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Items: 1 to 20 of 171

1.

Casein kinase 2-mediated synaptic GluN2A up-regulation increases N-methyl-D-aspartate receptor activity and excitability of hypothalamic neurons in hypertension.

Ye ZY, Li L, Li DP, Pan HL.

J Biol Chem. 2012 May 18;287(21):17438-46. doi: 10.1074/jbc.M111.331165. Epub 2012 Apr 3.

2.

Endogenous casein kinase-1 modulates NMDA receptor activity of hypothalamic presympathetic neurons and sympathetic outflow in hypertension.

Li DP, Zhou JJ, Pan HL.

J Physiol. 2015 Oct 1;593(19):4439-52. doi: 10.1113/JP270831. Epub 2015 Aug 18.

3.

Protein kinase CK2 increases glutamatergic input in the hypothalamus and sympathetic vasomotor tone in hypertension.

Ye ZY, Li DP, Li L, Pan HL.

J Neurosci. 2011 Jun 1;31(22):8271-9. doi: 10.1523/JNEUROSCI.1147-11.2011.

4.

Protein kinase CK2 contributes to diminished small conductance Ca2+-activated K+ channel activity of hypothalamic pre-sympathetic neurons in hypertension.

Pachuau J, Li DP, Chen SR, Lee HA, Pan HL.

J Neurochem. 2014 Sep;130(5):657-67. doi: 10.1111/jnc.12758. Epub 2014 May 24.

5.

mGluR5 Upregulation increases excitability of hypothalamic presympathetic neurons through NMDA receptor trafficking in spontaneously hypertensive rats.

Li DP, Zhu LH, Pachuau J, Lee HA, Pan HL.

J Neurosci. 2014 Mar 19;34(12):4309-17. doi: 10.1523/JNEUROSCI.4295-13.2014.

6.

Plasticity of pre- and postsynaptic GABAB receptor function in the paraventricular nucleus in spontaneously hypertensive rats.

Li DP, Yang Q, Pan HM, Pan HL.

Am J Physiol Heart Circ Physiol. 2008 Aug;295(2):H807-15. doi: 10.1152/ajpheart.00259.2008. Epub 2008 Jun 20.

7.
8.

Src Kinases Regulate Glutamatergic Input to Hypothalamic Presympathetic Neurons and Sympathetic Outflow in Hypertension.

Qiao X, Zhou JJ, Li DP, Pan HL.

Hypertension. 2017 Jan;69(1):154-162. Epub 2016 Oct 31.

PMID:
27802416
9.

Regulation of Hypothalamic Presympathetic Neurons and Sympathetic Outflow by Group II Metabotropic Glutamate Receptors in Spontaneously Hypertensive Rats.

Ye ZY, Li DP, Pan HL.

Hypertension. 2013 Aug;62(2):255-62. doi: 10.1161/HYPERTENSIONAHA.113.01466. Epub 2013 May 28.

10.

Pre- and postsynaptic plasticity underlying augmented glutamatergic inputs to hypothalamic presympathetic neurons in spontaneously hypertensive rats.

Li DP, Yang Q, Pan HM, Pan HL.

J Physiol. 2008 Mar 15;586(6):1637-47. doi: 10.1113/jphysiol.2007.149732. Epub 2008 Jan 31.

11.
12.

Increased group I metabotropic glutamate receptor activity in paraventricular nucleus supports elevated sympathetic vasomotor tone in hypertension.

Li DP, Pan HL.

Am J Physiol Regul Integr Comp Physiol. 2010 Aug;299(2):R552-61. doi: 10.1152/ajpregu.00195.2010. Epub 2010 Jun 2.

13.

Apelin-13 and APJ in paraventricular nucleus contribute to hypertension via sympathetic activation and vasopressin release in spontaneously hypertensive rats.

Zhang F, Sun HJ, Xiong XQ, Chen Q, Li YH, Kang YM, Wang JJ, Gao XY, Zhu GQ.

Acta Physiol (Oxf). 2014 Sep;212(1):17-27. doi: 10.1111/apha.12342. Epub 2014 Jul 18.

PMID:
24995933
14.

Plasticity of GABAergic control of hypothalamic presympathetic neurons in hypertension.

Li DP, Pan HL.

Am J Physiol Heart Circ Physiol. 2006 Mar;290(3):H1110-9. Epub 2005 Oct 21.

15.

NKCC1 upregulation disrupts chloride homeostasis in the hypothalamus and increases neuronal activity-sympathetic drive in hypertension.

Ye ZY, Li DP, Byun HS, Li L, Pan HL.

J Neurosci. 2012 Jun 20;32(25):8560-8. doi: 10.1523/JNEUROSCI.1346-12.2012.

16.

Calcineurin inhibitor induces pain hypersensitivity by potentiating pre- and postsynaptic NMDA receptor activity in spinal cords.

Chen SR, Hu YM, Chen H, Pan HL.

J Physiol. 2014 Jan 1;592(1):215-27. doi: 10.1113/jphysiol.2013.263814. Epub 2013 Sep 30.

17.

Exercise training normalizes an increased neuronal excitability of NTS-projecting neurons of the hypothalamic paraventricular nucleus in hypertensive rats.

Stern JE, Sonner PM, Son SJ, Silva FC, Jackson K, Michelini LC.

J Neurophysiol. 2012 May;107(10):2912-21. doi: 10.1152/jn.00884.2011. Epub 2012 Feb 22.

18.

Synaptic and extrasynaptic plasticity in glutamatergic circuits involving dentate granule cells following chronic N-methyl-D-aspartate receptor inhibition.

He S, Shao LR, Wang Y, Bausch SB.

J Neurophysiol. 2013 Mar;109(6):1535-47. doi: 10.1152/jn.00667.2012. Epub 2012 Dec 19.

19.

Casein kinase II inhibition reverses pain hypersensitivity and potentiated spinal N-methyl-D-aspartate receptor activity caused by calcineurin inhibitor.

Hu YM, Chen SR, Chen H, Pan HL.

J Pharmacol Exp Ther. 2014 May;349(2):239-47. doi: 10.1124/jpet.113.212563. Epub 2014 Mar 7.

20.

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