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Items: 1 to 20 of 105

1.

CD36 overexpression predisposes to arrhythmias but reduces infarct size in spontaneously hypertensive rats: gene expression profile analysis.

Neckář J, Šilhavy J, Zídek V, Landa V, Mlejnek P, Šimáková M, Seidman JG, Seidman C, Kazdová L, Klevstig M, Novák F, Vecka M, Papoušek F, Houštěk J, Drahota Z, Kurtz TW, Kolář F, Pravenec M.

Physiol Genomics. 2012 Feb 1;44(2):173-82. doi: 10.1152/physiolgenomics.00083.2011.

2.

Transgenic rescue of defective Cd36 enhances myocardial adenylyl cyclase signaling in spontaneously hypertensive rats.

Klevstig M, Manakov D, Kasparova D, Brabcova I, Papousek F, Zurmanova J, Zidek V, Silhavy J, Neckar J, Pravenec M, Kolar F, Novakova O, Novotny J.

Pflugers Arch. 2013 Oct;465(10):1477-86. doi: 10.1007/s00424-013-1281-5.

PMID:
23636771
3.

Defective fatty acid uptake in the spontaneously hypertensive rat is a primary determinant of altered glucose metabolism, hyperinsulinemia, and myocardial hypertrophy.

Hajri T, Ibrahimi A, Coburn CT, Knapp FF Jr, Kurtz T, Pravenec M, Abumrad NA.

J Biol Chem. 2001 Jun 29;276(26):23661-6.

4.

The benefit of medium-chain triglyceride therapy on the cardiac function of SHRs is associated with a reversal of metabolic and signaling alterations.

Iemitsu M, Shimojo N, Maeda S, Irukayama-Tomobe Y, Sakai S, Ohkubo T, Tanaka Y, Miyauchi T.

Am J Physiol Heart Circ Physiol. 2008 Jul;295(1):H136-44. doi: 10.1152/ajpheart.01417.2006.

5.

Transgenic expression of CD36 in the spontaneously hypertensive rat is associated with amelioration of metabolic disturbances but has no effect on hypertension.

Pravenec M, Landa V, Zídek V, Musilová A, Kazdová L, Qi N, Wang J, St Lezin E, Kurtz TW.

Physiol Res. 2003;52(6):681-8.

6.

Increased myocardial vulnerability to ischemia-reperfusion injury in the presence of left ventricular hypertrophy.

Mølgaard S, Faricelli B, Salomonsson M, Engstrøm T, Treiman M.

J Hypertens. 2016 Mar;34(3):513-23; discussion 523. doi: 10.1097/HJH.0000000000000826.

PMID:
26820478
7.

Role of FAT/CD36 in novel PKC isoform activation in heart of spontaneously hypertensive rats.

Klevstig MJ, Markova I, Burianova J, Kazdova L, Pravenec M, Novakova O, Novak F.

Mol Cell Biochem. 2011 Nov;357(1-2):163-9. doi: 10.1007/s11010-011-0886-2.

PMID:
21625957
8.

Cd36 and molecular mechanisms of insulin resistance in the stroke-prone spontaneously hypertensive rat.

Collison M, Glazier AM, Graham D, Morton JJ, Dominiczak MH, Aitman TJ, Connell JM, Gould GW, Dominiczak AF.

Diabetes. 2000 Dec;49(12):2222-6.

PMID:
11118030
9.

Alterations in the cardiac proteome of the spontaneously hypertensive rat induced by transgenic expression of CD36.

Manakov D, Ujcikova H, Pravenec M, Novotny J.

J Proteomics. 2016 Aug 11;145:177-86. doi: 10.1016/j.jprot.2016.04.041.

PMID:
27132684
10.

Gene-environment interactions in wet beriberi: effects of thiamine depletion in CD36-defect rats.

Tanaka T, Kono T, Terasaki F, Kintaka T, Sohmiya K, Mishima T, Kitaura Y.

Am J Physiol Heart Circ Physiol. 2003 Oct;285(4):H1546-53.

11.

Cardiac hypertrophy by hypertension and exercise training exhibits different gene expression of enzymes in energy metabolism.

Iemitsu M, Miyauchi T, Maeda S, Sakai S, Fujii N, Miyazaki H, Kakinuma Y, Matsuda M, Yamaguchi I.

Hypertens Res. 2003 Oct;26(10):829-37.

12.

Metabolic effects of telmisartan in spontaneously hypertensive rats.

Li YQ, Ji H, Zhang YH, Ding DY, Ye XL.

Naunyn Schmiedebergs Arch Pharmacol. 2006 Jul;373(4):264-70.

PMID:
16715213
13.

Ischemic tolerance of rat hearts in acute and chronic phases of experimental diabetes.

Ravingerová T, Neckár J, Kolár F.

Mol Cell Biochem. 2003 Jul;249(1-2):167-74.

PMID:
12956412
14.

FAT/CD36 expression is not ablated in spontaneously hypertensive rats.

Bonen A, Han XX, Tandon NN, Glatz JF, Lally J, Snook LA, Luiken JJ.

J Lipid Res. 2009 Apr;50(4):740-8. doi: 10.1194/jlr.M800237-JLR200.

15.

Effects of medium-chain triglyceride (MCT) application to SHR on cardiac function, hypertrophy and expression of endothelin-1 mRNA and other genes.

Shimojo N, Miyauchi T, Iemitsu M, Irukayama-Tomobe Y, Maeda S, Ohkubo T, Tanaka Y, Goto K, Yamaguchi I.

J Cardiovasc Pharmacol. 2004 Nov;44 Suppl 1:S181-5.

PMID:
15838274
16.

CD36-deficient congenic strains show improved glucose tolerance and distinct shifts in metabolic and transcriptomic profiles.

Šedová L, Liška F, Křenová D, Kazdová L, Tremblay J, Krupková M, Corbeil G, Hamet P, Křen V, Šeda O.

Heredity (Edinb). 2012 Jul;109(1):63-70. doi: 10.1038/hdy.2012.14.

17.

Identification of Cd36 (Fat) as an insulin-resistance gene causing defective fatty acid and glucose metabolism in hypertensive rats.

Aitman TJ, Glazier AM, Wallace CA, Cooper LD, Norsworthy PJ, Wahid FN, Al-Majali KM, Trembling PM, Mann CJ, Shoulders CC, Graf D, St Lezin E, Kurtz TW, Kren V, Pravenec M, Ibrahimi A, Abumrad NA, Stanton LW, Scott J.

Nat Genet. 1999 Jan;21(1):76-83.

PMID:
9916795
18.

Post-translational modifications, a key process in CD36 function: lessons from the spontaneously hypertensive rat heart.

Lauzier B, Merlen C, Vaillant F, McDuff J, Bouchard B, Beguin PC, Dolinsky VW, Foisy S, Villeneuve LR, Labarthe F, Dyck JR, Allen BG, Charron G, Des Rosiers C.

J Mol Cell Cardiol. 2011 Jul;51(1):99-108. doi: 10.1016/j.yjmcc.2011.04.001.

PMID:
21510957
19.

Induction of Cd36 expression elicited by fish oil PUFA in spontaneously hypertensive rats.

Alexander Aguilera A, Hernández Díaz G, Lara Barcelata M, Angulo Guerrero O, Oliart Ros RM.

J Nutr Biochem. 2006 Nov;17(11):760-5.

PMID:
16517147
20.

Ventricular arrhythmias following coronary artery occlusion in rats: is the diabetic heart less or more sensitive to ischaemia?

Ravingerova T, Neckar J, Kolar F, Stetka R, Volkovova K, Ziegelhöffer A, Styk J.

Basic Res Cardiol. 2001 Apr;96(2):160-8.

PMID:
11327334
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