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Items: 1 to 20 of 128

1.

The stimulation of adenosine 2A receptor reduces inflammatory response in mouse articular chondrocytes treated with hyaluronan oligosaccharides.

Campo GM, Avenoso A, D'Ascola A, Prestipino V, Scuruchi M, Nastasi G, Calatroni A, Campo S.

Matrix Biol. 2012 Jul;31(6):338-51. doi: 10.1016/j.matbio.2012.07.001. Epub 2012 Jul 13.

PMID:
22796382
2.

Adenosine A2A receptor activation and hyaluronan fragment inhibition reduce inflammation in mouse articular chondrocytes stimulated with interleukin-1β.

Campo GM, Avenoso A, D'Ascola A, Scuruchi M, Prestipino V, Nastasi G, Calatroni A, Campo S.

FEBS J. 2012 Jun;279(12):2120-33. doi: 10.1111/j.1742-4658.2012.08598.x. Epub 2012 May 9.

3.

Protein kinase a mediated anti-inflammatory effects exerted by adenosine treatment in mouse chondrocytes stimulated with IL-1β.

Campo GM, Avenoso A, D'Ascola A, Prestipino V, Scuruchi M, Nastasi G, Calatroni A, Campo S.

Biofactors. 2012 Nov-Dec;38(6):429-39. doi: 10.1002/biof.1040. Epub 2012 Oct 25.

PMID:
23097357
4.

The SOD mimic MnTM-2-PyP(5+) reduces hyaluronan degradation-induced inflammation in mouse articular chondrocytes stimulated with Fe (II) plus ascorbate.

Campo GM, Avenoso A, D'Ascola A, Scuruchi M, Nastasi G, Micali A, Puzzolo D, Pisani A, Calatroni A, Campo S.

Int J Biochem Cell Biol. 2013 Aug;45(8):1610-9. doi: 10.1016/j.biocel.2013.05.007. Epub 2013 May 18.

PMID:
23692848
5.

Combined treatment with hyaluronan inhibitor Pep-1 and a selective adenosine A2 receptor agonist reduces inflammation in experimental arthritis.

Campo GM, Avenoso A, D'Ascola A, Nastasi G, Micali A, Puzzolo D, Pisani A, Prestipino V, Scuruchi M, Calatroni A, Campo S.

Innate Immun. 2013 Oct;19(5):462-78. doi: 10.1177/1753425912470391. Epub 2013 Jan 2.

PMID:
23283732
6.

Beta-arrestin-2 negatively modulates inflammation response in mouse chondrocytes induced by 4-mer hyaluronan oligosaccharide.

Campo GM, Avenoso A, D'Ascola A, Scuruchi M, Calatroni A, Campo S.

Mol Cell Biochem. 2015 Jan;399(1-2):201-8. doi: 10.1007/s11010-014-2246-5. Epub 2014 Oct 16.

PMID:
25318610
7.

The inhibition of hyaluronan degradation reduced pro-inflammatory cytokines in mouse synovial fibroblasts subjected to collagen-induced arthritis.

Campo GM, Avenoso A, D'Ascola A, Scuruchi M, Prestipino V, Nastasi G, Calatroni A, Campo S.

J Cell Biochem. 2012 Jun;113(6):1852-67. doi: 10.1002/jcb.24054.

PMID:
22234777
8.

Inhibition of the hyaluronan oligosaccharides inflammatory response: reduction of adenosine 2A receptor activation by EPAC and PKA.

Campo GM, Avenoso A, D'Ascola A, Scuruchi M, Nastasi G, Calatroni A, Campo S.

Cell Biochem Funct. 2014 Dec;32(8):692-701. doi: 10.1002/cbf.3073. Epub 2014 Nov 4.

PMID:
25367782
9.

Hyaluronan differently modulates TLR-4 and the inflammatory response in mouse chondrocytes.

Campo GM, Avenoso A, D'Ascola A, Prestipino V, Scuruchi M, Nastasi G, Calatroni A, Campo S.

Biofactors. 2012 Jan-Feb;38(1):69-76. doi: 10.1002/biof.202. Epub 2012 Jan 30.

PMID:
22287316
10.

Small hyaluronan oligosaccharides induce inflammation by engaging both toll-like-4 and CD44 receptors in human chondrocytes.

Campo GM, Avenoso A, Campo S, D'Ascola A, Nastasi G, Calatroni A.

Biochem Pharmacol. 2010 Aug 15;80(4):480-90. doi: 10.1016/j.bcp.2010.04.024. Epub 2010 May 8.

PMID:
20435021
11.

Molecular size hyaluronan differently modulates toll-like receptor-4 in LPS-induced inflammation in mouse chondrocytes.

Campo GM, Avenoso A, Campo S, D'Ascola A, Nastasi G, Calatroni A.

Biochimie. 2010 Feb;92(2):204-15. doi: 10.1016/j.biochi.2009.10.006. Epub 2009 Oct 29.

PMID:
19879319
12.

Hyaluronan in part mediates IL-1beta-induced inflammation in mouse chondrocytes by up-regulating CD44 receptors.

Campo GM, Avenoso A, D'Ascola A, Scuruchi M, Prestipino V, Calatroni A, Campo S.

Gene. 2012 Feb 15;494(1):24-35. doi: 10.1016/j.gene.2011.11.064. Epub 2011 Dec 13.

PMID:
22192912
13.

6-Mer hyaluronan oligosaccharides increase IL-18 and IL-33 production in mouse synovial fibroblasts subjected to collagen-induced arthritis.

Campo GM, Avenoso A, D'Ascola A, Scuruchi M, Prestipino V, Calatroni A, Campo S.

Innate Immun. 2012 Oct;18(5):675-84. Epub 2012 Jan 25.

PMID:
22278935
14.

Inhibition of hyaluronan synthesis reduced inflammatory response in mouse synovial fibroblasts subjected to collagen-induced arthritis.

Campo GM, Avenoso A, D'Ascola A, Prestipino V, Scuruchi M, Nastasi G, Calatroni A, Campo S.

Arch Biochem Biophys. 2012 Feb 1;518(1):42-52. doi: 10.1016/j.abb.2011.12.005. Epub 2011 Dec 14.

PMID:
22197458
15.

4-mer hyaluronan oligosaccharides stimulate inflammation response in synovial fibroblasts in part via TAK-1 and in part via p38-MAPK.

Campo GM, Avenoso A, D'Ascola A, Prestipino V, Scuruchi M, Nastasi G, Calatroni A, Campo S.

Curr Med Chem. 2013;20(9):1162-72.

PMID:
23298137
16.

Differential effect of molecular size HA in mouse chondrocytes stimulated with PMA.

Campo GM, Avenoso A, Campo S, D'Ascola A, Traina P, Calatroni A.

Biochim Biophys Acta. 2009 Oct;1790(10):1353-67. doi: 10.1016/j.bbagen.2009.07.003. Epub 2009 Jul 14.

PMID:
19607883
17.

Beta-arrestin 1 is involved in the catabolic response stimulated by hyaluronan degradation in mouse chondrocytes.

Campo GM, Avenoso A, D'Ascola A, Scuruchi M, Calatroni A, Campo S.

Cell Tissue Res. 2015 Aug;361(2):567-79. doi: 10.1007/s00441-015-2112-0. Epub 2015 Feb 13.

PMID:
25673209
18.

Inhibition of small HA fragment activity and stimulation of A2A adenosine receptor pathway limit apoptosis and reduce cartilage damage in experimental arthritis.

Campo GM, Micali A, Avenoso A, D'Ascola A, Scuruchi M, Pisani A, Bruschetta A, Calatroni A, Puzzolo D, Campo S.

Histochem Cell Biol. 2015 May;143(5):531-43. doi: 10.1007/s00418-014-1298-7. Epub 2014 Dec 16.

PMID:
25511416
19.

Differential regulation of HIF-1alpha isoforms in murine macrophages by TLR4 and adenosine A(2A) receptor agonists.

Ramanathan M, Luo W, Csóka B, Haskó G, Lukashev D, Sitkovsky MV, Leibovich SJ.

J Leukoc Biol. 2009 Sep;86(3):681-9. doi: 10.1189/jlb.0109021. Epub 2009 May 28.

20.

Activation of adenosine A(2A) receptor reduces osteoclast formation via PKA- and ERK1/2-mediated suppression of NFκB nuclear translocation.

Mediero A, Perez-Aso M, Cronstein BN.

Br J Pharmacol. 2013 Jul;169(6):1372-88. doi: 10.1111/bph.12227.

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