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Items: 1 to 20 of 93

1.

Multiple CDK/CYCLIND genes are amplified in medulloblastoma and supratentorial primitive neuroectodermal brain tumor.

Li M, Lockwood W, Zielenska M, Northcott P, Ra YS, Bouffet E, Yoshimoto M, Rutka JT, Yan H, Taylor MD, Eberhart C, Hawkins CE, Lam W, Squire JA, Huang A.

Cancer Genet. 2012 May;205(5):220-31. doi: 10.1016/j.cancergen.2012.03.002.

PMID:
22682621
2.

High promoter hypermethylation frequency of p14/ARF in supratentorial PNET but not in medulloblastoma.

Inda MM, Muñoz J, Coullin P, Fauvet D, Danglot G, Tuñón T, Bernheim A, Castresana JS.

Histopathology. 2006 Apr;48(5):579-87.

PMID:
16623784
3.
4.

The p16-cyclin D/Cdk4-pRb pathway as a functional unit frequently altered in melanoma pathogenesis.

Bartkova J, Lukas J, Guldberg P, Alsner J, Kirkin AF, Zeuthen J, Bartek J.

Cancer Res. 1996 Dec 1;56(23):5475-83.

5.

Amplification and expression of cyclin D genes (CCND1, CCND2 and CCND3) in human malignant gliomas.

Büschges R, Weber RG, Actor B, Lichter P, Collins VP, Reifenberger G.

Brain Pathol. 1999 Jul;9(3):435-42; discussion 432-3.

PMID:
10416984
6.

Epigenetic inactivation of DLC-1 in supratentorial primitive neuroectodermal tumor.

Pang JC, Chang Q, Chung YF, Teo JG, Poon WS, Zhou LF, Kong X, Ng HK.

Hum Pathol. 2005 Jan;36(1):36-43.

PMID:
15712180
7.

Multiple functions of D-type cyclins can antagonize pRb-mediated suppression of proliferation.

Baker GL, Landis MW, Hinds PW.

Cell Cycle. 2005 Feb;4(2):330-8. Epub 2005 Feb 21.

PMID:
15684604
8.
10.

Supratentorial primitive neuroectodermal tumors of the central nervous system frequently harbor deletions of the CDKN2A locus and other genomic aberrations distinct from medulloblastomas.

Pfister S, Remke M, Toedt G, Werft W, Benner A, Mendrzyk F, Wittmann A, Devens F, von Hoff K, Rutkowski S, Kulozik A, Radlwimmer B, Scheurlen W, Lichter P, Korshunov A.

Genes Chromosomes Cancer. 2007 Sep;46(9):839-51.

PMID:
17592618
11.

Upregulation of SOX2, NOTCH1, and ID1 in supratentorial primitive neuroectodermal tumors: a distinct differentiation pattern from that of medulloblastomas.

Phi JH, Kim JH, Eun KM, Wang KC, Park KH, Choi SA, Kim YY, Park SH, Cho BK, Kim SK.

J Neurosurg Pediatr. 2010 Jun;5(6):608-14. doi: 10.3171/2010.2.PEDS1065.

PMID:
20515335
12.

Mutation analysis of the pRb pathway in 2',3'-dideoxycytidine- and 1, 3-butadiene-induced mouse lymphomas.

Zhuang SM, Wiseman RW, Söderkvist P.

Cancer Lett. 2000 May 1;152(2):129-34.

PMID:
10773403
13.

Downregulation of cyclin D1 alters cdk 4- and cdk 2-specific phosphorylation of retinoblastoma protein.

Yu B, Lane ME, Pestell RG, Albanese C, Wadler S.

Mol Cell Biol Res Commun. 2000 Jun;3(6):352-9.

PMID:
11032757
14.

Genetic heterogeneity in supratentorial and infratentorial primitive neuroectodermal tumours of the central nervous system.

Inda MM, Perot C, Guillaud-Bataille M, Danglot G, Rey JA, Bello MJ, Fan X, Eberhart C, Zazpe I, Portillo E, Tuñón T, Martínez-Peñuela JM, Bernheim A, Castresana JS.

Histopathology. 2005 Dec;47(6):631-7.

PMID:
16324202
16.

Cell cycle and biochemical effects of PD 0183812. A potent inhibitor of the cyclin D-dependent kinases CDK4 and CDK6.

Fry DW, Bedford DC, Harvey PH, Fritsch A, Keller PR, Wu Z, Dobrusin E, Leopold WR, Fattaey A, Garrett MD.

J Biol Chem. 2001 May 18;276(20):16617-23. Epub 2001 Feb 6.

17.

Involvement of the pRb/p16/cdk4/cyclin D1 pathway in the tumorigenesis of sporadic malignant melanomas.

Maelandsmo GM, Flørenes VA, Hovig E, Oyjord T, Engebraaten O, Holm R, Børresen AL, Fodstad O.

Br J Cancer. 1996 Apr;73(8):909-16.

18.

Cyclin D1 amplification is independent of p16 inactivation in head and neck squamous cell carcinoma.

Okami K, Reed AL, Cairns P, Koch WM, Westra WH, Wehage S, Jen J, Sidransky D.

Oncogene. 1999 Jun 10;18(23):3541-5.

19.

Among genes involved in the RB dependent cell cycle regulatory cascade, the p16 tumor suppressor gene is frequently lost in the Ewing family of tumors.

Kovar H, Jug G, Aryee DN, Zoubek A, Ambros P, Gruber B, Windhager R, Gadner H.

Oncogene. 1997 Oct;15(18):2225-32.

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