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Items: 1 to 20 of 114

1.

β-N-methylamino-L-alanine induces neurological deficits and shortened life span in Drosophila.

Zhou X, Escala W, Papapetropoulos S, Zhai RG.

Toxins (Basel). 2010 Nov;2(11):2663-79. doi: 10.3390/toxins2112663. Epub 2010 Nov 3.

2.

beta-N-methylamino-l-alanine induces oxidative stress and glutamate release through action on system Xc(-).

Liu X, Rush T, Zapata J, Lobner D.

Exp Neurol. 2009 Jun;217(2):429-33. doi: 10.1016/j.expneurol.2009.04.002. Epub 2009 Apr 15.

PMID:
19374900
3.

Transfer of developmental neurotoxin β-N-methylamino-l-alanine (BMAA) via milk to nursed offspring: Studies by mass spectrometry and image analysis.

Andersson M, Karlsson O, Banack SA, Brandt I.

Toxicol Lett. 2016 Sep 6;258:108-14. doi: 10.1016/j.toxlet.2016.06.015. Epub 2016 Jun 16.

PMID:
27320960
4.

Beyond Guam: the cyanobacteria/BMAA hypothesis of the cause of ALS and other neurodegenerative diseases.

Bradley WG, Mash DC.

Amyotroph Lateral Scler. 2009;10 Suppl 2:7-20. doi: 10.3109/17482960903286009.

PMID:
19929726
5.

Long-term cognitive impairments in adult rats treated neonatally with beta-N-Methylamino-L-Alanine.

Karlsson O, Roman E, Brittebo EB.

Toxicol Sci. 2009 Nov;112(1):185-95. doi: 10.1093/toxsci/kfp196. Epub 2009 Aug 19.

PMID:
19692667
6.

The cyanobacteria derived toxin Beta-N-methylamino-L-alanine and amyotrophic lateral sclerosis.

Banack SA, Caller TA, Stommel EW.

Toxins (Basel). 2010 Dec;2(12):2837-50. doi: 10.3390/toxins2122837. Epub 2010 Dec 20. Review.

7.

A critical review of the postulated role of the non-essential amino acid, β-N-methylamino-L-alanine, in neurodegenerative disease in humans.

Chernoff N, Hill DJ, Diggs DL, Faison BD, Francis BM, Lang JR, Larue MM, Le TT, Loftin KA, Lugo JN, Schmid JE, Winnik WM.

J Toxicol Environ Health B Crit Rev. 2017;20(4):1-47. doi: 10.1080/10937404.2017.1297592. Epub 2017 Jun 9. Review.

PMID:
28598725
8.

Animal models of BMAA neurotoxicity: a critical review.

Karamyan VT, Speth RC.

Life Sci. 2008 Jan 30;82(5-6):233-46. doi: 10.1016/j.lfs.2007.11.020. Epub 2007 Dec 7. Review.

PMID:
18191417
9.

Metabotropic glutamate receptor 1 mediates the electrophysiological and toxic actions of the cycad derivative beta-N-Methylamino-L-alanine on substantia nigra pars compacta DAergic neurons.

Cucchiaroni ML, Viscomi MT, Bernardi G, Molinari M, Guatteo E, Mercuri NB.

J Neurosci. 2010 Apr 14;30(15):5176-88. doi: 10.1523/JNEUROSCI.5351-09.2010.

10.

Is there a role for naturally occurring cyanobacterial toxins in neurodegeneration? The beta-N-methylamino-L-alanine (BMAA) paradigm.

Papapetropoulos S.

Neurochem Int. 2007 Jun;50(7-8):998-1003. Epub 2007 Jan 14. Review.

PMID:
17296249
11.

β-N-methylamino-l-alanine causes neurological and pathological phenotypes mimicking Amyotrophic Lateral Sclerosis (ALS): the first step towards an experimental model for sporadic ALS.

de Munck E, Muñoz-Sáez E, Miguel BG, Solas MT, Ojeda I, Martínez A, Gil C, Arahuetes RM.

Environ Toxicol Pharmacol. 2013 Sep;36(2):243-55. doi: 10.1016/j.etap.2013.04.007. Epub 2013 Apr 25.

PMID:
23688553
12.

Detection of the neurotoxin BMAA within cyanobacteria isolated from freshwater in China.

Li A, Tian Z, Li J, Yu R, Banack SA, Wang Z.

Toxicon. 2010 May;55(5):947-53. doi: 10.1016/j.toxicon.2009.09.023. Epub 2009 Oct 12.

PMID:
19822166
13.

Intrathecal infusion of BMAA induces selective motor neuron damage and astrogliosis in the ventral horn of the spinal cord.

Yin HZ, Yu S, Hsu CI, Liu J, Acab A, Wu R, Tao A, Chiang BJ, Weiss JH.

Exp Neurol. 2014 Nov;261:1-9. doi: 10.1016/j.expneurol.2014.06.003. Epub 2014 Jun 8.

14.

The cyanobacterial amino acid β-N-methylamino-l-alanine perturbs the intermediary metabolism in neonatal rats.

Engskog MK, Karlsson O, Haglöf J, Elmsjö A, Brittebo E, Arvidsson T, Pettersson C.

Toxicology. 2013 Oct 4;312:6-11. doi: 10.1016/j.tox.2013.07.010. Epub 2013 Jul 22.

PMID:
23886855
15.

L-BMAA induced ER stress and enhanced caspase 12 cleavage in human neuroblastoma SH-SY5Y cells at low nonexcitotoxic concentrations.

Okle O, Stemmer K, Deschl U, Dietrich DR.

Toxicol Sci. 2013 Jan;131(1):217-24. doi: 10.1093/toxsci/kfs291. Epub 2012 Oct 9.

PMID:
23047912
16.

Cyanobacterial neurotoxin BMAA in ALS and Alzheimer's disease.

Pablo J, Banack SA, Cox PA, Johnson TE, Papapetropoulos S, Bradley WG, Buck A, Mash DC.

Acta Neurol Scand. 2009 Oct;120(4):216-25. doi: 10.1111/j.1600-0404.2008.01150.x. Epub 2009 Feb 26.

PMID:
19254284
17.

Biomagnification of cyanobacterial neurotoxins and neurodegenerative disease among the Chamorro people of Guam.

Cox PA, Banack SA, Murch SJ.

Proc Natl Acad Sci U S A. 2003 Nov 11;100(23):13380-3.

18.
19.

The cyanobacterial neurotoxin β-N-methylamino-l-alanine (BMAA) induces neuronal and behavioral changes in honeybees.

Okle O, Rath L, Galizia CG, Dietrich DR.

Toxicol Appl Pharmacol. 2013 Jul 1;270(1):9-15. doi: 10.1016/j.taap.2013.04.003. Epub 2013 Apr 13.

PMID:
23591064
20.

Does α-amino-β-methylaminopropionic acid (BMAA) play a role in neurodegeneration?

Chiu AS, Gehringer MM, Welch JH, Neilan BA.

Int J Environ Res Public Health. 2011 Sep;8(9):3728-46. doi: 10.3390/ijerph8093728. Epub 2011 Sep 16. Review.

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