Format
Sort by
Items per page

Send to

Choose Destination

Links from PubMed

Items: 1 to 20 of 149

1.

Role of the CaMKII/NMDA receptor complex in the maintenance of synaptic strength.

Sanhueza M, Fernandez-Villalobos G, Stein IS, Kasumova G, Zhang P, Bayer KU, Otmakhov N, Hell JW, Lisman J.

J Neurosci. 2011 Jun 22;31(25):9170-8. doi: 10.1523/JNEUROSCI.1250-11.2011.

2.

On the mechanism of synaptic depression induced by CaMKIIN, an endogenous inhibitor of CaMKII.

Gouet C, Aburto B, Vergara C, Sanhueza M.

PLoS One. 2012;7(11):e49293. doi: 10.1371/journal.pone.0049293. Epub 2012 Nov 8.

3.

The CaMKII/GluN2B Protein Interaction Maintains Synaptic Strength.

Barcomb K, Hell JW, Benke TA, Bayer KU.

J Biol Chem. 2016 Jul 29;291(31):16082-9. doi: 10.1074/jbc.M116.734822. Epub 2016 May 31.

4.

Mechanisms of CaMKII action in long-term potentiation.

Lisman J, Yasuda R, Raghavachari S.

Nat Rev Neurosci. 2012 Feb 15;13(3):169-82. doi: 10.1038/nrn3192. Review.

5.

Reversal of synaptic memory by Ca2+/calmodulin-dependent protein kinase II inhibitor.

Sanhueza M, McIntyre CC, Lisman JE.

J Neurosci. 2007 May 9;27(19):5190-9.

6.

Biochemical principles underlying the stable maintenance of LTP by the CaMKII/NMDAR complex.

Lisman J, Raghavachari S.

Brain Res. 2015 Sep 24;1621:51-61. doi: 10.1016/j.brainres.2014.12.010. Epub 2014 Dec 13.

7.

CaMKII regulation in information processing and storage.

Coultrap SJ, Bayer KU.

Trends Neurosci. 2012 Oct;35(10):607-18. doi: 10.1016/j.tins.2012.05.003. Epub 2012 Jun 19. Review.

8.

The CaMKII/NMDAR complex as a molecular memory.

Sanhueza M, Lisman J.

Mol Brain. 2013 Feb 14;6:10. doi: 10.1186/1756-6606-6-10. Review.

9.

Modelling the dynamics of CaMKII-NMDAR complex related to memory formation in synapses: the possible roles of threonine 286 autophosphorylation of CaMKII in long term potentiation.

He Y, Kulasiri D, Samarasinghe S.

J Theor Biol. 2015 Jan 21;365:403-19. doi: 10.1016/j.jtbi.2014.11.001. Epub 2014 Nov 11.

PMID:
25446714
10.

Uncoupling the D1-N-methyl-D-aspartate (NMDA) receptor complex promotes NMDA-dependent long-term potentiation and working memory.

Nai Q, Li S, Wang SH, Liu J, Lee FJ, Frankland PW, Liu F.

Biol Psychiatry. 2010 Feb 1;67(3):246-54. doi: 10.1016/j.biopsych.2009.08.011. Epub 2009 Oct 28.

PMID:
19846062
11.

Co-induction of LTP and LTD and its regulation by protein kinases and phosphatases.

Grey KB, Burrell BD.

J Neurophysiol. 2010 May;103(5):2737-46. doi: 10.1152/jn.01112.2009. Epub 2010 Mar 24.

12.

Is persistent activity of calcium/calmodulin-dependent kinase required for the maintenance of LTP?

Chen HX, Otmakhov N, Strack S, Colbran RJ, Lisman JE.

J Neurophysiol. 2001 Apr;85(4):1368-76.

PMID:
11287461
13.

Protein kinase C promotes N-methyl-D-aspartate (NMDA) receptor trafficking by indirectly triggering calcium/calmodulin-dependent protein kinase II (CaMKII) autophosphorylation.

Yan JZ, Xu Z, Ren SQ, Hu B, Yao W, Wang SH, Liu SY, Lu W.

J Biol Chem. 2011 Jul 15;286(28):25187-200. doi: 10.1074/jbc.M110.192708. Epub 2011 May 23.

14.

Synaptic strength at the temporoammonic input to the hippocampal CA1 region in vivo is regulated by NMDA receptors, metabotropic glutamate receptors and voltage-gated calcium channels.

Aksoy-Aksel A, Manahan-Vaughan D.

Neuroscience. 2015 Nov 19;309:191-9. doi: 10.1016/j.neuroscience.2015.03.014. Epub 2015 Mar 17.

16.

The effects of NR2 subunit-dependent NMDA receptor kinetics on synaptic transmission and CaMKII activation.

Santucci DM, Raghavachari S.

PLoS Comput Biol. 2008 Oct;4(10):e1000208. doi: 10.1371/journal.pcbi.1000208. Epub 2008 Oct 31.

17.

Kv4.2 block of long-term potentiation is partially dependent on synaptic NMDA receptor remodeling.

Jung SC, Eun SY, Kim J, Hoffman DA.

Brain Res Bull. 2011 Jan 15;84(1):17-21. doi: 10.1016/j.brainresbull.2010.09.012. Epub 2010 Oct 8.

18.

CaMKII-dependent phosphorylation of NR2A and NR2B is decreased in animals characterized by hippocampal damage and impaired LTP.

Caputi A, Gardoni F, Cimino M, Pastorino L, Cattabeni F, Di Luca M.

Eur J Neurosci. 1999 Jan;11(1):141-8.

PMID:
9987018
19.

Ca2+/calmodulin-dependent protein kinase II and protein kinase C activities mediate extracellular glucose-regulated hippocampal synaptic efficacy.

Moriguchi S, Oomura Y, Shioda N, Han F, Hori N, Aou S, Fukunaga K.

Mol Cell Neurosci. 2011 Jan;46(1):101-7. doi: 10.1016/j.mcn.2010.08.010. Epub 2010 Aug 31.

PMID:
20807573
20.

Supplemental Content

Support Center