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Items: 1 to 20 of 98

1.

Tumor necrosis factor alpha stimulates cathepsin K and V activity via juxtacrine monocyte-endothelial cell signaling and JNK activation.

Keegan PM, Wilder CL, Platt MO.

Mol Cell Biochem. 2012 Aug;367(1-2):65-72. doi: 10.1007/s11010-012-1320-0.

2.

Biomechanical and biochemical regulation of cathepsin K expression in endothelial cells converge at AP-1 and NF-κB.

Keegan PM, Anbazhakan S, Kang B, Pace BS, Platt MO.

Biol Chem. 2016 May;397(5):459-68. doi: 10.1515/hsz-2015-0244.

PMID:
26760306
3.

Manipulating substrate and pH in zymography protocols selectively distinguishes cathepsins K, L, S, and V activity in cells and tissues.

Wilder CL, Park KY, Keegan PM, Platt MO.

Arch Biochem Biophys. 2011 Dec 1;516(1):52-7. doi: 10.1016/j.abb.2011.09.009.

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Increased expression of elastolytic cathepsins S, K, and V and their inhibitor cystatin C in stenotic aortic valves.

Helske S, Syväranta S, Lindstedt KA, Lappalainen J, Oörni K, Mäyränpää MI, Lommi J, Turto H, Werkkala K, Kupari M, Kovanen PT.

Arterioscler Thromb Vasc Biol. 2006 Aug;26(8):1791-8.

8.

Regulation of elastinolytic cysteine proteinase activity in normal and cathepsin K-deficient human macrophages.

Punturieri A, Filippov S, Allen E, Caras I, Murray R, Reddy V, Weiss SJ.

J Exp Med. 2000 Sep 18;192(6):789-99.

9.

Cathepsin S cannibalism of cathepsin K as a mechanism to reduce type I collagen degradation.

Barry ZT, Platt MO.

J Biol Chem. 2012 Aug 10;287(33):27723-30. doi: 10.1074/jbc.M111.332684.

10.

Effect and possible mechanism of monocyte-derived VEGF on monocyte-endothelial cellular adhesion after electrical burns.

Ruan Q, Zhao C, Ye Z, Ruan J, Xie Q, Xie W.

Burns. 2015 Jun;41(4):825-32. doi: 10.1016/j.burns.2014.10.030.

PMID:
25466960
11.

IL-6/sIL-6R enhances cathepsin B and L production via caveolin-1-mediated JNK-AP-1 pathway in human gingival fibroblasts.

Yamaguchi T, Naruishi K, Arai H, Nishimura F, Takashiba S.

J Cell Physiol. 2008 Nov;217(2):423-32. doi: 10.1002/jcp.21517.

PMID:
18543249
12.

Comparison of cathepsins K and S expression within the rheumatoid and osteoarthritic synovium.

Hou WS, Li W, Keyszer G, Weber E, Levy R, Klein MJ, Gravallese EM, Goldring SR, Brömme D.

Arthritis Rheum. 2002 Mar;46(3):663-74.

13.

META060 attenuates TNF-α-activated inflammation, endothelial-monocyte interactions, and matrix metalloproteinase-9 expression, and inhibits NF-κB and AP-1 in THP-1 monocytes.

Desai A, Darland G, Bland JS, Tripp ML, Konda VR.

Atherosclerosis. 2012 Jul;223(1):130-6. doi: 10.1016/j.atherosclerosis.2012.05.004.

PMID:
22658256
15.

Enhancement of proinflammatory and procoagulant responses to silica particles by monocyte-endothelial cell interactions.

Liu X, Xue Y, Ding T, Sun J.

Part Fibre Toxicol. 2012 Sep 18;9:36. doi: 10.1186/1743-8977-9-36.

17.

Cathepsin V, a novel and potent elastolytic activity expressed in activated macrophages.

Yasuda Y, Li Z, Greenbaum D, Bogyo M, Weber E, Brömme D.

J Biol Chem. 2004 Aug 27;279(35):36761-70.

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Postprandial VLDL lipolysis products increase monocyte adhesion and lipid droplet formation via activation of ERK2 and NFκB.

den Hartigh LJ, Altman R, Norman JE, Rutledge JC.

Am J Physiol Heart Circ Physiol. 2014 Jan 1;306(1):H109-20. doi: 10.1152/ajpheart.00137.2013.

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