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Items: 1 to 20 of 107

1.

Interleukin 5 is protective during sepsis in an eosinophil-independent manner.

Linch SN, Danielson ET, Kelly AM, Tamakawa RA, Lee JJ, Gold JA.

Am J Respir Crit Care Med. 2012 Aug 1;186(3):246-54. doi: 10.1164/rccm.201201-0134OC.

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Depletion of neutrophil extracellular traps in vivo results in hypersusceptibility to polymicrobial sepsis in mice.

Meng W, Paunel-Görgülü A, Flohé S, Hoffmann A, Witte I, MacKenzie C, Baldus SE, Windolf J, Lögters TT.

Crit Care. 2012 Jul 26;16(4):R137. doi: 10.1186/cc11442.

4.

Elevated expression of IL-23/IL-17 pathway-related mediators correlates with exacerbation of pulmonary inflammation during polymicrobial sepsis.

Cauvi DM, Williams MR, Bermudez JA, Armijo G, De Maio A.

Shock. 2014 Sep;42(3):246-55. doi: 10.1097/SHK.0000000000000207.

5.

OX40 ligand regulates inflammation and mortality in the innate immune response to sepsis.

Karulf M, Kelly A, Weinberg AD, Gold JA.

J Immunol. 2010 Oct 15;185(8):4856-62. doi: 10.4049/jimmunol.1000404.

6.

Interleukin-7 (IL-7) treatment accelerates neutrophil recruitment through gamma delta T-cell IL-17 production in a murine model of sepsis.

Kasten KR, Prakash PS, Unsinger J, Goetzman HS, England LG, Cave CM, Seitz AP, Mazuski CN, Zhou TT, Morre M, Hotchkiss RS, Hildeman DA, Caldwell CC.

Infect Immun. 2010 Nov;78(11):4714-22. doi: 10.1128/IAI.00456-10.

7.

A2B adenosine receptor blockade enhances macrophage-mediated bacterial phagocytosis and improves polymicrobial sepsis survival in mice.

Belikoff BG, Hatfield S, Georgiev P, Ohta A, Lukashev D, Buras JA, Remick DG, Sitkovsky M.

J Immunol. 2011 Feb 15;186(4):2444-53. doi: 10.4049/jimmunol.1001567.

8.

Diversity of interferon gamma and granulocyte-macrophage colony-stimulating factor in restoring immune dysfunction of dendritic cells and macrophages during polymicrobial sepsis.

Flohé SB, Agrawal H, Flohé S, Rani M, Bangen JM, Schade FU.

Mol Med. 2008 May-Jun;14(5-6):247-56. doi: 10.2119/2007-00120.Flohe.

9.

ATP-gated P2X1 ion channels protect against endotoxemia by dampening neutrophil activation.

Lecut C, Faccinetto C, Delierneux C, van Oerle R, Spronk HM, Evans RJ, El Benna J, Bours V, Oury C.

J Thromb Haemost. 2012 Mar;10(3):453-65. doi: 10.1111/j.1538-7836.2011.04606.x.

10.

Immune response in human visceral leishmaniasis: analysis of the correlation between innate immunity cytokine profile and disease outcome.

Peruhype-Magalhães V, Martins-Filho OA, Prata A, Silva Lde A, Rabello A, Teixeira-Carvalho A, Figueiredo RM, Guimarães-Carvalho SF, Ferrari TC, Correa-Oliveira R.

Scand J Immunol. 2005 Nov;62(5):487-95.

11.

Innate cellular sources of interleukin-17A regulate macrophage accumulation in cigarette- smoke-induced lung inflammation in mice.

Bozinovski S, Seow HJ, Chan SP, Anthony D, McQualter J, Hansen M, Jenkins BJ, Anderson GP, Vlahos R.

Clin Sci (Lond). 2015 Nov;129(9):785-96. doi: 10.1042/CS20140703.

12.

NOD2-mediated suppression of CD55 on neutrophils enhances C5a generation during polymicrobial sepsis.

Oh SJ, Kim JH, Chung DH.

PLoS Pathog. 2013 May;9(5):e1003351. doi: 10.1371/journal.ppat.1003351.

13.

IL-27 controls sepsis-induced impairment of lung antibacterial host defence.

Cao J, Xu F, Lin S, Song Z, Zhang L, Luo P, Xu H, Li D, Zheng K, Ren G, Yin Y.

Thorax. 2014 Oct;69(10):926-37. doi: 10.1136/thoraxjnl-2014-205777. Erratum in: Thorax. 2015 Mar;70(3):243.

PMID:
25074706
14.

Human mesenchymal stem cells reduce mortality and bacteremia in gram-negative sepsis in mice in part by enhancing the phagocytic activity of blood monocytes.

Krasnodembskaya A, Samarani G, Song Y, Zhuo H, Su X, Lee JW, Gupta N, Petrini M, Matthay MA.

Am J Physiol Lung Cell Mol Physiol. 2012 May 15;302(10):L1003-13. doi: 10.1152/ajplung.00180.2011.

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TLR2-induced IL-10 production impairs neutrophil recruitment to infected tissues during neonatal bacterial sepsis.

Andrade EB, Alves J, Madureira P, Oliveira L, Ribeiro A, Cordeiro-da-Silva A, Correia-Neves M, Trieu-Cuot P, Ferreira P.

J Immunol. 2013 Nov 1;191(9):4759-68. doi: 10.4049/jimmunol.1301752.

17.

BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction.

Shubin NJ, Chung CS, Heffernan DS, Irwin LR, Monaghan SF, Ayala A.

J Leukoc Biol. 2012 Sep;92(3):593-603. doi: 10.1189/jlb.1211641.

18.

Neutrophil IL-10 suppresses peritoneal inflammatory monocytes during polymicrobial sepsis.

Ocuin LM, Bamboat ZM, Balachandran VP, Cavnar MJ, Obaid H, Plitas G, DeMatteo RP.

J Leukoc Biol. 2011 Mar;89(3):423-32. doi: 10.1189/jlb.0810479.

19.

The phosphatidylinositol 3-kinase signaling pathway exerts protective effects during sepsis by controlling C5a-mediated activation of innate immune functions.

Wrann CD, Tabriz NA, Barkhausen T, Klos A, van Griensven M, Pape HC, Kendoff DO, Guo R, Ward PA, Krettek C, Riedemann NC.

J Immunol. 2007 May 1;178(9):5940-8.

20.

CD36 Is Essential for Regulation of the Host Innate Response to Staphylococcus aureus α-Toxin-Mediated Dermonecrosis.

Castleman MJ, Febbraio M, Hall PR.

J Immunol. 2015 Sep 1;195(5):2294-302. doi: 10.4049/jimmunol.1500500.

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