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Items: 1 to 20 of 140

1.

Age-related changes in core body temperature and activity in triple-transgenic Alzheimer's disease (3xTgAD) mice.

Knight EM, Brown TM, Gümüsgöz S, Smith JC, Waters EJ, Allan SM, Lawrence CB.

Dis Model Mech. 2013 Jan;6(1):160-70. doi: 10.1242/dmm.010173. Epub 2012 Aug 3.

2.

Impaired satiation and increased feeding behaviour in the triple-transgenic Alzheimer's disease mouse model.

Adebakin A, Bradley J, Gümüsgöz S, Waters EJ, Lawrence CB.

PLoS One. 2012;7(10):e45179. doi: 10.1371/journal.pone.0045179. Epub 2012 Oct 4.

3.

Intermittent fasting and caloric restriction ameliorate age-related behavioral deficits in the triple-transgenic mouse model of Alzheimer's disease.

Halagappa VK, Guo Z, Pearson M, Matsuoka Y, Cutler RG, Laferla FM, Mattson MP.

Neurobiol Dis. 2007 Apr;26(1):212-20. Epub 2007 Jan 13.

PMID:
17306982
4.

High-fat diet-induced memory impairment in triple-transgenic Alzheimer's disease (3xTgAD) mice is independent of changes in amyloid and tau pathology.

Knight EM, Martins IV, Gümüsgöz S, Allan SM, Lawrence CB.

Neurobiol Aging. 2014 Aug;35(8):1821-32. doi: 10.1016/j.neurobiolaging.2014.02.010. Epub 2014 Feb 15.

5.

Circadian system functionality, hippocampal oxidative stress, and spatial memory in the APPswe/PS1dE9 transgenic model of Alzheimer disease: effects of melatonin or ramelteon.

Baño Otalora B, Popovic N, Gambini J, Popovic M, Viña J, Bonet-Costa V, Reiter RJ, Camello PJ, Rol MÁ, Madrid JA.

Chronobiol Int. 2012 Aug;29(7):822-34. doi: 10.3109/07420528.2012.699119.

PMID:
22823866
6.

Cognitive and emotional profiles of aged Alzheimer's disease (3×TgAD) mice: effects of environmental enrichment and sexual dimorphism.

Blázquez G, Cañete T, Tobeña A, Giménez-Llort L, Fernández-Teruel A.

Behav Brain Res. 2014 Jul 15;268:185-201. doi: 10.1016/j.bbr.2014.04.008. Epub 2014 Apr 15.

PMID:
24746486
7.

Chronic mild sleep restriction accentuates contextual memory impairments, and accumulations of cortical Aβ and pTau in a mouse model of Alzheimer's disease.

Rothman SM, Herdener N, Frankola KA, Mughal MR, Mattson MP.

Brain Res. 2013 Sep 5;1529:200-8. doi: 10.1016/j.brainres.2013.07.010. Epub 2013 Jul 13.

8.

Episodic-like memory is sensitive to both Alzheimer's-like pathological accumulation and normal ageing processes in mice.

Davis KE, Eacott MJ, Easton A, Gigg J.

Behav Brain Res. 2013 Oct 1;254:73-82. doi: 10.1016/j.bbr.2013.03.009. Epub 2013 Mar 15.

PMID:
23500896
9.

Prophylactic treatment with paroxetine ameliorates behavioral deficits and retards the development of amyloid and tau pathologies in 3xTgAD mice.

Nelson RL, Guo Z, Halagappa VM, Pearson M, Gray AJ, Matsuoka Y, Brown M, Martin B, Iyun T, Maudsley S, Clark RF, Mattson MP.

Exp Neurol. 2007 May;205(1):166-76. Epub 2007 Feb 13.

10.

Cyclooxygenase-1 inhibition reduces amyloid pathology and improves memory deficits in a mouse model of Alzheimer's disease.

Choi SH, Aid S, Caracciolo L, Minami SS, Niikura T, Matsuoka Y, Turner RS, Mattson MP, Bosetti F.

J Neurochem. 2013 Jan;124(1):59-68. doi: 10.1111/jnc.12059. Epub 2012 Nov 21.

11.

Effects of accelerated senescence on learning and memory, locomotion and anxiety-like behavior in APP/PS1 mouse model of Alzheimer's disease.

Lok K, Zhao H, Zhang C, He N, Shen H, Wang Z, Zhao W, Yin M.

J Neurol Sci. 2013 Dec 15;335(1-2):145-54. doi: 10.1016/j.jns.2013.09.018. Epub 2013 Sep 21.

PMID:
24095271
12.

Cognitive and emotional alterations in young Alzheimer's disease (3xTgAD) mice: effects of neonatal handling stimulation and sexual dimorphism.

Cañete T, Blázquez G, Tobeña A, Giménez-Llort L, Fernández-Teruel A.

Behav Brain Res. 2015 Mar 15;281:156-71. doi: 10.1016/j.bbr.2014.11.004. Epub 2014 Nov 13.

PMID:
25446741
13.

Synergistic Interactions between Abeta, tau, and alpha-synuclein: acceleration of neuropathology and cognitive decline.

Clinton LK, Blurton-Jones M, Myczek K, Trojanowski JQ, LaFerla FM.

J Neurosci. 2010 May 26;30(21):7281-9. doi: 10.1523/JNEUROSCI.0490-10.2010.

14.

Allopregnanolone increases the number of dopaminergic neurons in substantia nigra of a triple transgenic mouse model of Alzheimer's disease.

Sun C, Ou X, Farley JM, Stockmeier C, Bigler S, Brinton RD, Wang JM.

Curr Alzheimer Res. 2012 May;9(4):473-80.

15.

Reducing Ribosomal Protein S6 Kinase 1 Expression Improves Spatial Memory and Synaptic Plasticity in a Mouse Model of Alzheimer's Disease.

Caccamo A, Branca C, Talboom JS, Shaw DM, Turner D, Ma L, Messina A, Huang Z, Wu J, Oddo S.

J Neurosci. 2015 Oct 14;35(41):14042-56. doi: 10.1523/JNEUROSCI.2781-15.2015.

16.

3xTgAD mice exhibit altered behavior and elevated Aβ after chronic mild social stress.

Rothman SM, Herdener N, Camandola S, Texel SJ, Mughal MR, Cong WN, Martin B, Mattson MP.

Neurobiol Aging. 2012 Apr;33(4):830.e1-12. doi: 10.1016/j.neurobiolaging.2011.07.005. Epub 2011 Aug 19.

17.

Effects of aging and genotype on circadian rhythms, sleep, and clock gene expression in APPxPS1 knock-in mice, a model for Alzheimer's disease.

Duncan MJ, Smith JT, Franklin KM, Beckett TL, Murphy MP, St Clair DK, Donohue KD, Striz M, O'Hara BF.

Exp Neurol. 2012 Aug;236(2):249-58. doi: 10.1016/j.expneurol.2012.05.011. Epub 2012 May 22.

PMID:
22634208
18.

L-3-n-butylphthalide improves cognitive impairment and reduces amyloid-beta in a transgenic model of Alzheimer's disease.

Peng Y, Sun J, Hon S, Nylander AN, Xia W, Feng Y, Wang X, Lemere CA.

J Neurosci. 2010 Jun 16;30(24):8180-9. doi: 10.1523/JNEUROSCI.0340-10.2010.

19.

Neurovascular uncoupling in the triple transgenic model of Alzheimer's disease: Impaired cerebral blood flow response to neuronal-derived nitric oxide signaling.

Lourenço CF, Ledo A, Barbosa RM, Laranjinha J.

Exp Neurol. 2017 May;291:36-43. doi: 10.1016/j.expneurol.2017.01.013. Epub 2017 Feb 1.

PMID:
28161255
20.

Biochemical and morphological characterization of the AβPP/PS/tau triple transgenic mouse model and its relevance to sporadic Alzheimer's disease.

Hunter JM, Bowers WJ, Maarouf CL, Mastrangelo MA, Daugs ID, Kokjohn TA, Kalback WM, Luehrs DC, Valla J, Beach TG, Roher AE.

J Alzheimers Dis. 2011;27(2):361-76. doi: 10.3233/JAD-2011-110608.

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