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Items: 1 to 20 of 105

1.

Functional PU.1 in macrophages has a pivotal role in NF-κB activation and neutrophilic lung inflammation during endotoxemia.

Karpurapu M, Wang X, Deng J, Park H, Xiao L, Sadikot RT, Frey RS, Maus UA, Park GY, Scott EW, Christman JW.

Blood. 2011 Nov 10;118(19):5255-66. doi: 10.1182/blood-2011-03-341123. Epub 2011 Sep 21.

2.

Myeloid cells control termination of lung inflammation through the NF-kappaB pathway.

Han W, Joo M, Everhart MB, Christman JW, Yull FE, Blackwell TS.

Am J Physiol Lung Cell Mol Physiol. 2009 Mar;296(3):L320-7. doi: 10.1152/ajplung.90485.2008. Epub 2008 Dec 19.

3.

GM-CSF regulates a PU.1-dependent transcriptional program determining the pulmonary response to LPS.

Berclaz PY, Carey B, Fillipi MD, Wernke-Dollries K, Geraci N, Cush S, Richardson T, Kitzmiller J, O'connor M, Hermoyian C, Korfhagen T, Whitsett JA, Trapnell BC.

Am J Respir Cell Mol Biol. 2007 Jan;36(1):114-21. Epub 2006 Aug 17.

4.

Both PU.1 and nuclear factor-kappa B mediate lipopolysaccharide- induced HIV-1 long terminal repeat transcription in macrophages.

Lodie TA, Reiner M, Coniglio S, Viglianti G, Fenton MJ.

J Immunol. 1998 Jul 1;161(1):268-76.

5.

Bone marrow and nonbone marrow Toll like receptor 4 regulate acute hepatic injury induced by endotoxemia.

Hochhauser E, Avlas O, Fallach R, Bachmetov L, Zemel R, Pappo O, Shainberg A, Ben Ari Z.

PLoS One. 2013 Aug 15;8(8):e73041. doi: 10.1371/journal.pone.0073041. eCollection 2013.

6.

Apolipoprotein E enhances microRNA-146a in monocytes and macrophages to suppress nuclear factor-κB-driven inflammation and atherosclerosis.

Li K, Ching D, Luk FS, Raffai RL.

Circ Res. 2015 Jun 19;117(1):e1-e11. doi: 10.1161/CIRCRESAHA.117.305844. Epub 2015 Apr 22.

7.

Myeloid-derived NF-κB negative regulation of PU.1 and c/EBP-β-driven pro-inflammatory cytokine production restrains LPS-induced shock.

Vanoni S, Tsai YT, Waddell A, Waggoner L, Klarquist J, Divanovic S, Hoebe K, Steinbrecher KA, Hogan SP.

Innate Immun. 2017 Feb;23(2):175-187. doi: 10.1177/1753425916681444. Epub 2016 Dec 8.

8.

Suppression of PU.1-linked TLR4 expression by cilostazol with decrease of cytokine production in macrophages from patients with rheumatoid arthritis.

Park SY, Lee SW, Baek SH, Lee CW, Lee WS, Rhim BY, Hong KW, Kim CD.

Br J Pharmacol. 2013 Mar;168(6):1401-11. doi: 10.1111/bph.12021.

9.

Lipopolysaccharide suppresses RANK gene expression in macrophages by down-regulating PU.1 and MITF.

Ishii J, Kitazawa R, Mori K, McHugh KP, Morii E, Kondo T, Kitazawa S.

J Cell Biochem. 2008 Oct 15;105(3):896-904. doi: 10.1002/jcb.21886.

PMID:
18759249
10.

The transcription factor PU.1 promotes alternative macrophage polarization and asthmatic airway inflammation.

Qian F, Deng J, Lee YG, Zhu J, Karpurapu M, Chung S, Zheng JN, Xiao L, Park GY, Christman JW.

J Mol Cell Biol. 2015 Dec;7(6):557-67. doi: 10.1093/jmcb/mjv042. Epub 2015 Jun 22.

11.

Echinocystic acid ameliorates lung inflammation in mice and alveolar macrophages by inhibiting the binding of LPS to TLR4 in NF-κB and MAPK pathways.

Joh EH, Gu W, Kim DH.

Biochem Pharmacol. 2012 Aug 1;84(3):331-40. doi: 10.1016/j.bcp.2012.04.020. Epub 2012 May 4.

PMID:
22564908
12.

Inducible binding of PU.1 and interacting proteins to the Toll-like receptor 4 promoter during endotoxemia.

Pedchenko TV, Park GY, Joo M, Blackwell TS, Christman JW.

Am J Physiol Lung Cell Mol Physiol. 2005 Sep;289(3):L429-37.

13.

Spi-C has opposing effects to PU.1 on gene expression in progenitor B cells.

Schweitzer BL, Huang KJ, Kamath MB, Emelyanov AV, Birshtein BK, DeKoter RP.

J Immunol. 2006 Aug 15;177(4):2195-207.

14.

Osteopetrosis in mice lacking haematopoietic transcription factor PU.1.

Tondravi MM, McKercher SR, Anderson K, Erdmann JM, Quiroz M, Maki R, Teitelbaum SL.

Nature. 1997 Mar 6;386(6620):81-4.

PMID:
9052784
15.
16.

In vivo depletion of CD206+ M2 macrophages exaggerates lung injury in endotoxemic mice.

Kambara K, Ohashi W, Tomita K, Takashina M, Fujisaka S, Hayashi R, Mori H, Tobe K, Hattori Y.

Am J Pathol. 2015 Jan;185(1):162-71. doi: 10.1016/j.ajpath.2014.09.005. Epub 2014 Oct 27.

PMID:
25447055
17.

Suppression of inflammation response by a novel A₃ adenosine receptor agonist thio-Cl-IB-MECA through inhibition of Akt and NF-κB signaling.

Lee HS, Chung HJ, Lee HW, Jeong LS, Lee SK.

Immunobiology. 2011 Sep;216(9):997-1003. doi: 10.1016/j.imbio.2011.03.008. Epub 2011 Apr 7.

PMID:
21514967
18.

Myeloid development is selectively disrupted in PU.1 null mice.

Anderson KL, Smith KA, Conners K, McKercher SR, Maki RA, Torbett BE.

Blood. 1998 May 15;91(10):3702-10.

19.

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