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Items: 1 to 20 of 75

1.

Cleavage of IgG1 and IgG3 by gingipain K from Porphyromonas gingivalis may compromise host defense in progressive periodontitis.

Vincents B, Guentsch A, Kostolowska D, von Pawel-Rammingen U, Eick S, Potempa J, Abrahamson M.

FASEB J. 2011 Oct;25(10):3741-50. doi: 10.1096/fj.11-187799. Epub 2011 Jul 18.

2.

Cleavage of IgG1 in gingival crevicular fluid is associated with the presence of Porphyromonas gingivalis.

Guentsch A, Hirsch C, Pfister W, Vincents B, Abrahamson M, Sroka A, Potempa J, Eick S.

J Periodontal Res. 2013 Aug;48(4):458-65. doi: 10.1111/jre.12027. Epub 2012 Nov 1.

3.

Inactive Gingipains from P. gingivalis Selectively Skews T Cells toward a Th17 Phenotype in an IL-6 Dependent Manner.

Glowczyk I, Wong A, Potempa B, Babyak O, Lech M, Lamont RJ, Potempa J, Koziel J.

Front Cell Infect Microbiol. 2017 Apr 27;7:140. doi: 10.3389/fcimb.2017.00140. eCollection 2017.

4.

Gingipain-specific IgG in the sera of patients with periodontal disease is necessary for opsonophagocytosis of Porphyromonas gingivalis.

Gibson FC 3rd, Savelli J, Van Dyke TE, Genco CA.

J Periodontol. 2005 Oct;76(10):1629-36.

PMID:
16253083
5.

Porphyromonas gingivalis Gingipain-Dependently Enhances IL-33 Production in Human Gingival Epithelial Cells.

Tada H, Matsuyama T, Nishioka T, Hagiwara M, Kiyoura Y, Shimauchi H, Matsushita K.

PLoS One. 2016 Apr 8;11(4):e0152794. doi: 10.1371/journal.pone.0152794. eCollection 2016.

6.

Porphyromonas gingivalis-derived lysine gingipain enhances osteoclast differentiation induced by tumor necrosis factor-α and interleukin-1β but suppresses that by interleukin-17A: importance of proteolytic degradation of osteoprotegerin by lysine gingipain.

Akiyama T, Miyamoto Y, Yoshimura K, Yamada A, Takami M, Suzawa T, Hoshino M, Imamura T, Akiyama C, Yasuhara R, Mishima K, Maruyama T, Kohda C, Tanaka K, Potempa J, Yasuda H, Baba K, Kamijo R.

J Biol Chem. 2014 May 30;289(22):15621-30. doi: 10.1074/jbc.M113.520510. Epub 2014 Apr 22.

7.
8.

Role of gingipains in growth of Porphyromonas gingivalis in the presence of human serum albumin.

Grenier D, Imbeault S, Plamondon P, Grenier G, Nakayama K, Mayrand D.

Infect Immun. 2001 Aug;69(8):5166-72.

9.

Cleavage of host cytokeratin-6 by lysine-specific gingipain induces gingival inflammation in periodontitis patients.

Tancharoen S, Matsuyama T, Kawahara K, Tanaka K, Lee LJ, Machigashira M, Noguchi K, Ito T, Imamura T, Potempa J, Kikuchi K, Maruyama I.

PLoS One. 2015 Feb 17;10(2):e0117775. doi: 10.1371/journal.pone.0117775. eCollection 2015.

10.

Gingipain-dependent degradation of mammalian target of rapamycin pathway proteins by the periodontal pathogen Porphyromonas gingivalis during invasion.

Stafford P, Higham J, Pinnock A, Murdoch C, Douglas CW, Stafford GP, Lambert DW.

Mol Oral Microbiol. 2013 Oct;28(5):366-78. doi: 10.1111/omi.12030. Epub 2013 May 29.

11.

Structure and mechanism of cysteine peptidase gingipain K (Kgp), a major virulence factor of Porphyromonas gingivalis in periodontitis.

de Diego I, Veillard F, Sztukowska MN, Guevara T, Potempa B, Pomowski A, Huntington JA, Potempa J, Gomis-Rüth FX.

J Biol Chem. 2014 Nov 14;289(46):32291-302. doi: 10.1074/jbc.M114.602052. Epub 2014 Sep 29.

12.

Characterization of the binding activities of proteinase-adhesin complexes from Porphyromonas gingivalis.

Pike RN, Potempa J, McGraw W, Coetzer TH, Travis J.

J Bacteriol. 1996 May;178(10):2876-82.

13.

Porphyromonas gingivalis proteinases as virulence factors in the development of periodontitis.

Travis J, Pike R, Imamura T, Potempa J.

J Periodontal Res. 1997 Jan;32(1 Pt 2):120-5. Review.

PMID:
9085221
14.

Modification of cystatin C activity by bacterial proteinases and neutrophil elastase in periodontitis.

Abrahamson M, Wikström M, Potempa J, Renvert S, Hall A.

Mol Pathol. 1997 Dec;50(6):291-7.

15.

Kinin danger signals proteolytically released by gingipain induce Fimbriae-specific IFN-gamma- and IL-17-producing T cells in mice infected intramucosally with Porphyromonas gingivalis.

Monteiro AC, Scovino A, Raposo S, Gaze VM, Cruz C, Svensjö E, Narciso MS, Colombo AP, Pesquero JB, Feres-Filho E, Nguyen KA, Sroka A, Potempa J, Scharfstein J.

J Immunol. 2009 Sep 15;183(6):3700-11. doi: 10.4049/jimmunol.0900895. Epub 2009 Aug 17.

16.

Hemoglobinase activity of the lysine gingipain protease (Kgp) of Porphyromonas gingivalis W83.

Lewis JP, Dawson JA, Hannis JC, Muddiman D, Macrina FL.

J Bacteriol. 1999 Aug;181(16):4905-13.

17.

Inhibition of C3 and IgG proteolysis enhances phagocytosis of Porphyromonas gingivalis.

Cutler CW, Arnold RR, Schenkein HA.

J Immunol. 1993 Dec 15;151(12):7016-29.

PMID:
8258706
18.

Structure of the lysine specific protease Kgp from Porphyromonas gingivalis, a target for improved oral health.

Gorman MA, Seers CA, Michell BJ, Feil SC, Huq NL, Cross KJ, Reynolds EC, Parker MW.

Protein Sci. 2015 Jan;24(1):162-6. doi: 10.1002/pro.2589. Epub 2014 Dec 3.

19.

Porphyromonas gingivalis: an invasive and evasive opportunistic oral pathogen.

Bostanci N, Belibasakis GN.

FEMS Microbiol Lett. 2012 Aug;333(1):1-9. doi: 10.1111/j.1574-6968.2012.02579.x. Epub 2012 May 28. Review.

20.

Gingipains from Porphyromonas gingivalis increase the chemotactic and respiratory burst-priming properties of the 77-amino-acid interleukin-8 variant.

Dias IH, Marshall L, Lambert PA, Chapple IL, Matthews JB, Griffiths HR.

Infect Immun. 2008 Jan;76(1):317-23. Epub 2007 Nov 19.

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