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Items: 1 to 20 of 155

1.

Expansion of T helper type 17 lymphocytes in patients with chronic granulomatous disease.

Horváth R, Rožková D, Lašťovička J, Poloučková A, Sedláček P, Sedivá A, Spíšek R.

Clin Exp Immunol. 2011 Oct;166(1):26-33. doi: 10.1111/j.1365-2249.2011.04449.x.

2.

Deficiency of Th17 cells in hyper IgE syndrome due to mutations in STAT3.

Ma CS, Chew GY, Simpson N, Priyadarshi A, Wong M, Grimbacher B, Fulcher DA, Tangye SG, Cook MC.

J Exp Med. 2008 Jul 7;205(7):1551-7. doi: 10.1084/jem.20080218.

3.

Clinical features, STAT3 gene mutations and Th17 cell analysis in nine children with hyper-IgE syndrome in mainland China.

Zhang LY, Tian W, Shu L, Jiang LP, Zhan YZ, Liu W, Zhao XD, Cui YX, Tang XM, Wang M, Wu DQ, Yang XQ.

Scand J Immunol. 2013 Sep;78(3):258-65. doi: 10.1111/sji.12063.

4.

Cutaneous manifestations of Hyper IgE syndrome.

Minegishi Y, Saito M.

Allergol Int. 2012 Jun;61(2):191-6. doi: 10.2332/allergolint.12-RAI-0423. Epub 2012 Mar 25. Review.

5.

Milder clinical hyperimmunoglobulin E syndrome phenotype is associated with partial interleukin-17 deficiency.

van de Veerdonk FL, Marijnissen RJ, Joosten LA, Kullberg BJ, Drenth JP, Netea MG, van der Meer JW.

Clin Exp Immunol. 2010 Jan;159(1):57-64. doi: 10.1111/j.1365-2249.2009.04043.x. Epub 2009 Oct 30. Erratum in: Clin Exp Immunol. 2011 May;164(2):289. Marijnissen, R [corrected to Marijnissen R J].

6.

Defective IL-10 signaling in hyper-IgE syndrome results in impaired generation of tolerogenic dendritic cells and induced regulatory T cells.

Saito M, Nagasawa M, Takada H, Hara T, Tsuchiya S, Agematsu K, Yamada M, Kawamura N, Ariga T, Tsuge I, Nonoyama S, Karasuyama H, Minegishi Y.

J Exp Med. 2011 Feb 14;208(2):235-49. doi: 10.1084/jem.20100799. Epub 2011 Feb 7.

7.

Blood CD4+CD45RO+CXCR5+ T cells are decreased but partially functional in signal transducer and activator of transcription 3 deficiency.

Mazerolles F, Picard C, Kracker S, Fischer A, Durandy A.

J Allergy Clin Immunol. 2013 Apr;131(4):1146-56, 1156.e1-5. doi: 10.1016/j.jaci.2012.12.1519. Epub 2013 Feb 10.

PMID:
23403044
8.

Cytokine imbalance in hyper-IgE syndrome: reduced expression of transforming growth factor beta and interferon gamma genes in circulating activated T cells.

Ohga S, Nomura A, Ihara K, Takahata Y, Suga N, Akeda H, Shibata R, Okamura J, Kinukawa N, Hara T.

Br J Haematol. 2003 Apr;121(2):324-31.

PMID:
12694256
9.

Molecular mechanisms of the immunological abnormalities in hyper-IgE syndrome.

Minegishi Y, Saito M.

Ann N Y Acad Sci. 2011 Dec;1246:34-40. doi: 10.1111/j.1749-6632.2011.06280.x. Review.

PMID:
22236428
10.

Molecular explanation for the contradiction between systemic Th17 defect and localized bacterial infection in hyper-IgE syndrome.

Minegishi Y, Saito M, Nagasawa M, Takada H, Hara T, Tsuchiya S, Agematsu K, Yamada M, Kawamura N, Ariga T, Tsuge I, Karasuyama H.

J Exp Med. 2009 Jun 8;206(6):1291-301. doi: 10.1084/jem.20082767. Epub 2009 Jun 1.

11.

Defects in Jak-STAT-mediated cytokine signals cause hyper-IgE syndrome: lessons from a primary immunodeficiency.

Minegishi Y, Karasuyama H.

Int Immunol. 2009 Feb;21(2):105-12. doi: 10.1093/intimm/dxn134. Epub 2008 Dec 15. Review.

PMID:
19088064
12.

Genetic origins of hyper-IgE syndrome.

Minegishi Y, Karasuyama H.

Curr Allergy Asthma Rep. 2008 Sep;8(5):386-91.

PMID:
18682102
13.

Clinical, immunological and genetic features in Taiwanese patients with the phenotype of hyper-immunoglobulin E recurrent infection syndromes (HIES).

Lee WI, Huang JL, Lin SJ, Yeh KW, Chen LC, Ou LS, Yao TC, Hsieh MY, Huang YC, Yu HR, Kuo HC, Yang KD, Jaing TH.

Immunobiology. 2011 Aug;216(8):909-17. doi: 10.1016/j.imbio.2011.01.008. Epub 2011 Jan 18.

PMID:
21324546
14.

Clinical aspects and genetic analysis of Taiwanese patients with the phenotype of hyper-immunoglobulin E recurrent infection syndromes (HIES).

Lee WI, Huang JL, Lin SJ, Yeh KW, Chen LC, Hsieh MY, Huang YC, Kuo HC, Yang KD, Yu HR, Jaing TH, Yang CH.

J Clin Immunol. 2011 Apr;31(2):272-80. doi: 10.1007/s10875-010-9479-1. Epub 2010 Dec 1.

PMID:
21120687
15.

Reduced memory B cells in patients with hyper IgE syndrome.

Speckmann C, Enders A, Woellner C, Thiel D, Rensing-Ehl A, Schlesier M, Rohr J, Jakob T, Oswald E, Kopp MV, Sanal O, Litzman J, Plebani A, Pietrogrande MC, Franco JL, Espanol T, Grimbacher B, Ehl S.

Clin Immunol. 2008 Dec;129(3):448-54. doi: 10.1016/j.clim.2008.08.002. Epub 2008 Oct 2.

PMID:
18835223
16.

STAT3 mutations correlated with hyper-IgE syndrome lead to blockage of IL-6/STAT3 signalling pathway.

He J, Shi J, Xu X, Zhang W, Wang Y, Chen X, Du Y, Zhu N, Zhang J, Wang Q, Yang J.

J Biosci. 2012 Jun;37(2):243-57.

17.

Defective trained immunity in patients with STAT-1-dependent chronic mucocutaneaous candidiasis.

Ifrim DC, Quintin J, Meerstein-Kessel L, Plantinga TS, Joosten LA, van der Meer JW, van de Veerdonk FL, Netea MG.

Clin Exp Immunol. 2015 Sep;181(3):434-40. doi: 10.1111/cei.12642. Epub 2015 Jul 7.

18.

Challenges of genetic counseling in patients with autosomal dominant diseases, such as the hyper-IgE syndrome (STAT3-HIES).

Spielberger BD, Woellner C, Dueckers G, Sawalle-Belohradsky J, Hagl B, Anslinger K, Bayer B, Siepermann K, Niehues T, Grimbacher B, Belohradsky BH, Renner ED.

J Allergy Clin Immunol. 2012 Dec;130(6):1426-8. doi: 10.1016/j.jaci.2012.07.030. Epub 2012 Sep 13. No abstract available.

PMID:
22981789
19.

A novel mutation in the signal transducer and activator of transcription 3 (STAT3) gene, in hyper-IgE syndrome.

Papanastasiou AD, Mantagos S, Papanastasiou DA, Zarkadis IK.

Mol Immunol. 2010 Apr;47(7-8):1629-34. doi: 10.1016/j.molimm.2010.01.010. Epub 2010 Feb 9.

PMID:
20149460
20.

Autosomal dominant STAT3 deficiency and hyper-IgE syndrome: molecular, cellular, and clinical features from a French national survey.

Chandesris MO, Melki I, Natividad A, Puel A, Fieschi C, Yun L, Thumerelle C, Oksenhendler E, Boutboul D, Thomas C, Hoarau C, Lebranchu Y, Stephan JL, Cazorla C, Aladjidi N, Micheau M, Tron F, Baruchel A, Barlogis V, Palenzuela G, Mathey C, Dominique S, Body G, Munzer M, Fouyssac F, Jaussaud R, Bader-Meunier B, Mahlaoui N, Blanche S, Debré M, Le Bourgeois M, Gandemer V, Lambert N, Grandin V, Ndaga S, Jacques C, Harre C, Forveille M, Alyanakian MA, Durandy A, Bodemer C, Suarez F, Hermine O, Lortholary O, Casanova JL, Fischer A, Picard C.

Medicine (Baltimore). 2012 Jul;91(4):e1-19. doi: 10.1097/MD.0b013e31825f95b9.

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