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Items: 1 to 20 of 281

1.

In vivo expression of Helicobacter pylori virulence genes in patients with gastritis, ulcer, and gastric cancer.

Avilés-Jiménez F, Reyes-Leon A, Nieto-Patlán E, Hansen LM, Burgueño J, Ramos IP, Camorlinga-Ponce M, Bermúdez H, Blancas JM, Cabrera L, Ribas-Aparicio RM, Solnick JV, Torres-López J.

Infect Immun. 2012 Feb;80(2):594-601. doi: 10.1128/IAI.05845-11. Epub 2011 Nov 28.

3.

Topographic expression of MUC5AC and MUC6 in the gastric mucosa infected by Helicobacter pylori and in associated diseases.

Marques T, David L, Reis C, Nogueira A.

Pathol Res Pract. 2005;201(10):665-72. Epub 2005 Sep 26.

PMID:
16325508
4.
5.

Functional analysis of the cag pathogenicity island in Helicobacter pylori isolates from patients with gastritis, peptic ulcer, and gastric cancer.

Backert S, Schwarz T, Miehlke S, Kirsch C, Sommer C, Kwok T, Gerhard M, Goebel UB, Lehn N, Koenig W, Meyer TF.

Infect Immun. 2004 Feb;72(2):1043-56.

6.

The genetic diversity of Helicobacter pylori virulence genes is not associated with gastric atrophy progression.

Kita M, Yokota K, Okada H, Take S, Takenaka R, Kawahara Y, Oguma K, Matsushita O, Yamamoto K.

Acta Med Okayama. 2013;67(2):93-8.

8.

Disease-specific Helicobacter pylori virulence factors: the unfulfilled promise.

Graham DY, Yamaoka Y.

Helicobacter. 2000;5 Suppl 1:S3-9; discussion S27-31. Review.

PMID:
10828748
9.

Helicobacter pylori cag pathogenicity island is associated with enhanced interleukin-8 expression in human gastric mucosa.

Orsini B, Ciancio G, Censini S, Surrenti E, Pellegrini G, Milani S, Herbst H, Amorosi A, Surrenti C.

Dig Liver Dis. 2000 Aug-Sep;32(6):458-67.

PMID:
11057919
10.

Higher number of Helicobacter pylori CagA EPIYA C phosphorylation sites increases the risk of gastric cancer, but not duodenal ulcer.

Batista SA, Rocha GA, Rocha AM, Saraiva IE, Cabral MM, Oliveira RC, Queiroz DM.

BMC Microbiol. 2011 Mar 24;11:61. doi: 10.1186/1471-2180-11-61.

11.
12.

Association of polymorphisms in virulence factor of Helicobacter pylori and gastroduodenal diseases in South Korea.

Kim JY, Kim N, Nam RH, Suh JH, Chang H, Lee JW, Kim YS, Kim JM, Choi JW, Park JG, Lee YS, Lee DH, Jung HC.

J Gastroenterol Hepatol. 2014 May;29(5):984-91. doi: 10.1111/jgh.12509.

PMID:
24372834
13.

Helicobacter pylori vacA, iceA, and cagA status and pattern of gastritis in patients with malignant and benign gastroduodenal disease.

Miehlke S, Yu J, Schuppler M, Frings C, Kirsch C, Negraszus N, Morgner A, Stolte M, Ehninger G, Bayerdörffer E.

Am J Gastroenterol. 2001 Apr;96(4):1008-13.

PMID:
11316139
14.

Helicobacter pylori babA2, cagA, and s1 vacA genes work synergistically in causing intestinal metaplasia.

Zambon CF, Navaglia F, Basso D, Rugge M, Plebani M.

J Clin Pathol. 2003 Apr;56(4):287-91.

15.
16.

Amino acid polymorphisms flanking the EPIYA-A motif of Helicobacter pylori CagA C-terminal region is associated with gastric cancer in east China: experience from a single center.

Chen CY, Wang FY, Wan HJ, Jin XX, Wei J, Wang ZK, Liu C, Lu H, Shi H, Li DH, Liu J.

J Dig Dis. 2013 Jul;14(7):358-65. doi: 10.1111/1751-2980.12056.

PMID:
23517408
17.

Clinical and histological associations of cagA and vacA genotypes in Helicobacter pylori gastritis.

Warburton VJ, Everett S, Mapstone NP, Axon AT, Hawkey P, Dixon MF.

J Clin Pathol. 1998 Jan;51(1):55-61.

18.

The EPIYA-ABCC motif pattern in CagA of Helicobacter pylori is associated with peptic ulcer and gastric cancer in Mexican population.

Beltrán-Anaya FO, Poblete TM, Román-Román A, Reyes S, de Sampedro J, Peralta-Zaragoza O, Rodríguez MÁ, del Moral-Hernández O, Illades-Aguiar B, Fernández-Tilapa G.

BMC Gastroenterol. 2014 Dec 24;14:223. doi: 10.1186/s12876-014-0223-9.

19.

TNF-alpha expression in gastric mucosa of individuals infected with different virulent Helicobacter pylori strains.

Zalewska-Ziob M, Adamek B, Strzelczyk JK, Gawron K, Jarzab B, Gubała E, Kula D, Krakowczyk Ł, Sieroń A, Wiczkowski A.

Med Sci Monit. 2009 Jun;15(6):BR166-71.

PMID:
19478694
20.

Determination of Helicobacter pylori virulence by simple gene analysis of the cag pathogenicity island.

Ikenoue T, Maeda S, Ogura K, Akanuma M, Mitsuno Y, Imai Y, Yoshida H, Shiratori Y, Omata M.

Clin Diagn Lab Immunol. 2001 Jan;8(1):181-6.

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