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Items: 1 to 20 of 252

1.

Interferon regulatory transcription factor 3 protects mice from uterine horn pathology during Chlamydia muridarum genital infection.

Prantner D, Sikes JD, Hennings L, Savenka AV, Basnakian AG, Nagarajan UM.

Infect Immun. 2011 Oct;79(10):3922-33. doi: 10.1128/IAI.00140-11. Epub 2011 Jul 25.

2.

Type I interferon signaling exacerbates Chlamydia muridarum genital infection in a murine model.

Nagarajan UM, Prantner D, Sikes JD, Andrews CW Jr, Goodwin AM, Nagarajan S, Darville T.

Infect Immun. 2008 Oct;76(10):4642-8. doi: 10.1128/IAI.00629-08. Epub 2008 Jul 28.

3.

Antigen-specific CD4+ T cells produce sufficient IFN-gamma to mediate robust protective immunity against genital Chlamydia muridarum infection.

Li W, Murthy AK, Guentzel MN, Seshu J, Forsthuber TG, Zhong G, Arulanandam BP.

J Immunol. 2008 Mar 1;180(5):3375-82.

4.
5.

Vaginal chlamydial clearance following primary or secondary infection in mice occurs independently of TNF-α.

Kamalakaran S, Chaganty BK, Gupta R, Guentzel MN, Chambers JP, Murthy AK, Arulanandam BP.

Front Cell Infect Microbiol. 2013 Mar 11;3:11. doi: 10.3389/fcimb.2013.00011. eCollection 2013.

6.

Role of STAT1 in Chlamydia-Induced Type-1 Interferon Production in Oviduct Epithelial Cells.

Hosey KL, Hu S, Derbigny WA.

J Interferon Cytokine Res. 2015 Nov;35(11):901-16. doi: 10.1089/jir.2015.0013. Epub 2015 Aug 11.

8.

The contribution of Chlamydia-specific CD8⁺ T cells to upper genital tract pathology.

Vlcek KR, Li W, Manam S, Zanotti B, Nicholson BJ, Ramsey KH, Murthy AK.

Immunol Cell Biol. 2016 Feb;94(2):208-12. doi: 10.1038/icb.2015.74. Epub 2015 Sep 1.

9.

Tumor necrosis factor alpha production from CD8+ T cells mediates oviduct pathological sequelae following primary genital Chlamydia muridarum infection.

Murthy AK, Li W, Chaganty BK, Kamalakaran S, Guentzel MN, Seshu J, Forsthuber TG, Zhong G, Arulanandam BP.

Infect Immun. 2011 Jul;79(7):2928-35. doi: 10.1128/IAI.05022-11. Epub 2011 May 2.

10.

Significant role of IL-1 signaling, but limited role of inflammasome activation, in oviduct pathology during Chlamydia muridarum genital infection.

Nagarajan UM, Sikes JD, Yeruva L, Prantner D.

J Immunol. 2012 Mar 15;188(6):2866-75. doi: 10.4049/jimmunol.1103461. Epub 2012 Feb 13.

11.

Mice deficient in MyD88 Develop a Th2-dominant response and severe pathology in the upper genital tract following Chlamydia muridarum infection.

Chen L, Lei L, Chang X, Li Z, Lu C, Zhang X, Wu Y, Yeh IT, Zhong G.

J Immunol. 2010 Mar 1;184(5):2602-10. doi: 10.4049/jimmunol.0901593. Epub 2010 Feb 1.

12.

OT-1 mice display minimal upper genital tract pathology following primary intravaginal Chlamydia muridarum infection.

Manam S, Nicholson BJ, Murthy AK.

Pathog Dis. 2013 Apr;67(3):221-4. doi: 10.1111/2049-632X.12032. Epub 2013 Mar 11.

13.

MAVS-dependent IRF3/7 bypass of interferon β-induction restricts the response to measles infection in CD150Tg mouse bone marrow-derived dendritic cells.

Takaki H, Honda K, Atarashi K, Kobayashi F, Ebihara T, Oshiumi H, Matsumoto M, Shingai M, Seya T.

Mol Immunol. 2014 Feb;57(2):100-10. doi: 10.1016/j.molimm.2013.08.007. Epub 2013 Oct 4.

PMID:
24096085
14.

Analyses of the pathways involved in early- and late-phase induction of IFN-beta during C. muridarum infection of oviduct epithelial cells.

Hu S, Hosey KL, Derbigny WA.

PLoS One. 2015 Mar 23;10(3):e0119235. doi: 10.1371/journal.pone.0119235. eCollection 2015.

15.

The p47phox deficiency significantly attenuates the pathogenicity of Chlamydia muridarum in the mouse oviduct but not uterine tissues.

Dai J, Tang L, Chen J, Yu P, Chen Z, Zhong G.

Microbes Infect. 2016 Mar;18(3):190-8. doi: 10.1016/j.micinf.2015.11.003. Epub 2015 Dec 2.

PMID:
26645958
16.

The contribution of interleukin-12/interferon-gamma axis in protection against neonatal pulmonary Chlamydia muridarum challenge.

Jupelli M, Selby DM, Guentzel MN, Chambers JP, Forsthuber TG, Zhong G, Murthy AK, Arulanandam BP.

J Interferon Cytokine Res. 2010 Jun;30(6):407-15. doi: 10.1089/jir.2009.0083.

17.

Anti-chlamydial Th17 responses are controlled by the inducible costimulator partially through phosphoinositide 3-kinase signaling.

Gao X, Gigoux M, Yang J, Leconte J, Yang X, Suh WK.

PLoS One. 2012;7(12):e52657. doi: 10.1371/journal.pone.0052657. Epub 2012 Dec 20.

18.

The recall response induced by genital challenge with Chlamydia muridarum protects the oviduct from pathology but not from reinfection.

Riley MM, Zurenski MA, Frazer LC, O'Connell CM, Andrews CW Jr, Mintus M, Darville T.

Infect Immun. 2012 Jun;80(6):2194-203. doi: 10.1128/IAI.00169-12. Epub 2012 Mar 19.

19.

Plasmacytoid dendritic cells modulate nonprotective T-cell responses to genital infection by Chlamydia muridarum.

Moniz RJ, Chan AM, Gordon LK, Braun J, Arditi M, Kelly KA.

FEMS Immunol Med Microbiol. 2010 Apr;58(3):397-404. doi: 10.1111/j.1574-695X.2010.00653.x. Epub 2010 Jan 19.

20.

Interruption of CXCL13-CXCR5 axis increases upper genital tract pathology and activation of NKT cells following chlamydial genital infection.

Jiang J, Karimi O, Ouburg S, Champion CI, Khurana A, Liu G, Freed A, Pleijster J, Rozengurt N, Land JA, Surcel HM, Tiitinen A, Paavonen J, Kronenberg M, Morré SA, Kelly KA.

PLoS One. 2012;7(11):e47487. doi: 10.1371/journal.pone.0047487. Epub 2012 Nov 26. Erratum in: PLoS One. 2013;8(5). doi:10.1371/annotation/2b6aab70-3c92-490e-86f0-ae15787dfa6e.

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