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Items: 1 to 20 of 97

1.

Plasminogen activator inhibitor-1 mitigates brain injury in a rat model of infection-sensitized neonatal hypoxia-ischemia.

Yang D, Sun YY, Nemkul N, Baumann JM, Shereen A, Dunn RS, Wills-Karp M, Lawrence DA, Lindquist DM, Kuan CY.

Cereb Cortex. 2013 May;23(5):1218-29. doi: 10.1093/cercor/bhs115. Epub 2012 May 3.

2.

Taming neonatal hypoxic-ischemic brain injury by intranasal delivery of plasminogen activator inhibitor-1.

Yang D, Sun YY, Lin X, Baumann JM, Warnock M, Lawrence DA, Kuan CY.

Stroke. 2013 Sep;44(9):2623-2627. doi: 10.1161/STROKEAHA.113.001233. Epub 2013 Jul 23.

3.

Therapeutic administration of plasminogen activator inhibitor-1 prevents hypoxic-ischemic brain injury in newborns.

Yang D, Nemkul N, Shereen A, Jone A, Dunn RS, Lawrence DA, Lindquist D, Kuan CY.

J Neurosci. 2009 Jul 8;29(27):8669-74. doi: 10.1523/JNEUROSCI.1117-09.2009.

4.

Deleterious effects of plasminogen activators in neonatal cerebral hypoxia-ischemia.

Adhami F, Yu D, Yin W, Schloemer A, Burns KA, Liao G, Degen JL, Chen J, Kuan CY.

Am J Pathol. 2008 Jun;172(6):1704-16. doi: 10.2353/ajpath.2008.070979. Epub 2008 May 8.

5.

TNFR1-JNK signaling is the shared pathway of neuroinflammation and neurovascular damage after LPS-sensitized hypoxic-ischemic injury in the immature brain.

Wang LW, Chang YC, Chen SJ, Tseng CH, Tu YF, Liao NS, Huang CC, Ho CJ.

J Neuroinflammation. 2014 Dec 24;11:215. doi: 10.1186/s12974-014-0215-2.

6.

Anti-tissue plasminogen activator (tPA) as an effective therapy of neonatal hypoxia-ischemia with and without inflammation.

Yang D, Kuan CY.

CNS Neurosci Ther. 2015 Apr;21(4):367-73. doi: 10.1111/cns.12365. Epub 2014 Dec 5. Review.

7.

Blocking lymphocyte trafficking with FTY720 prevents inflammation-sensitized hypoxic-ischemic brain injury in newborns.

Yang D, Sun YY, Bhaumik SK, Li Y, Baumann JM, Lin X, Zhang Y, Lin SH, Dunn RS, Liu CY, Shie FS, Lee YH, Wills-Karp M, Chougnet CA, Kallapur SG, Lewkowich IP, Lindquist DM, Murali-Krishna K, Kuan CY.

J Neurosci. 2014 Dec 3;34(49):16467-81. doi: 10.1523/JNEUROSCI.2582-14.2014.

8.

Hypothermia is not neuroprotective after infection-sensitized neonatal hypoxic-ischemic brain injury.

Osredkar D, Thoresen M, Maes E, Flatebø T, Elstad M, Sabir H.

Resuscitation. 2014 Apr;85(4):567-72. doi: 10.1016/j.resuscitation.2013.12.006. Epub 2013 Dec 19.

PMID:
24361672
9.

Intranasal delivery of cell-penetrating anti-NF-κB peptides (Tat-NBD) alleviates infection-sensitized hypoxic-ischemic brain injury.

Yang D, Sun YY, Lin X, Baumann JM, Dunn RS, Lindquist DM, Kuan CY.

Exp Neurol. 2013 Sep;247:447-455. doi: 10.1016/j.expneurol.2013.01.015. Epub 2013 Jan 23.

10.

Biphasic regulation of tissue plasminogen activator activity in ischemic rat brain and in cultured neural cells: essential role of astrocyte-derived plasminogen activator inhibitor-1.

Kim JW, Lee SH, Ko HM, Kwon KJ, Cho KS, Choi CS, Park JH, Kim HY, Lee J, Han SH, Ignarro LJ, Cheong JH, Kim WK, Shin CY.

Neurochem Int. 2011 Feb;58(3):423-33. doi: 10.1016/j.neuint.2010.12.020. Epub 2010 Dec 28.

PMID:
21193004
11.

Neuroprotection by the histone deacetylase inhibitor trichostatin A in a model of lipopolysaccharide-sensitised neonatal hypoxic-ischaemic brain injury.

Fleiss B, Nilsson MK, Blomgren K, Mallard C.

J Neuroinflammation. 2012 Apr 18;9:70. doi: 10.1186/1742-2094-9-70.

12.

[Changes and significance of endogenous tissue plasminogen activators in cerebral hypoxia-ischemia in neonatal rats].

Yu D, Mao M, Lei MY.

Zhongguo Dang Dai Er Ke Za Zhi. 2008 Oct;10(5):651-5. Chinese.

PMID:
18947492
13.
14.
15.

Lipopolysaccharide administration enhances hypoxic-ischemic brain damage in newborn rats.

Yang L, Sameshima H, Ikeda T, Ikenoue T.

J Obstet Gynaecol Res. 2004 Apr;30(2):142-7.

PMID:
15009619
16.

Development of cerebral gray and white matter injury and cerebral inflammation over time after inflammatory perinatal asphyxia.

Bonestroo HJ, Heijnen CJ, Groenendaal F, van Bel F, Nijboer CH.

Dev Neurosci. 2015;37(1):78-94. doi: 10.1159/000368770. Epub 2015 Jan 23.

PMID:
25634435
17.

Peptidylarginine deiminases: novel drug targets for prevention of neuronal damage following hypoxic ischemic insult (HI) in neonates.

Lange S, Rocha-Ferreira E, Thei L, Mawjee P, Bennett K, Thompson PR, Subramanian V, Nicholas AP, Peebles D, Hristova M, Raivich G.

J Neurochem. 2014 Aug;130(4):555-62. doi: 10.1111/jnc.12744. Epub 2014 May 24.

18.

Innate defense regulator peptide 1018 protects against perinatal brain injury.

Bolouri H, Sävman K, Wang W, Thomas A, Maurer N, Dullaghan E, Fjell CD, Ek CJ, Hagberg H, Hancock RE, Brown KL, Mallard C.

Ann Neurol. 2014 Mar;75(3):395-410. doi: 10.1002/ana.24087. Epub 2014 Mar 7.

PMID:
24339166
19.

Lipopolysaccharide sensitizes neonatal hypoxic-ischemic brain injury in a MyD88-dependent manner.

Wang X, Stridh L, Li W, Dean J, Elmgren A, Gan L, Eriksson K, Hagberg H, Mallard C.

J Immunol. 2009 Dec 1;183(11):7471-7. doi: 10.4049/jimmunol.0900762. Epub 2009 Nov 16.

20.

Endotoxin-induced hypoxic-ischemic tolerance is mediated by up-regulation of corticosterone in neonatal rat.

Ikeda T, Yang L, Ikenoue T, Mallard C, Hagberg H.

Pediatr Res. 2006 Jan;59(1):56-60. Epub 2005 Dec 2.

PMID:
16327010

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