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Items: 1 to 20 of 138

1.

Interleukin-10 alters effector functions of multiple genes induced by Borrelia burgdorferi in macrophages to regulate Lyme disease inflammation.

Gautam A, Dixit S, Philipp MT, Singh SR, Morici LA, Kaushal D, Dennis VA.

Infect Immun. 2011 Dec;79(12):4876-92. doi: 10.1128/IAI.05451-11. Epub 2011 Sep 26.

2.

Different patterns of expression and of IL-10 modulation of inflammatory mediators from macrophages of Lyme disease-resistant and -susceptible mice.

Gautam A, Dixit S, Embers M, Gautam R, Philipp MT, Singh SR, Morici L, Dennis VA.

PLoS One. 2012;7(9):e43860. Epub 2012 Sep 14.

3.
4.

MicroRNA-146a provides feedback regulation of lyme arthritis but not carditis during infection with Borrelia burgdorferi.

Lochhead RB, Ma Y, Zachary JF, Baltimore D, Zhao JL, Weis JH, O'Connell RM, Weis JJ.

PLoS Pathog. 2014 Jun 26;10(6):e1004212. doi: 10.1371/journal.ppat.1004212. eCollection 2014 Jun.

5.
6.

Borrelia burgdorferi induces the production and release of proinflammatory cytokines in canine synovial explant cultures.

Straubinger RK, Straubinger AF, Summers BA, Erb HN, Härter L, Appel MJ.

Infect Immun. 1998 Jan;66(1):247-58.

7.

Live Borrelia burgdorferi spirochetes elicit inflammatory mediators from human monocytes via the Toll-like receptor signaling pathway.

Dennis VA, Dixit S, O'Brien SM, Alvarez X, Pahar B, Philipp MT.

Infect Immun. 2009 Mar;77(3):1238-45. doi: 10.1128/IAI.01078-08. Epub 2009 Jan 12.

8.

Borrelia burgdorferi stimulation of chemokine secretion by cells of monocyte lineage in patients with Lyme arthritis.

Shin JJ, Strle K, Glickstein LJ, Luster AD, Steere AC.

Arthritis Res Ther. 2010;12(5):R168. doi: 10.1186/ar3128. Epub 2010 Sep 9.

9.

Borrelia burgdorferi, an extracellular pathogen, circumvents osteopontin in inducing an inflammatory cytokine response.

Craig-Mylius K, Weber GF, Coburn J, Glickstein L.

J Leukoc Biol. 2005 May;77(5):710-8. Epub 2005 Feb 4.

PMID:
15695554
10.

Borrelia burgdorferi RST1 (OspC type A) genotype is associated with greater inflammation and more severe Lyme disease.

Strle K, Jones KL, Drouin EE, Li X, Steere AC.

Am J Pathol. 2011 Jun;178(6):2726-39. doi: 10.1016/j.ajpath.2011.02.018.

11.

Autocrine and exocrine regulation of interleukin-10 production in THP-1 cells stimulated with Borrelia burgdorferi lipoproteins.

Giambartolomei GH, Dennis VA, Lasater BL, Murthy PK, Philipp MT.

Infect Immun. 2002 Apr;70(4):1881-8.

12.

Flavonoid naringenin: a potential immunomodulator for Chlamydia trachomatis inflammation.

Yilma AN, Singh SR, Morici L, Dennis VA.

Mediators Inflamm. 2013;2013:102457. doi: 10.1155/2013/102457. Epub 2013 May 23.

13.

Interaction of the Lyme disease spirochete Borrelia burgdorferi with brain parenchyma elicits inflammatory mediators from glial cells as well as glial and neuronal apoptosis.

Ramesh G, Borda JT, Dufour J, Kaushal D, Ramamoorthy R, Lackner AA, Philipp MT.

Am J Pathol. 2008 Nov;173(5):1415-27. doi: 10.2353/ajpath.2008.080483. Epub 2008 Oct 2.

14.

The Lyme disease spirochete Borrelia burgdorferi utilizes multiple ligands, including RNA, for interferon regulatory factor 3-dependent induction of type I interferon-responsive genes.

Miller JC, Maylor-Hagen H, Ma Y, Weis JH, Weis JJ.

Infect Immun. 2010 Jul;78(7):3144-53. doi: 10.1128/IAI.01070-09. Epub 2010 Apr 19.

15.

Regulation of hyaluronan-induced chemokine gene expression by IL-10 and IFN-gamma in mouse macrophages.

Horton MR, Burdick MD, Strieter RM, Bao C, Noble PW.

J Immunol. 1998 Mar 15;160(6):3023-30.

16.

The Lyme disease spirochete Borrelia burgdorferi induces inflammation and apoptosis in cells from dorsal root ganglia.

Ramesh G, Santana-Gould L, Inglis FM, England JD, Philipp MT.

J Neuroinflammation. 2013 Jul 18;10:88. doi: 10.1186/1742-2094-10-88.

17.

Borrelia burgdorferi elicited-IL-10 suppresses the production of inflammatory mediators, phagocytosis, and expression of co-stimulatory receptors by murine macrophages and/or dendritic cells.

Chung Y, Zhang N, Wooten RM.

PLoS One. 2013 Dec 19;8(12):e84980. doi: 10.1371/journal.pone.0084980. eCollection 2013. Erratum in: PLoS One. 2014;9(1). doi:10.1371/annotation/2ce59bc4-fcf0-498f-86f0-376432428bf4. PLoS One. 2014;9(1). doi:10.1371/annotation/680090aa-3e1b-4135-94d6-8082c09180d4.

18.

Activation of human monocytes by live Borrelia burgdorferi generates TLR2-dependent and -independent responses which include induction of IFN-beta.

Salazar JC, Duhnam-Ems S, La Vake C, Cruz AR, Moore MW, Caimano MJ, Velez-Climent L, Shupe J, Krueger W, Radolf JD.

PLoS Pathog. 2009 May;5(5):e1000444. doi: 10.1371/journal.ppat.1000444. Epub 2009 May 22.

19.

IFN-gamma alters the response of Borrelia burgdorferi-activated endothelium to favor chronic inflammation.

Dame TM, Orenzoff BL, Palmer LE, Furie MB.

J Immunol. 2007 Jan 15;178(2):1172-9.

20.

Concentrations of macrophage inflammatory proteins MIP-1alpha and MIP-1beta and interleukin 8 (il-8) in lyme borreliosis.

Grygorczuk S, Pancewicz S, Zajkowska J, Kondrusik M, Rwierzbińska R, Hermanowska-Szpakowicz T.

Infection. 2004 Dec;32(6):350-5.

PMID:
15597225

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