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Items: 1 to 20 of 301

1.

Upregulation of N-acetylglucosaminyltransferase-V by heparin-binding EGF-like growth factor induces keratinocyte proliferation and epidermal hyperplasia.

Kimura A, Terao M, Kato A, Hanafusa T, Murota H, Katayama I, Miyoshi E.

Exp Dermatol. 2012 Jul;21(7):515-9. doi: 10.1111/j.1600-0625.2012.01515.x.

PMID:
22716246
2.

Physiological roles of N-acetylglucosaminyltransferase V(GnT-V) in mice.

Miyoshi E, Terao M, Kamada Y.

BMB Rep. 2012 Oct;45(10):554-9. Review.

3.

Enhanced epithelial-mesenchymal transition-like phenotype in N-acetylglucosaminyltransferase V transgenic mouse skin promotes wound healing.

Terao M, Ishikawa A, Nakahara S, Kimura A, Kato A, Moriwaki K, Kamada Y, Murota H, Taniguchi N, Katayama I, Miyoshi E.

J Biol Chem. 2011 Aug 12;286(32):28303-11. doi: 10.1074/jbc.M111.220376.

5.

The role of heparin-binding EGF-like growth factor and amphiregulin in the epidermal proliferation of psoriasis in cooperation with TNFalpha.

Yoshida A, Kanno H, Watabe D, Akasaka T, Sawai T.

Arch Dermatol Res. 2008 Jan;300(1):37-45.

PMID:
17960400
6.
7.

Overexpression of N-acetylglucosaminyltransferase III enhances the epidermal growth factor-induced phosphorylation of ERK in HeLaS3 cells by up-regulation of the internalization rate of the receptors.

Sato Y, Takahashi M, Shibukawa Y, Jain SK, Hamaoka R, Miyagawa Ji, Yaginuma Y, Honke K, Ishikawa M, Taniguchi N.

J Biol Chem. 2001 Apr 13;276(15):11956-62.

8.

Altered β1,6-GlcNAc branched N-glycans impair TGF-β-mediated epithelial-to-mesenchymal transition through Smad signalling pathway in human lung cancer.

Li N, Xu H, Fan K, Liu X, Qi J, Zhao C, Yin P, Wang L, Li Z, Zha X.

J Cell Mol Med. 2014 Oct;18(10):1975-91. doi: 10.1111/jcmm.12331.

9.
10.

HaCaT keratinocyte migration is dependent on epidermal growth factor receptor signaling and glycogen synthase kinase-3alpha.

Koivisto L, Jiang G, Häkkinen L, Chan B, Larjava H.

Exp Cell Res. 2006 Sep 10;312(15):2791-805.

PMID:
16806170
11.

Bisecting GlcNAc residues on laminin-332 down-regulate galectin-3-dependent keratinocyte motility.

Kariya Y, Kawamura C, Tabei T, Gu J.

J Biol Chem. 2010 Jan 29;285(5):3330-40. doi: 10.1074/jbc.M109.038836.

12.

The tumor-suppressive function of Connexin43 in keratinocytes is mediated in part via interaction with caveolin-1.

Langlois S, Cowan KN, Shao Q, Cowan BJ, Laird DW.

Cancer Res. 2010 May 15;70(10):4222-32. doi: 10.1158/0008-5472.CAN-09-3281.

13.

Epidermal growth factor receptor-mediated activation of Stat3 during multistage skin carcinogenesis.

Chan KS, Carbajal S, Kiguchi K, Clifford J, Sano S, DiGiovanni J.

Cancer Res. 2004 Apr 1;64(7):2382-9.

14.

Autocrine transforming growth factor alpha is dispensible for v-rasHa-induced epidermal neoplasia: potential involvement of alternate epidermal growth factor receptor ligands.

Dlugosz AA, Cheng C, Williams EK, Darwiche N, Dempsey PJ, Mann B, Dunn AR, Coffey RJ Jr, Yuspa SH.

Cancer Res. 1995 May 1;55(9):1883-93.

15.

Oligosaccharide modification by N-acetylglucosaminyltransferase-V in macrophages are involved in pathogenesis of bleomycin-induced scleroderma.

Kato A, Yutani M, Terao M, Kimura A, Itoi S, Murota H, Miyoshi E, Katayama I.

Exp Dermatol. 2015 Aug;24(8):585-90. doi: 10.1111/exd.12730.

PMID:
25876794
16.
17.

Roles of N-acetylglucosaminyltransferase III in epithelial-to-mesenchymal transition induced by transforming growth factor β1 (TGF-β1) in epithelial cell lines.

Xu Q, Isaji T, Lu Y, Gu W, Kondo M, Fukuda T, Du Y, Gu J.

J Biol Chem. 2012 May 11;287(20):16563-74. doi: 10.1074/jbc.M111.262154.

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20.

Gene transfection-mediated overexpression of beta1,4-N-acetylglucosamine bisecting oligosaccharides in glioma cell line U373 MG inhibits epidermal growth factor receptor function.

Rebbaa A, Yamamoto H, Saito T, Meuillet E, Kim P, Kersey DS, Bremer EG, Taniguchi N, Moskal JR.

J Biol Chem. 1997 Apr 4;272(14):9275-9.

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