Warning: The NCBI web site requires JavaScript to function. more...
Fetching bibliography...
Generate a file for use with external citation management software.
Modelling the interaction of keratinocytes and fibroblasts during normal and abnormal wound healing processes.
Menon SN, Flegg JA, McCue SW, Schugart RC, Dawson RA, McElwain DL.
Proc Biol Sci. 2012 Aug 22;279(1741):3329-38. doi: 10.1098/rspb.2012.0319. Epub 2012 May 23.
Similar articles
Glu-Leu-Arg-negative CXC chemokine interferon gamma inducible protein-9 as a mediator of epidermal-dermal communication during wound repair.
Satish L, Yager D, Wells A.
J Invest Dermatol. 2003 Jun;120(6):1110-7.
Keratinocyte-fibroblast interactions in wound healing.
Werner S, Krieg T, Smola H.
J Invest Dermatol. 2007 May;127(5):998-1008. Review.
The use of an in vitro wound healing model, the tri-layered skin equivalent, to study the effects of cytokines on the repopulation of the wound defect by fibroblasts and keratinocytes.
O'Leary R, Arrowsmith M, Wood EJ.
Biochem Soc Trans. 1997 May;25(2):369S. No abstract available.
Biological implications of a discrete mathematical model for collagen deposition and alignment in dermal wound repair.
Dallon J, Sherratt J, Maini P, Ferguson M.
IMA J Math Appl Med Biol. 2000 Dec;17(4):379-93.
Keratinocyte-derived granulocyte-macrophage colony stimulating factor accelerates wound healing: Stimulation of keratinocyte proliferation, granulation tissue formation, and vascularization.
Mann A, Breuhahn K, Schirmacher P, Blessing M.
J Invest Dermatol. 2001 Dec;117(6):1382-90.
Modeling keratinocyte wound healing dynamics: Cell-cell adhesion promotes sustained collective migration.
Nardini JT, Chapnick DA, Liu X, Bortz DM.
J Theor Biol. 2016 Jul 7;400:103-17. doi: 10.1016/j.jtbi.2016.04.015. Epub 2016 Apr 19.
Role of keratinocyte-fibroblast cross-talk in development of hypertrophic scar.
Ghahary A, Ghaffari A.
Wound Repair Regen. 2007 Sep-Oct;15 Suppl 1:S46-53. doi: 10.1111/j.1524-475X.2007.00225.x. Review. Erratum in: Wound Repair Regen. 2008 Jul-Aug;16(4):582.
Fibrinogen and fibrin are anti-adhesive for keratinocytes: a mechanism for fibrin eschar slough during wound repair.
Kubo M, Van de Water L, Plantefaber LC, Mosesson MW, Simon M, Tonnesen MG, Taichman L, Clark RA.
J Invest Dermatol. 2001 Dec;117(6):1369-81. Erratum in: J Invest Dermatol 2002 May;118(5):910.
The role of the tetraspanin CD151 in primary keratinocyte and fibroblast functions: implications for wound healing.
Geary SM, Cowin AJ, Copeland B, Baleato RM, Miyazaki K, Ashman LK.
Exp Cell Res. 2008 Jul 1;314(11-12):2165-75. doi: 10.1016/j.yexcr.2008.04.011. Epub 2008 May 3.
Effects of hydrogen peroxide in a keratinocyte-fibroblast co-culture model of wound healing.
Loo AE, Halliwell B.
Biochem Biophys Res Commun. 2012 Jun 29;423(2):253-8. doi: 10.1016/j.bbrc.2012.05.100. Epub 2012 May 24.
Fibroblasts change spreading capability and mechanical properties in a direct interaction with keratinocytes in conditions mimicking wound healing.
Orzechowska B, Pabijan J, Wiltowska-Zuber J, Zemła J, Lekka M.
J Biomech. 2018 Jun 6;74:134-142. doi: 10.1016/j.jbiomech.2018.04.033. Epub 2018 Apr 27.
Fibroblast sheets enable epithelialization of sounds that do not support keratinocyte migration.
Krejci-Papa NC, Hoang A, Hansbrough JF.
Tissue Eng. 1999 Dec;5(6):555-62.
Paracrine factors from fibroblast aggregates in a fibrin-matrix carrier enhance keratinocyte viability and migration.
Peura M, Siltanen A, Saarinen I, Soots A, Bizik J, Vuola J, Harjula A, Kankuri E.
J Biomed Mater Res A. 2010 Nov;95(2):658-64. doi: 10.1002/jbm.a.32881.
Role of keratinocytes in healing of chronic wounds.
Pastar I, Stojadinovic O, Tomic-Canic M.
Surg Technol Int. 2008;17:105-12. Review.
Cell suspensions of autologous keratinocytes or autologous fibroblasts accelerate the healing of full thickness skin wounds in a diabetic porcine wound healing model.
Velander P, Theopold C, Bleiziffer O, Bergmann J, Svensson H, Feng Y, Eriksson E.
J Surg Res. 2009 Nov;157(1):14-20. doi: 10.1016/j.jss.2008.10.001. Epub 2008 Nov 4.
Differential activation of migration by hypoxia in keratinocytes isolated from donors of increasing age: implication for chronic wounds in the elderly.
Xia YP, Zhao Y, Tyrone JW, Chen A, Mustoe TA.
J Invest Dermatol. 2001 Jan;116(1):50-6.
Extracellular matrix-derived tripeptide proline-glycine-proline inhibits keratinocyte proliferation and migration.
Ma Y, Kleinbeck K, Kao WJ.
Wound Repair Regen. 2011 Nov;19(6):718-26. doi: 10.1111/j.1524-475X.2011.00734.x. Epub 2011 Oct 12.
[The modern approach to wound treatment].
Komarcević A.
Med Pregl. 2000 Jul-Aug;53(7-8):363-8. Review. Croatian.
Potential Wound Healing Activities of Galla Rhois in Human Fibroblasts and Keratinocytes.
Park HH, Park NY, Kim SG, Jeong KT, Lee EJ, Lee E.
Am J Chin Med. 2015;43(8):1625-36. doi: 10.1142/S0192415X15500925. Epub 2015 Nov 30.
Filters: Manage Filters