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Items: 1 to 20 of 93


IFN-γ-deficient mice develop IL-1-dependent cutaneous and musculoskeletal inflammation during experimental brucellosis.

Skyberg JA, Thornburg T, Kochetkova I, Layton W, Callis G, Rollins MF, Riccardi C, Becker T, Golden S, Pascual DW.

J Leukoc Biol. 2012 Aug;92(2):375-87. doi: 10.1189/jlb.1211626. Epub 2012 May 25.


CXCR2 Mediates Brucella-Induced Arthritis in Interferon γ-Deficient Mice.

Lacey CA, Keleher LL, Mitchell WJ, Brown CR, Skyberg JA.

J Infect Dis. 2016 Jul 1;214(1):151-60. doi: 10.1093/infdis/jiw087. Epub 2016 Mar 6.


Temporal Role for MyD88 in a Model of Brucella-Induced Arthritis and Musculoskeletal Inflammation.

Lacey CA, Mitchell WJ, Brown CR, Skyberg JA.

Infect Immun. 2017 Feb 23;85(3). pii: e00961-16. doi: 10.1128/IAI.00961-16. Print 2017 Mar.


Host susceptibility to Brucella abortus infection is more pronounced in IFN-γ knockout than IL-12/β2-microglobulin double-deficient mice.

Brandão AP, Oliveira FS, Carvalho NB, Vieira LQ, Azevedo V, Macedo GC, Oliveira SC.

Clin Dev Immunol. 2012;2012:589494. doi: 10.1155/2012/589494. Epub 2011 Dec 11.


Nramp1 is not a major determinant in the control of Brucella melitensis infection in mice.

Guilloteau LA, Dornand J, Gross A, Olivier M, Cortade F, Vern YL, Kerboeuf D.

Infect Immun. 2003 Feb;71(2):621-8.


Comparison of T cell cytokines in resistant and susceptible mice infected with virulent Brucella abortus strain 2308.

Fernandes DM, Jiang X, Jung JH, Baldwin CL.

FEMS Immunol Med Microbiol. 1996 Dec 31;16(3-4):193-203.


MyD88-dependent activation of B220-CD11b+LY-6C+ dendritic cells during Brucella melitensis infection.

Copin R, De Baetselier P, Carlier Y, Letesson JJ, Muraille E.

J Immunol. 2007 Apr 15;178(8):5182-91.


Interferon-gamma is crucial for surviving a Brucella abortus infection in both resistant C57BL/6 and susceptible BALB/c mice.

Murphy EA, Sathiyaseelan J, Parent MA, Zou B, Baldwin CL.

Immunology. 2001 Aug;103(4):511-8.


Lack of endogenous IL-10 enhances production of proinflammatory cytokines and leads to Brucella abortus clearance in mice.

Corsetti PP, de Almeida LA, Carvalho NB, Azevedo V, Silva TM, Teixeira HC, Faria AC, Oliveira SC.

PLoS One. 2013 Sep 17;8(9):e74729. doi: 10.1371/journal.pone.0074729. eCollection 2013.


Protective live oral brucellosis vaccines stimulate Th1 and th17 cell responses.

Clapp B, Skyberg JA, Yang X, Thornburg T, Walters N, Pascual DW.

Infect Immun. 2011 Oct;79(10):4165-74. doi: 10.1128/IAI.05080-11. Epub 2011 Jul 18.


Endogenous gamma interferon and interleukin-10 in Brucella abortus 2308 infection in mice.

Fernández-Lago L, Monte M, Chordi A.

FEMS Immunol Med Microbiol. 1996 Sep;15(2-3):109-14.


What have we learned from brucellosis in the mouse model?

Grilló MJ, Blasco JM, Gorvel JP, Moriyón I, Moreno E.

Vet Res. 2012 Apr 13;43:29. doi: 10.1186/1297-9716-43-29. Review.


CD8+ T cell exhaustion, suppressed gamma interferon production, and delayed memory response induced by chronic Brucella melitensis infection.

Durward-Diioia M, Harms J, Khan M, Hall C, Smith JA, Splitter GA.

Infect Immun. 2015 Dec;83(12):4759-71. doi: 10.1128/IAI.01184-15. Epub 2015 Sep 28.


Helicobacter pylori-induced mucosal inflammation is Th1 mediated and exacerbated in IL-4, but not IFN-gamma, gene-deficient mice.

Smythies LE, Waites KB, Lindsey JR, Harris PR, Ghiara P, Smith PD.

J Immunol. 2000 Jul 15;165(2):1022-9.


Crucial role of gamma interferon-producing CD4+ Th1 cells but dispensable function of CD8+ T cell, B cell, Th2, and Th17 responses in the control of Brucella melitensis infection in mice.

Vitry MA, De Trez C, Goriely S, Dumoutier L, Akira S, Ryffel B, Carlier Y, Letesson JJ, Muraille E.

Infect Immun. 2012 Dec;80(12):4271-80. doi: 10.1128/IAI.00761-12. Epub 2012 Sep 24.


Treatment of Brucella-susceptible mice with IL-12 increases primary and secondary immunity.

Sathiyaseelan J, Goenka R, Parent M, Benson RM, Murphy EA, Fernandes DM, Foulkes AS, Baldwin CL.

Cell Immunol. 2006 Sep;243(1):1-9. Epub 2006 Dec 20.


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