Sauerwein RW.

Trends Parasitol. 2013 May;29(5):252-62. doi: 10.1016/ Epub 2013 Apr 4. Review.


Deregulation of c-Myc Confers distinct survival requirements for memory B cells, plasma cells, and their progenitors.

Khuda SE, Loo WM, Janz S, Van Ness B, Erickson LD.

J Immunol. 2008 Dec 1;181(11):7537-49.


Plasmodium yoelii can ablate vaccine-induced long-term protection in mice.

Wykes MN, Zhou YH, Liu XQ, Good MF.

J Immunol. 2005 Aug 15;175(4):2510-6.


Dendritic cells induce immunity and long-lasting protection against blood-stage malaria despite an in vitro parasite-induced maturation defect.

Pouniotis DS, Proudfoot O, Bogdanoska V, Apostolopoulos V, Fifis T, Plebanski M.

Infect Immun. 2004 Sep;72(9):5331-9.


Nitric oxide regulates BAFF expression and T cell-independent antibody responses.

Giordano D, Draves KE, Li C, Hohl TM, Clark EA.

J Immunol. 2014 Aug 1;193(3):1110-20. doi: 10.4049/jimmunol.1303158. Epub 2014 Jun 20.


An essential role for C5aR signaling in the optimal induction of a malaria-specific CD4+ T cell response by a whole-killed blood-stage vaccine.

Liu T, Xu G, Guo B, Fu Y, Qiu Y, Ding Y, Zheng H, Fu X, Wu Y, Xu W.

J Immunol. 2013 Jul 1;191(1):178-86. doi: 10.4049/jimmunol.1201190. Epub 2013 May 24.


Cutting edge: the dependence of plasma cells and independence of memory B cells on BAFF and APRIL.

Benson MJ, Dillon SR, Castigli E, Geha RS, Xu S, Lam KP, Noelle RJ.

J Immunol. 2008 Mar 15;180(6):3655-9.


Protective CD8+ T cells induced by malaria sporozoites do not undergo modulation of interleukin-7 receptor expression.

Morrot A, Cockburn IA, Overstreet M, Rodríguez D, Zavala F.

Infect Immun. 2006 Apr;74(4):2495-7.


Outcome of primary lethal and nonlethal Plasmodium yoelii malaria infection in BALB/c and IFN-γ receptor-deficient mice following chloroquine treatment.

Ishih A, Kawakami C, Todoroki A, Hirai H, Ohori K, Kobayashi F.

Parasitol Res. 2013 Feb;112(2):773-80. doi: 10.1007/s00436-012-3197-y. Epub 2012 Nov 21.


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