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Items: 1 to 20 of 101

1.

Delays and diversions mark the development of B cell responses to Borrelia burgdorferi infection.

Hastey CJ, Elsner RA, Barthold SW, Baumgarth N.

J Immunol. 2012 Jun 1;188(11):5612-22. doi: 10.4049/jimmunol.1103735. Epub 2012 Apr 30.

2.

CD4+ T cells promote antibody production but not sustained affinity maturation during Borrelia burgdorferi infection.

Elsner RA, Hastey CJ, Baumgarth N.

Infect Immun. 2015 Jan;83(1):48-56. doi: 10.1128/IAI.02471-14. Epub 2014 Oct 13.

3.

Suppression of Long-Lived Humoral Immunity Following Borrelia burgdorferi Infection.

Elsner RA, Hastey CJ, Olsen KJ, Baumgarth N.

PLoS Pathog. 2015 Jul 2;11(7):e1004976. doi: 10.1371/journal.ppat.1004976. eCollection 2015 Jul.

4.

Lymphoadenopathy during lyme borreliosis is caused by spirochete migration-induced specific B cell activation.

Tunev SS, Hastey CJ, Hodzic E, Feng S, Barthold SW, Baumgarth N.

PLoS Pathog. 2011 May;7(5):e1002066. doi: 10.1371/journal.ppat.1002066. Epub 2011 May 26.

5.

MyD88- and TRIF-independent induction of type I interferon drives naive B cell accumulation but not loss of lymph node architecture in Lyme disease.

Hastey CJ, Ochoa J, Olsen KJ, Barthold SW, Baumgarth N.

Infect Immun. 2014 Apr;82(4):1548-58. doi: 10.1128/IAI.00969-13. Epub 2014 Jan 22.

6.

Marginal zone B-cell depletion impairs murine host defense against Borrelia burgdorferi infection.

Belperron AA, Dailey CM, Booth CJ, Bockenstedt LK.

Infect Immun. 2007 Jul;75(7):3354-60. Epub 2007 Apr 30.

7.
8.

IL-10 deficiency promotes increased Borrelia burgdorferi clearance predominantly through enhanced innate immune responses.

Lazarus JJ, Meadows MJ, Lintner RE, Wooten RM.

J Immunol. 2006 Nov 15;177(10):7076-85.

9.

Protective antibodies in murine Lyme disease arise independently of CD40 ligand.

Fikrig E, Barthold SW, Chen M, Grewal IS, Craft J, Flavell RA.

J Immunol. 1996 Jul 1;157(1):1-3.

PMID:
8683101
10.

Borrelia burgdorferi adhere to blood vessels in the dura mater and are associated with increased meningeal T cells during murine disseminated borreliosis.

Divan A, Casselli T, Narayanan SA, Mukherjee S, Zawieja DC, Watt JA, Brissette CA, Newell-Rogers MK.

PLoS One. 2018 May 3;13(5):e0196893. doi: 10.1371/journal.pone.0196893. eCollection 2018.

11.
13.

Cyclooxygenase-1 orchestrates germinal center formation and antibody class-switch via regulation of IL-17.

Blaho VA, Buczynski MW, Dennis EA, Brown CR.

J Immunol. 2009 Nov 1;183(9):5644-53. doi: 10.4049/jimmunol.0901499.

14.

A critical role for type I IFN in arthritis development following Borrelia burgdorferi infection of mice.

Miller JC, Ma Y, Bian J, Sheehan KC, Zachary JF, Weis JH, Schreiber RD, Weis JJ.

J Immunol. 2008 Dec 15;181(12):8492-503.

15.

Borrelia burgdorferi, an extracellular pathogen, circumvents osteopontin in inducing an inflammatory cytokine response.

Craig-Mylius K, Weber GF, Coburn J, Glickstein L.

J Leukoc Biol. 2005 May;77(5):710-8. Epub 2005 Feb 4.

PMID:
15695554
16.
17.

Analysis of some peripheral blood lymphocyte subsets in relation to Borrelia burgdorferi antibodies in patients with Lyme disease.

Zajkowska JM, Hermanowska-Szpakowicz T, Wysocka J, Kondrusik M, Pancewicz SA, Grygorczuk S.

Rocz Akad Med Bialymst. 2000;45:184-98.

PMID:
11712430
18.

Dual role for Fcγ receptors in host defense and disease in Borrelia burgdorferi-infected mice.

Belperron AA, Liu N, Booth CJ, Bockenstedt LK.

Front Cell Infect Microbiol. 2014 Jun 11;4:75. doi: 10.3389/fcimb.2014.00075. eCollection 2014.

19.
20.

MicroRNA-146a provides feedback regulation of lyme arthritis but not carditis during infection with Borrelia burgdorferi.

Lochhead RB, Ma Y, Zachary JF, Baltimore D, Zhao JL, Weis JH, O'Connell RM, Weis JJ.

PLoS Pathog. 2014 Jun 26;10(6):e1004212. doi: 10.1371/journal.ppat.1004212. eCollection 2014 Jun.

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