Format
Sort by
Items per page

Send to

Choose Destination

Links from PubMed

Items: 1 to 20 of 100

1.

PSTPIP2 deficiency in mice causes osteopenia and increased differentiation of multipotent myeloid precursors into osteoclasts.

Chitu V, Nacu V, Charles JF, Henne WM, McMahon HT, Nandi S, Ketchum H, Harris R, Nakamura MC, Stanley ER.

Blood. 2012 Oct 11;120(15):3126-35. doi: 10.1182/blood-2012-04-425595. Epub 2012 Aug 24.

2.

Primed innate immunity leads to autoinflammatory disease in PSTPIP2-deficient cmo mice.

Chitu V, Ferguson PJ, de Bruijn R, Schlueter AJ, Ochoa LA, Waldschmidt TJ, Yeung YG, Stanley ER.

Blood. 2009 Sep 17;114(12):2497-505. doi: 10.1182/blood-2009-02-204925. Epub 2009 Jul 16.

3.

PSTPIP2, a Protein Associated with Autoinflammatory Disease, Interacts with Inhibitory Enzymes SHIP1 and Csk.

Drobek A, Kralova J, Skopcova T, Kucova M, Novák P, Angelisová P, Otahal P, Alberich-Jorda M, Brdicka T.

J Immunol. 2015 Oct 1;195(7):3416-26. doi: 10.4049/jimmunol.1401494. Epub 2015 Aug 24.

4.

Dietary modulation of the microbiome affects autoinflammatory disease.

Lukens JR, Gurung P, Vogel P, Johnson GR, Carter RA, McGoldrick DJ, Bandi SR, Calabrese CR, Vande Walle L, Lamkanfi M, Kanneganti TD.

Nature. 2014 Dec 11;516(7530):246-9. doi: 10.1038/nature13788. Epub 2014 Sep 28.

5.

SH3BP2 cherubism mutation potentiates TNF-α-induced osteoclastogenesis via NFATc1 and TNF-α-mediated inflammatory bone loss.

Mukai T, Ishida S, Ishikawa R, Yoshitaka T, Kittaka M, Gallant R, Lin YL, Rottapel R, Brotto M, Reichenberger EJ, Ueki Y.

J Bone Miner Res. 2014 Dec;29(12):2618-35. doi: 10.1002/jbmr.2295.

6.

Targeted disruption of ephrin B1 in cells of myeloid lineage increases osteoclast differentiation and bone resorption in mice.

Cheng S, Zhao SL, Nelson B, Kesavan C, Qin X, Wergedal J, Mohan S, Xing W.

PLoS One. 2012;7(3):e32887. doi: 10.1371/journal.pone.0032887. Epub 2012 Mar 5.

7.

Increased myeloid cell responses to M-CSF and RANKL cause bone loss and inflammation in SH3BP2 "cherubism" mice.

Ueki Y, Lin CY, Senoo M, Ebihara T, Agata N, Onji M, Saheki Y, Kawai T, Mukherjee PM, Reichenberger E, Olsen BR.

Cell. 2007 Jan 12;128(1):71-83.

8.

Critical role for inflammasome-independent IL-1β production in osteomyelitis.

Lukens JR, Gross JM, Calabrese C, Iwakura Y, Lamkanfi M, Vogel P, Kanneganti TD.

Proc Natl Acad Sci U S A. 2014 Jan 21;111(3):1066-71. doi: 10.1073/pnas.1318688111. Epub 2014 Jan 6.

9.

BAR Proteins PSTPIP1/2 Regulate Podosome Dynamics and the Resorption Activity of Osteoclasts.

Sztacho M, Segeletz S, Sanchez-Fernandez MA, Czupalla C, Niehage C, Hoflack B.

PLoS One. 2016 Oct 19;11(10):e0164829. doi: 10.1371/journal.pone.0164829. eCollection 2016.

10.

Role of IGF-I signaling in regulating osteoclastogenesis.

Wang Y, Nishida S, Elalieh HZ, Long RK, Halloran BP, Bikle DD.

J Bone Miner Res. 2006 Sep;21(9):1350-8.

11.

c-Fms signaling mediates neurofibromatosis Type-1 osteoclast gain-in-functions.

He Y, Rhodes SD, Chen S, Wu X, Yuan J, Yang X, Jiang L, Li X, Takahashi N, Xu M, Mohammad KS, Guise TA, Yang FC.

PLoS One. 2012;7(11):e46900. doi: 10.1371/journal.pone.0046900. Epub 2012 Nov 7.

12.

RARγ is a negative regulator of osteoclastogenesis.

Green AC, Poulton IJ, Vrahnas C, Häusler KD, Walkley CR, Wu JY, Martin TJ, Gillespie MT, Chandraratna RA, Quinn JM, Sims NA, Purton LE.

J Steroid Biochem Mol Biol. 2015 Jun;150:46-53. doi: 10.1016/j.jsbmb.2015.03.005. Epub 2015 Mar 20.

PMID:
25800721
13.

Increased neutrophil infiltration, IL-1 production and a SAPHO syndrome-like phenotype in PSTPIP2-deficient mice.

Liao HJ, Chyuan IT, Wu CS, Lin SW, Chen KH, Tsai HF, Hsu PN.

Rheumatology (Oxford). 2015 Jul;54(7):1317-26. doi: 10.1093/rheumatology/keu481. Epub 2015 Jan 18.

PMID:
25602062
14.

Inflammasome-independent IL-1β mediates autoinflammatory disease in Pstpip2-deficient mice.

Cassel SL, Janczy JR, Bing X, Wilson SP, Olivier AK, Otero JE, Iwakura Y, Shayakhmetov DM, Bassuk AG, Abu-Amer Y, Brogden KA, Burns TL, Sutterwala FS, Ferguson PJ.

Proc Natl Acad Sci U S A. 2014 Jan 21;111(3):1072-7. doi: 10.1073/pnas.1318685111. Epub 2014 Jan 6.

15.

Loss of phosphatase and tensin homolog (PTEN) in myeloid cells controls inflammatory bone destruction by regulating the osteoclastogenic potential of myeloid cells.

Blüml S, Friedrich M, Lohmeyer T, Sahin E, Saferding V, Brunner J, Puchner A, Mandl P, Niederreiter B, Smolen JS, Schabbauer G, Redlich K.

Ann Rheum Dis. 2015 Jan;74(1):227-33. doi: 10.1136/annrheumdis-2013-203486. Epub 2013 Sep 27.

PMID:
24078675
16.

The tyrosine kinase inhibitor GNF-2 suppresses osteoclast formation and activity.

Kim HJ, Yoon HJ, Choi JY, Lee IK, Kim SY.

J Leukoc Biol. 2014 Feb;95(2):337-45. doi: 10.1189/jlb.0713356. Epub 2013 Oct 15.

PMID:
24130113
17.

NLRP3 inflammasome plays a redundant role with caspase 8 to promote IL-1β-mediated osteomyelitis.

Gurung P, Burton A, Kanneganti TD.

Proc Natl Acad Sci U S A. 2016 Apr 19;113(16):4452-7. doi: 10.1073/pnas.1601636113. Epub 2016 Apr 6.

18.
19.

Loss of Cbl-b increases osteoclast bone-resorbing activity and induces osteopenia.

Nakajima A, Sanjay A, Chiusaroli R, Adapala NS, Neff L, Itzsteink C, Horne WC, Baron R.

J Bone Miner Res. 2009 Jul;24(7):1162-72. doi: 10.1359/jbmr.090205.

20.

Low-density lipoprotein receptor deficiency causes impaired osteoclastogenesis and increased bone mass in mice because of defect in osteoclastic cell-cell fusion.

Okayasu M, Nakayachi M, Hayashida C, Ito J, Kaneda T, Masuhara M, Suda N, Sato T, Hakeda Y.

J Biol Chem. 2012 Jun 1;287(23):19229-41. doi: 10.1074/jbc.M111.323600. Epub 2012 Apr 12.

Supplemental Content

Support Center