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Items: 1 to 20 of 267


The Entner-Doudoroff pathway is obligatory for gluconate utilization and contributes to the pathogenicity of Vibrio cholerae.

Patra T, Koley H, Ramamurthy T, Ghose AC, Nandy RK.

J Bacteriol. 2012 Jul;194(13):3377-85. doi: 10.1128/JB.06379-11. Epub 2012 Apr 27.


Characterization of the gluconate utilization system of Vibrio cholerae with special reference to virulence modulation.

Roy S, Patra T, Golder T, Chatterjee S, Koley H, Nandy RK.

Pathog Dis. 2016 Nov;74(8). pii: ftw085. Epub 2016 Sep 19.


Effect of anaerobiosis on expression of virulence factors in Vibrio cholerae.

Krishnan HH, Ghosh A, Paul K, Chowdhury R.

Infect Immun. 2004 Jul;72(7):3961-7.


Pleiotropic effects of the twin-arginine translocation system on biofilm formation, colonization, and virulence in Vibrio cholerae.

Zhang L, Zhu Z, Jing H, Zhang J, Xiong Y, Yan M, Gao S, Wu LF, Xu J, Kan B.

BMC Microbiol. 2009 May 31;9:114. doi: 10.1186/1471-2180-9-114.


The global regulator ArcA modulates expression of virulence factors in Vibrio cholerae.

Sengupta N, Paul K, Chowdhury R.

Infect Immun. 2003 Oct;71(10):5583-9.


Role of exopolysaccharide, the rugose phenotype and VpsR in the pathogenesis of epidemic Vibrio cholerae.

Rashid MH, Rajanna C, Zhang D, Pasquale V, Magder LS, Ali A, Dumontet S, Karaolis DK.

FEMS Microbiol Lett. 2004 Jan 15;230(1):105-13.


Role of a sensor histidine kinase ChiS of Vibrio cholerae in pathogenesis.

Chourashi R, Mondal M, Sinha R, Debnath A, Das S, Koley H, Chatterjee NS.

Int J Med Microbiol. 2016 Dec;306(8):657-665. doi: 10.1016/j.ijmm.2016.09.003. Epub 2016 Sep 19.


A bistable switch and anatomical site control Vibrio cholerae virulence gene expression in the intestine.

Nielsen AT, Dolganov NA, Rasmussen T, Otto G, Miller MC, Felt SA, Torreilles S, Schoolnik GK.

PLoS Pathog. 2010 Sep 16;6(9):e1001102. doi: 10.1371/journal.ppat.1001102.


MetR-regulated Vibrio cholerae metabolism is required for virulence.

Bogard RW, Davies BW, Mekalanos JJ.

MBio. 2012 Sep 25;3(5). pii: e00236-12. doi: 10.1128/mBio.00236-12. Print 2012.


Contribution of hemagglutinin/protease and motility to the pathogenesis of El Tor biotype cholera.

Silva AJ, Leitch GJ, Camilli A, Benitez JA.

Infect Immun. 2006 Apr;74(4):2072-9.


Reduced virulence of the Vibrio cholerae fadD mutant is due to induction of the extracytoplasmic stress response.

Chatterjee E, Chowdhury R.

Infect Immun. 2013 Oct;81(10):3935-41. doi: 10.1128/IAI.00722-13. Epub 2013 Aug 5.


Bicarbonate Induces Vibrio cholerae virulence gene expression by enhancing ToxT activity.

Abuaita BH, Withey JH.

Infect Immun. 2009 Sep;77(9):4111-20. doi: 10.1128/IAI.00409-09. Epub 2009 Jun 29. Erratum in: Infect Immun. 2009 Nov;77(11):5202.


Role of melanin pigment in expression of Vibrio cholerae virulence factors.

Valeru SP, Rompikuntal PK, Ishikawa T, Vaitkevicius K, Sjöling A, Dolganov N, Zhu J, Schoolnik G, Wai SN.

Infect Immun. 2009 Mar;77(3):935-42. doi: 10.1128/IAI.00929-08. Epub 2008 Dec 22.


Termination of Vibrio cholerae virulence gene expression is mediated by proteolysis of the major virulence activator, ToxT.

Abuaita BH, Withey JH.

Mol Microbiol. 2011 Sep;81(6):1640-53. doi: 10.1111/j.1365-2958.2011.07798.x. Epub 2011 Aug 22.


Regulation and temporal expression patterns of Vibrio cholerae virulence genes during infection.

Lee SH, Hava DL, Waldor MK, Camilli A.

Cell. 1999 Dec 10;99(6):625-34.

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