Format
Sort by
Items per page

Send to

Choose Destination

Links from PubMed

Items: 1 to 20 of 74

1.

Prior laparotomy or corticosterone potentiates lipopolysaccharide-induced fever and sickness behaviors.

Hains LE, Loram LC, Taylor FR, Strand KA, Wieseler JL, Barrientos RM, Young JJ, Frank MG, Sobesky J, Martin TJ, Eisenach JC, Maier SF, Johnson JD, Fleshner M, Watkins LR.

J Neuroimmunol. 2011 Oct 28;239(1-2):53-60. doi: 10.1016/j.jneuroim.2011.08.011. Epub 2011 Sep 9.

2.

Prior exposure to glucocorticoids potentiates lipopolysaccharide induced mechanical allodynia and spinal neuroinflammation.

Loram LC, Taylor FR, Strand KA, Frank MG, Sholar P, Harrison JA, Maier SF, Watkins LR.

Brain Behav Immun. 2011 Oct;25(7):1408-15. doi: 10.1016/j.bbi.2011.04.013. Epub 2011 Apr 23.

3.

Prior exposure to corticosterone markedly enhances and prolongs the neuroinflammatory response to systemic challenge with LPS.

Kelly KA, Michalovicz LT, Miller JV, Castranova V, Miller DB, O'Callaghan JP.

PLoS One. 2018 Jan 5;13(1):e0190546. doi: 10.1371/journal.pone.0190546. eCollection 2018.

4.

Calorie restriction dose-dependently abates lipopolysaccharide-induced fever, sickness behavior, and circulating interleukin-6 while increasing corticosterone.

MacDonald L, Hazi A, Paolini AG, Kent S.

Brain Behav Immun. 2014 Aug;40:18-26. doi: 10.1016/j.bbi.2014.01.005. Epub 2014 Jan 15.

PMID:
24440143
5.

Differences in the relative involvement of peripherally released interleukin (IL)-6, brain IL-1β and prostanoids in mediating lipopolysaccharide-induced fever and sickness behavior.

Harden LM, du Plessis I, Roth J, Loram LC, Poole S, Laburn HP.

Psychoneuroendocrinology. 2011 Jun;36(5):608-22. doi: 10.1016/j.psyneuen.2010.09.003. Epub 2010 Oct 5.

PMID:
20926198
6.

Circulating cytokines and endotoxin are not necessary for the activation of the sickness or corticosterone response produced by peripheral E. coli challenge.

Campisi J, Hansen MK, O'Connor KA, Biedenkapp JC, Watkins LR, Maier SF, Fleshner M.

J Appl Physiol (1985). 2003 Nov;95(5):1873-82. Epub 2003 Jul 18.

7.

Interleukin-6 and leptin mediate lipopolysaccharide-induced fever and sickness behavior.

Harden LM, du Plessis I, Poole S, Laburn HP.

Physiol Behav. 2006 Sep 30;89(2):146-55. Epub 2006 Jul 12.

PMID:
16842830
8.

The role of IL-1beta in stress-induced sensitization of proinflammatory cytokine and corticosterone responses.

Johnson JD, O'Connor KA, Watkins LR, Maier SF.

Neuroscience. 2004;127(3):569-77.

PMID:
15283957
9.

Prior exposure to glucocorticoids sensitizes the neuroinflammatory and peripheral inflammatory responses to E. coli lipopolysaccharide.

Frank MG, Miguel ZD, Watkins LR, Maier SF.

Brain Behav Immun. 2010 Jan;24(1):19-30. doi: 10.1016/j.bbi.2009.07.008. Epub 2009 Jul 30.

PMID:
19647070
10.

Glucocorticoid receptor dimerization is required for proper recovery of LPS-induced inflammation, sickness behavior and metabolism in mice.

Silverman MN, Mukhopadhyay P, Belyavskaya E, Tonelli LH, Revenis BD, Doran JH, Ballard BE, Tam J, Pacher P, Sternberg EM.

Mol Psychiatry. 2013 Sep;18(9):1006-17. doi: 10.1038/mp.2012.131. Epub 2012 Oct 23.

PMID:
23089634
11.

Kinetic characteristics of euflammation: the induction of controlled inflammation without overt sickness behavior.

Tarr AJ, Liu X, Reed NS, Quan N.

Brain Behav Immun. 2014 Nov;42:96-108. doi: 10.1016/j.bbi.2014.06.002. Epub 2014 Jun 12.

12.

Pain intensity and duration can be enhanced by prior challenge: initial evidence suggestive of a role of microglial priming.

Hains LE, Loram LC, Weiseler JL, Frank MG, Bloss EB, Sholar P, Taylor FR, Harrison JA, Martin TJ, Eisenach JC, Maier SF, Watkins LR.

J Pain. 2010 Oct;11(10):1004-14. doi: 10.1016/j.jpain.2010.01.271.

14.

Inhibition of nitric oxide synthase accentuates endotoxin-induced sickness behavior in mice.

Ribeiro DE, Maiolini VM, Soncini R, Antunes-Rodrigues J, Elias LL, Vilela FC, Giusti-Paiva A.

Pharmacol Biochem Behav. 2013 Jan;103(3):535-40. doi: 10.1016/j.pbb.2012.09.022. Epub 2012 Oct 6.

15.

Isofraxidin exhibited anti-inflammatory effects in vivo and inhibited TNF-α production in LPS-induced mouse peritoneal macrophages in vitro via the MAPK pathway.

Niu X, Xing W, Li W, Fan T, Hu H, Li Y.

Int Immunopharmacol. 2012 Oct;14(2):164-71. doi: 10.1016/j.intimp.2012.06.022. Epub 2012 Jul 15.

PMID:
22800929
16.

Inhibition of nitric oxide in LPS-stimulated macrophages of young and senescent mice by δ-tocotrienol and quercetin.

Qureshi AA, Tan X, Reis JC, Badr MZ, Papasian CJ, Morrison DC, Qureshi N.

Lipids Health Dis. 2011 Dec 20;10:239. doi: 10.1186/1476-511X-10-239.

17.

Sex-specific social regulation of inflammatory responses and sickness behaviors.

Yee JR, Prendergast BJ.

Brain Behav Immun. 2010 Aug;24(6):942-51. doi: 10.1016/j.bbi.2010.03.006. Epub 2010 Mar 17.

18.

Neonatal programming of the rat neuroimmune response: stimulus specific changes elicited by bacterial and viral mimetics.

Ellis S, Mouihate A, Pittman QJ.

J Physiol. 2006 Mar 15;571(Pt 3):695-701. Epub 2006 Jan 19.

19.

Comparison of anorexia, lethargy, and fever induced by bacterial and viral mimetics in rats.

Hopwood N, Maswanganyi T, Harden LM.

Can J Physiol Pharmacol. 2009 Mar;87(3):211-20. doi: 10.1139/y09-003.

PMID:
19295662
20.

Chronic corticosterone administration does not potentiate unconditioned freezing to the predator odor, trimethylthiazoline.

Rosen JB, Donley MP, Gray D, West EA, Morgan MA, Schulkin J.

Behav Brain Res. 2008 Dec 1;194(1):32-8. doi: 10.1016/j.bbr.2008.06.019. Epub 2008 Jun 27.

PMID:
18634830

Supplemental Content

Support Center