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Items: 1 to 20 of 112

1.

Bovine PrP expression levels in transgenic mice influence transmission characteristics of atypical bovine spongiform encephalopathy.

Wilson R, Hart P, Piccardo P, Hunter N, Casalone C, Baron T, Barron RM.

J Gen Virol. 2012 May;93(Pt 5):1132-40. doi: 10.1099/vir.0.040030-0. Epub 2012 Feb 1.

2.

Presence of subclinical infection in gene-targeted human prion protein transgenic mice exposed to atypical bovine spongiform encephalopathy.

Wilson R, Dobie K, Hunter N, Casalone C, Baron T, Barron RM.

J Gen Virol. 2013 Dec;94(Pt 12):2819-27. doi: 10.1099/vir.0.052738-0. Epub 2013 Sep 17.

PMID:
24045112
3.

Intraspecies transmission of BASE induces clinical dullness and amyotrophic changes.

Lombardi G, Casalone C, D' Angelo A, Gelmetti D, Torcoli G, Barbieri I, Corona C, Fasoli E, Farinazzo A, Fiorini M, Gelati M, Iulini B, Tagliavini F, Ferrari S, Caramelli M, Monaco S, Capucci L, Zanusso G.

PLoS Pathog. 2008 May 23;4(5):e1000075. doi: 10.1371/journal.ppat.1000075.

4.

Transmissibility of H-Type Bovine Spongiform Encephalopathy to Hamster PrP Transgenic Mice.

Okada H, Masujin K, Miyazawa K, Yokoyama T.

PLoS One. 2015 Oct 14;10(10):e0138977. doi: 10.1371/journal.pone.0138977. eCollection 2015.

5.

A bovine prion acquires an epidemic bovine spongiform encephalopathy strain-like phenotype on interspecies transmission.

Béringue V, Andréoletti O, Le Dur A, Essalmani R, Vilotte JL, Lacroux C, Reine F, Herzog L, Biacabé AG, Baron T, Caramelli M, Casalone C, Laude H.

J Neurosci. 2007 Jun 27;27(26):6965-71.

6.

Characterization of Syrian hamster adapted prions derived from L-type and C-type bovine spongiform encephalopathies.

Shu Y, Masujin K, Okada H, Iwamaru Y, Imamura M, Matsuura Y, Mohri S, Yokoyama T.

Prion. 2011 Apr-Jun;5(2):103-8. Epub 2011 Apr 1.

7.

Transmission and characterization of bovine spongiform encephalopathy sources in two ovine transgenic mouse lines (TgOvPrP4 and TgOvPrP59).

Cordier C, Bencsik A, Philippe S, Bétemps D, Ronzon F, Calavas D, Crozet C, Baron T.

J Gen Virol. 2006 Dec;87(Pt 12):3763-71.

PMID:
17098996
8.

Unique properties of the classical bovine spongiform encephalopathy strain and its emergence from H-type bovine spongiform encephalopathy substantiated by VM transmission studies.

Bencsik A, Leboidre M, Debeer S, Aufauvre C, Baron T.

J Neuropathol Exp Neurol. 2013 Mar;72(3):211-8. doi: 10.1097/NEN.0b013e318285c7f9.

PMID:
23399901
9.

Guinea Pig Prion Protein Supports Rapid Propagation of Bovine Spongiform Encephalopathy and Variant Creutzfeldt-Jakob Disease Prions.

Watts JC, Giles K, Saltzberg DJ, Dugger BN, Patel S, Oehler A, Bhardwaj S, Sali A, Prusiner SB.

J Virol. 2016 Oct 14;90(21):9558-9569. Print 2016 Nov 1.

10.

Conversion of the BASE prion strain into the BSE strain: the origin of BSE?

Capobianco R, Casalone C, Suardi S, Mangieri M, Miccolo C, Limido L, Catania M, Rossi G, Di Fede G, Giaccone G, Bruzzone MG, Minati L, Corona C, Acutis P, Gelmetti D, Lombardi G, Groschup MH, Buschmann A, Zanusso G, Monaco S, Caramelli M, Tagliavini F.

PLoS Pathog. 2007 Mar;3(3):e31.

11.

Bovine spongiform encephalopathy induces misfolding of alleged prion-resistant species cellular prion protein without altering its pathobiological features.

Vidal E, Fernández-Borges N, Pintado B, Ordóñez M, Márquez M, Fondevila D, Torres JM, Pumarola M, Castilla J.

J Neurosci. 2013 May 1;33(18):7778-86. doi: 10.1523/JNEUROSCI.0244-13.2013.

12.

Evaluation of the human transmission risk of an atypical bovine spongiform encephalopathy prion strain.

Kong Q, Zheng M, Casalone C, Qing L, Huang S, Chakraborty B, Wang P, Chen F, Cali I, Corona C, Martucci F, Iulini B, Acutis P, Wang L, Liang J, Wang M, Li X, Monaco S, Zanusso G, Zou WQ, Caramelli M, Gambetti P.

J Virol. 2008 Apr;82(7):3697-701. doi: 10.1128/JVI.02561-07. Epub 2008 Jan 30.

13.

Absence of Evidence for a Causal Link between Bovine Spongiform Encephalopathy Strain Variant L-BSE and Known Forms of Sporadic Creutzfeldt-Jakob Disease in Human PrP Transgenic Mice.

Jaumain E, Quadrio I, Herzog L, Reine F, Rezaei H, Andréoletti O, Laude H, Perret-Liaudet A, Haïk S, Béringue V.

J Virol. 2016 Nov 14;90(23):10867-10874. Print 2016 Dec 1.

14.

Increased susceptibility of human-PrP transgenic mice to bovine spongiform encephalopathy infection following passage in sheep.

Plinston C, Hart P, Chong A, Hunter N, Foster J, Piccardo P, Manson JC, Barron RM.

J Virol. 2011 Feb;85(3):1174-81. doi: 10.1128/JVI.01578-10. Epub 2010 Nov 17.

15.

Emergence of a novel bovine spongiform encephalopathy (BSE) prion from an atypical H-type BSE.

Masujin K, Okada H, Miyazawa K, Matsuura Y, Imamura M, Iwamaru Y, Murayama Y, Yokoyama T.

Sci Rep. 2016 Mar 7;6:22753. doi: 10.1038/srep22753.

16.

Enhanced virulence of sheep-passaged bovine spongiform encephalopathy agent is revealed by decreased polymorphism barriers in prion protein conversion studies.

Priem J, Langeveld JP, van Keulen LJ, van Zijderveld FG, Andreoletti O, Bossers A.

J Virol. 2014 Mar;88(5):2903-12. doi: 10.1128/JVI.02446-13. Epub 2013 Dec 26.

17.

Atypical scrapie prions from sheep and lack of disease in transgenic mice overexpressing human prion protein.

Wadsworth JD, Joiner S, Linehan JM, Balkema-Buschmann A, Spiropoulos J, Simmons MM, Griffiths PC, Groschup MH, Hope J, Brandner S, Asante EA, Collinge J.

Emerg Infect Dis. 2013 Nov;19(11):1731-9. doi: 10.3201/eid1911.121341.

18.

Acquired transmissibility of sheep-passaged L-type bovine spongiform encephalopathy prion to wild-type mice.

Okada H, Masujin K, Miyazawa K, Yokoyama T.

Vet Res. 2015 Jul 13;46:81. doi: 10.1186/s13567-015-0211-2.

19.

Phenotypic similarity of transmissible mink encephalopathy in cattle and L-type bovine spongiform encephalopathy in a mouse model.

Baron T, Bencsik A, Biacabe AG, Morignat E, Bessen RA.

Emerg Infect Dis. 2007 Dec;13(12):1887-94. doi: 10.3201/eid1312.070635.

20.

Elements modulating the prion species barrier and its passage consequences.

Torres JM, Espinosa JC, Aguilar-Calvo P, Herva ME, Relaño-Ginés A, Villa-Diaz A, Morales M, Parra B, Alamillo E, Brun A, Castilla J, Molina S, Hawkins SA, Andreoletti O.

PLoS One. 2014 Mar 7;9(3):e89722. doi: 10.1371/journal.pone.0089722. eCollection 2014.

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